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🚨🚨🚨🚨🚨🚨🚨🚨 BREAKING: Matthew Budoff MD has now presented the matched analysis for: KETO (#LMHRstudy) vs Control (#MiHeart) -🙏Please RT🙏- METHODS – 80 Participants of #LMHRstudy fell within #MiHeart age range and were then matched 1:1 for age, gender, race, diabetes mellitus, hyperlipidemia, hypertension, and past smoking to asymptomatic subjects...

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Dave Feldman

88,086 subscribers

1,105,960 次观看 • 2 年前 •via X (Twitter)

教育健康养生科学技术#LMHRstudy#MiHeart#LMHRstudy#MiHeart
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Nick Norwitz 的头像
Nick Norwitz2 年前

@BudoffMd I can now report stats consistent with the hypothesis that @realDaveFeldman is, in fact, the devil How dare he rise to the challenge and publish papers and help produce an #LMHRstudy without an MD! How dare he chase his curiosity with integrity and an open-mind! The horror!

Dave Feldman 的头像
Dave Feldman2 年前

@BudoffMd 😂

Mario Kratz 的头像
Mario Kratz2 年前

Super interesting. It's early days, and a small cohort, but I feel that this line of work promises to teach us something new about the relationship between LDL-cholesterol/apo B-containing particles and ASCVD. Even though it's just the baseline data, given that participants were on keto, supposedly with similarly high LDL-cholesterol, for almost 5 years, one would have expected at least a small detectable difference in the amount of plaque. It's these unexpected findings that sometimes teach us the most, so I will be following publications and future follow-ups of this or similar cohorts closely. The longitudinal data with changes in plaque and CAC scores will be very interesting. Definitely looks like a good effort, and I know firsthand how tricky it can be to obtain funding for something that tests an against-the-grain hypothesis like this. Congrats, @realDaveFeldman, @BudoffMd, and team. On the flip side, I was just about to publish a 45-min video about blood lipids and ASCVD risk. This new finding will now necessitate a bit of a revision ...

Dave Feldman 的头像
Dave Feldman2 年前

Hi @mario_kratz -- First, sorry to hear about disrupting the planned video, I know you take a lot of time and care in putting them together (I've watched many). But of course, you also know I couldn't have discussed until released (yourself being in the craft). Second -- if you don't mind my asking, (and there's no wrong answer to this, btw) have these data impacted your own personal opinion on what you were going to share in the planned video?

Jeffry Gerber, MD 的头像
Jeffry Gerber, MD2 年前

@BudoffMd I've been waiting almost 25 years for a study such as this. Just what's been needed looking at CV risk, proper imaging (CAC, CTCA) and ketogenic diets. Kudos to @realDaveFeldman @BudoffMd @nicknorwitz @khurramn1 @Agatston_Center and many others...

The Metabolic Psychologist 的头像
The Metabolic Psychologist2 年前

@BudoffMd Hmm, so 4.7 yrs of sky high LDL levels didn't result in more coronary plaque compared with control participants with less than half the LDL level. Surprise, surprise! Maybe high LDL doesn't confer the risks we were told, at least for LMHRs (like me).

Dr Brad Stanfield 的头像
Dr Brad Stanfield2 年前

@BudoffMd Thanks for sharing Dave. When will the paper be published? Looking forward to reading it

Dave Feldman 的头像
Dave Feldman2 年前

@BudoffMd Abstract will be published in Metabolism. The full paper is submitted, but as you know, that's not a predictable process with regard to timing – but hopefully it will be out soon.

Remnant | MD 的头像
Remnant | MD2 年前

@BudoffMd the next q about "ascertainment bias" the whole claim is that high LDL, in and of itself, causes atherosclerosis. when it doesn't, they just waive it away with "well the people are just healthy." Exactly! That's the point.

Dave Feldman 的头像
Dave Feldman2 年前

@BudoffMd I’m interested in anything that would be considered a confounder in that it should also likewise be considered a means of treatment for those with diseases associated with high LDL and ASCVD (ie monogenetic FH)

相关视频

💥🫀#LMHRstudy DATA DROP🫀💥 👉 KETO (#LMHR) group had mean LDL-C 272 mg/dl (max 591 mg/dl), age ~55 👉NO INCREASED PLAQUE on CCTA after 4.7 years on #ketodiet vs. to matched control group 👉NO ASSOCIATION between LDL-C and Plaque 👇Clip from vid: More resources: PI, Matt Budoff MD, presentation: Dave's commentary: SELECTED PAST PAPERS Lipid Energy Model: LMHR, first paper: JCL LMHR editorial: RT' Share Widely! More to come... cc' Dave Feldman
1:38

💥🫀#LMHRstudy DATA DROP🫀💥 👉 KETO (#LMHR) group had mean LDL-C 272 mg/dl (max 591 mg/dl), age ~55 👉NO INCREASED PLAQUE on CCTA after 4.7 years on #ketodiet vs. to matched control group 👉NO ASSOCIATION between LDL-C and Plaque 👇Clip from vid: More resources: PI, Matt Budoff MD, presentation: Dave's commentary: SELECTED PAST PAPERS Lipid Energy Model: LMHR, first paper: JCL LMHR editorial: RT' Share Widely! More to come... cc' Dave Feldman

Nick Norwitz MD PhD

223,299 次观看 • 2 年前

🚨🚨 New Video! 🚨🚨 – A Simple Analogy to Explain High #ApoB/#LDL ...and Why #LMHR May Be Different – 🙏Please RT🙏 ✅ It's just 8 min, and layperson friendly -- watch it now ✅ It drills into the key mechanism of relevance: successful vs failed delivery ✅ Why modern science may be commonly looking at it backward (and why it's easy to miss) ✅ How the #LMHR could explain this – and why it could potentially change how we see #ApoB & #LDL #Cholesterol in the context of risk ⚠️ It should go without saying this video is intentionally simplified. For the full #LEM paper, see (and be sure to follow Nick Norwitz MD PhD, ofc) 😅 I put a tremendous level of work into this one (much more than I intended), but I wanted it ready before this weekend and the coming match analysis from our #LMHRstudy ( )
8:03

🚨🚨 New Video! 🚨🚨 – A Simple Analogy to Explain High #ApoB/#LDL ...and Why #LMHR May Be Different – 🙏Please RT🙏 ✅ It's just 8 min, and layperson friendly -- watch it now ✅ It drills into the key mechanism of relevance: successful vs failed delivery ✅ Why modern science may be commonly looking at it backward (and why it's easy to miss) ✅ How the #LMHR could explain this – and why it could potentially change how we see #ApoB & #LDL #Cholesterol in the context of risk ⚠️ It should go without saying this video is intentionally simplified. For the full #LEM paper, see (and be sure to follow Nick Norwitz MD PhD, ofc) 😅 I put a tremendous level of work into this one (much more than I intended), but I wanted it ready before this weekend and the coming match analysis from our #LMHRstudy ( )

Dave Feldman

78,305 次观看 • 2 年前

There’s been a lot of buzz—and confusion—about the new Keto-CTA study, examining plaque progression in Lean Mass Hyper-Responders (LMHRs). Much of the social media debate has centered on whether high LDL on keto is safe or dangerous, driven largely by how to interpret the supplemental table comparing this study to others on LDL and plaque progression. In this episode of the Metabolic Mind Podcast, we sit down with Dr. Matthew Budoff, a world-renowned cardiologist, cardiac CT researcher, and the study's lead investigator, to discuss the the supplemental table, what the plaque markers mean, and how this fits into the discussion of high- vs -low-risk plaque progression. In this episode, we cover: ✅ What PAV (Percent Atheroma Volume) is, what it actually measures, and why it matters ✅ Why a 50% increase in plaque may sound scary, but can be deceiving ✅ The difference between “treatment-naive” and “treated” participants ✅ What the Miami Heart Study comparison reveals about keto, LDL, and plaque ✅ Why LDL alone may not tell the whole story about heart disease risk ✅ How some high-risk individuals may still benefit from statins and other therapies This study doesn’t answer whether keto causes heart disease or not. Instead, it shows that high LDL on a ketogenic diet is not a reliable predictor of plaque progression across all individuals. What is predictive? The presence of existing plaque. 💡 Key takeaway: Relying on surrogate markers of heart disease, like LDL and ApoB, is not the best way to assess heart disease risk in all populations. If you're concerned about how elevated LDL may be affecting your heart health, the best next step is to speak with your doctor about cardiac imaging to directly assess plaque and gain a clearer picture of your individual risk. Expert Featured: - Dr. Matthew Budoff - X: - Resources Mentioned: Plaque Begets Plaque, ApoB Does Not CMEs Mentioned: Managing Major Mental Illness with Dietary Change: The New Science of Hope Brain Energy: The Metabolic Theory of Mental Illness Follow our channel for more information and education from Bret Scher, MD, FACC, including interviews with leading experts in Metabolic Psychiatry. Learn more about metabolic psychiatry and find helpful resources at
32:51

There’s been a lot of buzz—and confusion—about the new Keto-CTA study, examining plaque progression in Lean Mass Hyper-Responders (LMHRs). Much of the social media debate has centered on whether high LDL on keto is safe or dangerous, driven largely by how to interpret the supplemental table comparing this study to others on LDL and plaque progression. In this episode of the Metabolic Mind Podcast, we sit down with Dr. Matthew Budoff, a world-renowned cardiologist, cardiac CT researcher, and the study's lead investigator, to discuss the the supplemental table, what the plaque markers mean, and how this fits into the discussion of high- vs -low-risk plaque progression. In this episode, we cover: ✅ What PAV (Percent Atheroma Volume) is, what it actually measures, and why it matters ✅ Why a 50% increase in plaque may sound scary, but can be deceiving ✅ The difference between “treatment-naive” and “treated” participants ✅ What the Miami Heart Study comparison reveals about keto, LDL, and plaque ✅ Why LDL alone may not tell the whole story about heart disease risk ✅ How some high-risk individuals may still benefit from statins and other therapies This study doesn’t answer whether keto causes heart disease or not. Instead, it shows that high LDL on a ketogenic diet is not a reliable predictor of plaque progression across all individuals. What is predictive? The presence of existing plaque. 💡 Key takeaway: Relying on surrogate markers of heart disease, like LDL and ApoB, is not the best way to assess heart disease risk in all populations. If you're concerned about how elevated LDL may be affecting your heart health, the best next step is to speak with your doctor about cardiac imaging to directly assess plaque and gain a clearer picture of your individual risk. Expert Featured: - Dr. Matthew Budoff - X: - Resources Mentioned: Plaque Begets Plaque, ApoB Does Not CMEs Mentioned: Managing Major Mental Illness with Dietary Change: The New Science of Hope Brain Energy: The Metabolic Theory of Mental Illness Follow our channel for more information and education from Bret Scher, MD, FACC, including interviews with leading experts in Metabolic Psychiatry. Learn more about metabolic psychiatry and find helpful resources at

Metabolic Mind

98,147 次观看 • 1 年前

💥The response to yesterday's paper has been UNREAL! My phone hasn't stopped buzzing. If you've been under a rock, here are some highlights in <90 sec 👇 🫀Most participants showed no or minimal progression of coronary plaque 🫀Neither ApoB nor LDL exposure predicted plaque progression 🫀But plaque predicted plaque progression What does this mean, in a phrase: "While this paper doesn't disprove the lipid heart hypothesis, it does help expose a major blindspot." There will be a LOT more coming soon... I promise you that... #Cholesterol #LDL #KetoDiet #ApoB #CAC #CCTA #LMHR #LEM #SaturatedFat #HeartHealth #MetabolicHealth cc Dave Feldman Dr Aseem Malhotra Joseph Everett (WIL) Jan Ellison Baszucki Metabolic Mind Also not, this is not a one off. There have been 10+ papers leading to this moment... and we are just getting warmed up... PMID: 40192608, Plaque Begets Plaque. ApoB does not: Longitudinal data from the KETO-CTA trial PMID: 39372369, Carbohydrate restriction-induced elevations in LDL-cholesterol and atherosclerosis: The KETO trial PMID: 38237807, Increased LDL cholesterol in adults with normal but not high body weight: a meta-analysis PMID: 35629964, The Lipid Energy Model: reimagining lipoprotein function in the context of carbohydrate-restricted diets PMID: 35106434, Elevated LDL-cholesterol with a carbohydrate-restricted diet: evidence for a “lean mass hyper-responder” phenotype
1:28

💥The response to yesterday's paper has been UNREAL! My phone hasn't stopped buzzing. If you've been under a rock, here are some highlights in <90 sec 👇 🫀Most participants showed no or minimal progression of coronary plaque 🫀Neither ApoB nor LDL exposure predicted plaque progression 🫀But plaque predicted plaque progression What does this mean, in a phrase: "While this paper doesn't disprove the lipid heart hypothesis, it does help expose a major blindspot." There will be a LOT more coming soon... I promise you that... #Cholesterol #LDL #KetoDiet #ApoB #CAC #CCTA #LMHR #LEM #SaturatedFat #HeartHealth #MetabolicHealth cc Dave Feldman Dr Aseem Malhotra Joseph Everett (WIL) Jan Ellison Baszucki Metabolic Mind Also not, this is not a one off. There have been 10+ papers leading to this moment... and we are just getting warmed up... PMID: 40192608, Plaque Begets Plaque. ApoB does not: Longitudinal data from the KETO-CTA trial PMID: 39372369, Carbohydrate restriction-induced elevations in LDL-cholesterol and atherosclerosis: The KETO trial PMID: 38237807, Increased LDL cholesterol in adults with normal but not high body weight: a meta-analysis PMID: 35629964, The Lipid Energy Model: reimagining lipoprotein function in the context of carbohydrate-restricted diets PMID: 35106434, Elevated LDL-cholesterol with a carbohydrate-restricted diet: evidence for a “lean mass hyper-responder” phenotype

Nick Norwitz MD PhD

46,059 次观看 • 1 年前

🚨#OreoVsStatin - PUBLISHED!🚨 OREO Cookie cookies were 💥2X as potent💥 at lowering my LDL cholesterol (LDL-C) compared to high-intensity statin therapy! What you NEED to know... 👉This was a metabolic demonstration, a 'do not do this at home' experiment that makes no 'health' claims about cookies or cholesterol. 👉The purpose was to BOLDLY test a prediction of the Lipid Energy Model #LEM, i.e. that adding in carbs, including in the from of an Oreo cookies 'addition' (not carb-fat swap) would lower LDL-C in an LMHR on a #keto diet Why Oreos? ... It's (productively) provocative! 😱 Research on #cholesterol on #keto diets and the #LEM and #LMHR phenotype has so much to teach us about human lipid metabolism! ... But how do I (we) communicate the scientific 'awe' that we feel to the greatest number of people (as well as force a conversation about #LMHR and #LEM that needs to happen)? ... A dramatic demonstration designed to turn heads might work! It's not 'just click bait' ... it's 'legit-bait' I want YOU to feel the 'shock and awe' I feel every day studying this topic. And I want you to engage in the research journey. What you need to do... or should do... 🙏PRETTY PLEASE 🙏! 👉PLEASE WATCH 👀 VIDEO ABSTRACT here (<8 min): 👉 SHARE THIS LINK to the paper itself in a quote RT or original Tweet of your own: ... doing so helps promote the work and provoke the discussion ... and feel free to tag in your favorite influencers (or least favorite 😉)... A HUGE thanks to the senior author and expert lipidologist William Cromwell, MD for his guiding wisdom, insightful thoughts, and always open and curious mind. And to Dave Feldman, my friend, research wife, and the '#LEM daddy' Finally... there will be a LOT of coverage of this experiment. Keep in mind, we are not making any health claims, but through a dramatic demonstration, highlighting an uncomfortable tension that arises from a prediction of the Lipid Energy Model, and that is consistent with prior data (7+ papers, including 'highest tier' data like our recent meta-analysis of 41 RCTs). Science is supposed to embrace such uncomfortable questions... so, join us in being curious and advocating for much needed research on #LEM and #LMHR so we can make sense of what observations like this really mean... STAY CURIOUS 🤔
3:44

🚨#OreoVsStatin - PUBLISHED!🚨 OREO Cookie cookies were 💥2X as potent💥 at lowering my LDL cholesterol (LDL-C) compared to high-intensity statin therapy! What you NEED to know... 👉This was a metabolic demonstration, a 'do not do this at home' experiment that makes no 'health' claims about cookies or cholesterol. 👉The purpose was to BOLDLY test a prediction of the Lipid Energy Model #LEM, i.e. that adding in carbs, including in the from of an Oreo cookies 'addition' (not carb-fat swap) would lower LDL-C in an LMHR on a #keto diet Why Oreos? ... It's (productively) provocative! 😱 Research on #cholesterol on #keto diets and the #LEM and #LMHR phenotype has so much to teach us about human lipid metabolism! ... But how do I (we) communicate the scientific 'awe' that we feel to the greatest number of people (as well as force a conversation about #LMHR and #LEM that needs to happen)? ... A dramatic demonstration designed to turn heads might work! It's not 'just click bait' ... it's 'legit-bait' I want YOU to feel the 'shock and awe' I feel every day studying this topic. And I want you to engage in the research journey. What you need to do... or should do... 🙏PRETTY PLEASE 🙏! 👉PLEASE WATCH 👀 VIDEO ABSTRACT here (<8 min): 👉 SHARE THIS LINK to the paper itself in a quote RT or original Tweet of your own: ... doing so helps promote the work and provoke the discussion ... and feel free to tag in your favorite influencers (or least favorite 😉)... A HUGE thanks to the senior author and expert lipidologist William Cromwell, MD for his guiding wisdom, insightful thoughts, and always open and curious mind. And to Dave Feldman, my friend, research wife, and the '#LEM daddy' Finally... there will be a LOT of coverage of this experiment. Keep in mind, we are not making any health claims, but through a dramatic demonstration, highlighting an uncomfortable tension that arises from a prediction of the Lipid Energy Model, and that is consistent with prior data (7+ papers, including 'highest tier' data like our recent meta-analysis of 41 RCTs). Science is supposed to embrace such uncomfortable questions... so, join us in being curious and advocating for much needed research on #LEM and #LMHR so we can make sense of what observations like this really mean... STAY CURIOUS 🤔

Nick Norwitz MD PhD

3,217,928 次观看 • 2 年前

🚀Did your #LDL cholesterol sky-rocket on #keto? 🧈Were you told it' was because of Saturated Fat? 🚨NEW META OF 41 RCTs just busted that MYTH! 3 Main findings 👉 In studies with 'lean' BMI ( 35), LDL-C ⬇️ 💥BOOM, there's a money shot!💥 👉 Inverse association between BMI and LDL-C 👉 BMI dominates over saturated fat, which has comparatively minimal impact; thus, it's more likely having 6-pack abs is a severe risk factor for high LDL-C on #keto, not butter, beef, bacon or cream 🔗PAPER LINK 🔗 - (To share the data love, copy this link to a Quote tweet or original Tweet with your reaction. Please engage!) FULL Video Abstract (7 min): Major props to first-author Adrian Soto-Mota and senior Dr. David Ludwig. These man are humble & kind geniuses that I'm privileged to know and learn under And +100 pts for the #LEM ⚡️ Dave Feldman Of interest to More Plates More Dates Peter Attia @PlantChompers and Walter Willett (if he had a Twitter) Simon Hill MSc, BSc Thomas Dayspring William Cromwell, MD LipoFan Andrew D. Huberman, Ph.D. Dominic D'Agostino Layne Norton, PhD Metabolic Health Summit
3:06

🚀Did your #LDL cholesterol sky-rocket on #keto? 🧈Were you told it' was because of Saturated Fat? 🚨NEW META OF 41 RCTs just busted that MYTH! 3 Main findings 👉 In studies with 'lean' BMI ( 35), LDL-C ⬇️ 💥BOOM, there's a money shot!💥 👉 Inverse association between BMI and LDL-C 👉 BMI dominates over saturated fat, which has comparatively minimal impact; thus, it's more likely having 6-pack abs is a severe risk factor for high LDL-C on #keto, not butter, beef, bacon or cream 🔗PAPER LINK 🔗 - (To share the data love, copy this link to a Quote tweet or original Tweet with your reaction. Please engage!) FULL Video Abstract (7 min): Major props to first-author Adrian Soto-Mota and senior Dr. David Ludwig. These man are humble & kind geniuses that I'm privileged to know and learn under And +100 pts for the #LEM ⚡️ Dave Feldman Of interest to More Plates More Dates Peter Attia @PlantChompers and Walter Willett (if he had a Twitter) Simon Hill MSc, BSc Thomas Dayspring William Cromwell, MD LipoFan Andrew D. Huberman, Ph.D. Dominic D'Agostino Layne Norton, PhD Metabolic Health Summit

Nick Norwitz MD PhD

269,524 次观看 • 2 年前

"Statin Drugs Have Not Saved Millions Of Lives." Dr Jack Wolfson, Cardiologist "Statins Have Actually Killed Millions Of People." Low Cholesterol Causes 3X Increase In Brain Bleed Strokes. Every 30 mg Decrease In Cholesterol Causes A 22% Higher Risk Of Death From All Causes... Documented Peer Reviewed Data Shows: When LDL decreases below 70 mg/dL, hemorrhagic strokes (brain bleeds) increase 3 fold. The Minnesota Coronary Experiment showed that for every 30 mg/dL (0.78 mmol/L) decrease in serum cholesterol is linked with 22% higher risk of death from any cause. When total cholesterol is chemically lowered below 180 mg/dL the rate of cancer & dementia increases dramatically. And when total cholesterol stays under 150 mg/dL, chronic illnesses like cancer & autoimmune diseases strike. 36% of those on statin therapy will be diagnosed with new onset type 2 diabetes. Research has confirmed over & over that the higher your LDL the longer you live. LDL is a transport molecule for cholesterol & a vital immune system regulator. Lowering LDL thru statin drugs has deadly & deleterious effects. Vibrant Health Can Start To Be Attained When: Total Cholesterol is at minimum 200 mg/dL corresponding to a cholesterol level of 5.2 mmol/L in non US countries. Acceptable total cholesterol in 1960 was 350 mg/dL. Lowering cholesterol does not lower Cardiovascular risk or disease. Saturated fat & cholesterol have no effect on CVD outcomes, including heart attacks, strokes, CVD mortality & total mortality. There Are No Benefits To Cholesterol Lowering Statin Therapy..Only Risks & Side Effects: Alzheimer's Disease Cancer Pancreatitis Liver Damage Diabetes Depression/Suicidality Lupus Parkinson's Disease ALS (Lou Gehrig's Disease) Hormonal Deficiency Brain Damage Multiple Sclerosis Muscle Pain & Tearing Severe Weakness Neuropathy Heart Failure Vertigo Cognitive Impairment Dementia 👇Low LDL Causes 3X Hemorrhagic Strokes👇 👇Minnesota Coronary Experiment Buried Data👇 👇Low Cholesterol Causes Heart Disease & Cancer👇 👇The Higher Your LDL, The Longer You Live👇 Speaker: Dr Jack Wolfson, Cardiologist
1:30

"Statin Drugs Have Not Saved Millions Of Lives." Dr Jack Wolfson, Cardiologist "Statins Have Actually Killed Millions Of People." Low Cholesterol Causes 3X Increase In Brain Bleed Strokes. Every 30 mg Decrease In Cholesterol Causes A 22% Higher Risk Of Death From All Causes... Documented Peer Reviewed Data Shows: When LDL decreases below 70 mg/dL, hemorrhagic strokes (brain bleeds) increase 3 fold. The Minnesota Coronary Experiment showed that for every 30 mg/dL (0.78 mmol/L) decrease in serum cholesterol is linked with 22% higher risk of death from any cause. When total cholesterol is chemically lowered below 180 mg/dL the rate of cancer & dementia increases dramatically. And when total cholesterol stays under 150 mg/dL, chronic illnesses like cancer & autoimmune diseases strike. 36% of those on statin therapy will be diagnosed with new onset type 2 diabetes. Research has confirmed over & over that the higher your LDL the longer you live. LDL is a transport molecule for cholesterol & a vital immune system regulator. Lowering LDL thru statin drugs has deadly & deleterious effects. Vibrant Health Can Start To Be Attained When: Total Cholesterol is at minimum 200 mg/dL corresponding to a cholesterol level of 5.2 mmol/L in non US countries. Acceptable total cholesterol in 1960 was 350 mg/dL. Lowering cholesterol does not lower Cardiovascular risk or disease. Saturated fat & cholesterol have no effect on CVD outcomes, including heart attacks, strokes, CVD mortality & total mortality. There Are No Benefits To Cholesterol Lowering Statin Therapy..Only Risks & Side Effects: Alzheimer's Disease Cancer Pancreatitis Liver Damage Diabetes Depression/Suicidality Lupus Parkinson's Disease ALS (Lou Gehrig's Disease) Hormonal Deficiency Brain Damage Multiple Sclerosis Muscle Pain & Tearing Severe Weakness Neuropathy Heart Failure Vertigo Cognitive Impairment Dementia 👇Low LDL Causes 3X Hemorrhagic Strokes👇 👇Minnesota Coronary Experiment Buried Data👇 👇Low Cholesterol Causes Heart Disease & Cancer👇 👇The Higher Your LDL, The Longer You Live👇 Speaker: Dr Jack Wolfson, Cardiologist

Valerie Anne Smith

429,376 次观看 • 11 个月前

"Statin Drugs Have Not Saved Millions Of Lives." Dr. Jack Wolfson, Cardiologist "Statins have actually killed millions of people." Low cholesterol causes 3X increase in brain bleed strokes. Every 30 mg decrease in Cholesterol causes a 22% higher risk of death from all causes... Documented Peer Reviewed Data Shows: When LDL decreases below 70 mg/dL, hemorrhagic strokes (brain bleeds) increase 3 fold. The Minnesota Coronary Experiment showed that for every 30 mg/dL (0.78 mmol/L) decrease in serum cholesterol is linked with 22% higher risk of death from any cause. When total cholesterol is chemically lowered below 180 mg/dL the rate of cancer & dementia increases dramatically. And when total cholesterol stays under 150 mg/dL, chronic illnesses like cancer & autoimmune diseases strike. 36% of those on statin therapy will be diagnosed with new onset type 2 diabetes. Research has confirmed over & over that the higher your LDL the longer you live. LDL is a transport molecule for cholesterol & a vital immune system regulator. Lowering LDL thru statin drugs has deadly & deleterious effects. Vibrant Health Can Start To Be Attained When: Total Cholesterol is at minimum 200 mg/dL corresponding to a cholesterol level of 5.2 mmol/L in non US countries. Normal total cholesterol in 1960 was 300-350 mg/dL. Lowering cholesterol does not lower Cardiovascular risk or disease. Saturated fat & cholesterol have no effect on CVD outcomes, including heart attacks, strokes, CVD mortality & total mortality. There Are No Benefits To Cholesterol Lowering Statin Therapy..Only Risks & Side Effects: Alzheimer's Disease Cancer Pancreatitis Liver Damage Diabetes Depression/Suicidality Lupus Parkinson's Disease ALS (Lou Gehrig's Disease) Hormonal Deficiency Brain Damage Multiple Sclerosis Muscle Pain & Tearing Severe Weakness Neuropathy Heart Failure Vertigo Cognitive Impairment Dementia
1:35

"Statin Drugs Have Not Saved Millions Of Lives." Dr. Jack Wolfson, Cardiologist "Statins have actually killed millions of people." Low cholesterol causes 3X increase in brain bleed strokes. Every 30 mg decrease in Cholesterol causes a 22% higher risk of death from all causes... Documented Peer Reviewed Data Shows: When LDL decreases below 70 mg/dL, hemorrhagic strokes (brain bleeds) increase 3 fold. The Minnesota Coronary Experiment showed that for every 30 mg/dL (0.78 mmol/L) decrease in serum cholesterol is linked with 22% higher risk of death from any cause. When total cholesterol is chemically lowered below 180 mg/dL the rate of cancer & dementia increases dramatically. And when total cholesterol stays under 150 mg/dL, chronic illnesses like cancer & autoimmune diseases strike. 36% of those on statin therapy will be diagnosed with new onset type 2 diabetes. Research has confirmed over & over that the higher your LDL the longer you live. LDL is a transport molecule for cholesterol & a vital immune system regulator. Lowering LDL thru statin drugs has deadly & deleterious effects. Vibrant Health Can Start To Be Attained When: Total Cholesterol is at minimum 200 mg/dL corresponding to a cholesterol level of 5.2 mmol/L in non US countries. Normal total cholesterol in 1960 was 300-350 mg/dL. Lowering cholesterol does not lower Cardiovascular risk or disease. Saturated fat & cholesterol have no effect on CVD outcomes, including heart attacks, strokes, CVD mortality & total mortality. There Are No Benefits To Cholesterol Lowering Statin Therapy..Only Risks & Side Effects: Alzheimer's Disease Cancer Pancreatitis Liver Damage Diabetes Depression/Suicidality Lupus Parkinson's Disease ALS (Lou Gehrig's Disease) Hormonal Deficiency Brain Damage Multiple Sclerosis Muscle Pain & Tearing Severe Weakness Neuropathy Heart Failure Vertigo Cognitive Impairment Dementia

Valerie Anne Smith

228,090 次观看 • 14 天前

What if everything we thought we knew about cholesterol and heart disease risk… doesn’t apply to everyone? In this episode, world-renowned cardiologist Dr. Matthew Budoff unpacks the results of a landmark one-year study tracking 100 lean, metabolically healthy individuals on a ketogenic diet with extremely elevated LDL levels. Dr. Budoff is the Program Director, Director of Cardiac CT, and the endowed chair of preventive cardiology at The Lundquist Institute for Biomedical Innovation at Harbor-UCLA Medical Center. In this interview, Dr. Scher and Dr. Budoff further break down the results of his new publication, which used advanced imaging to demonstrate that LDL cholesterol and ApoB levels are not associated with plaque progression in Lean-Mass Hyper Responders following a #ketogenicdiet. 📊 Surprising insights: • Elevated LDL and ApoB did not predict plaque progression • Some participants with LDLs over 500 showed no plaque at all • A few participants even experienced plaque regression • Existing plaque—not LDL-C or ApoB—did predict plaque accumulation in this population Dr. Budoff explains what these results mean for clinicians, for patients using ketogenic therapy as a medical intervention, and for the broader conversation around cardiovascular disease risk. “It is important that clinicians, along with the general public, are made aware that personalized, data-driven approaches to assessing risk should be considered based on individual conditions,” said Dr. Budoff. “The existence of this phenotype suggests that alternative markers or tests should be used to establish metabolic health in some cases.” 🎬 These exciting new findings are featured in Dave Feldman and Jen Isenhart’s upcoming documentary, The Cholesterol Code, the story of how a software engineer conducts a groundbreaking study on an unusual group of people—lean, healthy individuals whose doctors are convinced they’ll die young. Real stories of healing with ketogenic diets provide a blueprint for using food as powerful medicine. Visit to learn more about the film and to be the first to hear about private screenings and the general release in the fall. Expert Featured: Dr. Matthew Budoff Matthew Budoff MD Resources Mentioned: Plaque Begets Plaque, ApoB Does Not Diagnostic and Preventative Cardiovascular Imaging Center CMEs Mentioned: Managing Major Mental Illness with Dietary Change: The New Science of Hope Brain Energy: The Metabolic Theory of Mental Illness Learn more about metabolic psychiatry and find helpful resources at
19:42

What if everything we thought we knew about cholesterol and heart disease risk… doesn’t apply to everyone? In this episode, world-renowned cardiologist Dr. Matthew Budoff unpacks the results of a landmark one-year study tracking 100 lean, metabolically healthy individuals on a ketogenic diet with extremely elevated LDL levels. Dr. Budoff is the Program Director, Director of Cardiac CT, and the endowed chair of preventive cardiology at The Lundquist Institute for Biomedical Innovation at Harbor-UCLA Medical Center. In this interview, Dr. Scher and Dr. Budoff further break down the results of his new publication, which used advanced imaging to demonstrate that LDL cholesterol and ApoB levels are not associated with plaque progression in Lean-Mass Hyper Responders following a #ketogenicdiet. 📊 Surprising insights: • Elevated LDL and ApoB did not predict plaque progression • Some participants with LDLs over 500 showed no plaque at all • A few participants even experienced plaque regression • Existing plaque—not LDL-C or ApoB—did predict plaque accumulation in this population Dr. Budoff explains what these results mean for clinicians, for patients using ketogenic therapy as a medical intervention, and for the broader conversation around cardiovascular disease risk. “It is important that clinicians, along with the general public, are made aware that personalized, data-driven approaches to assessing risk should be considered based on individual conditions,” said Dr. Budoff. “The existence of this phenotype suggests that alternative markers or tests should be used to establish metabolic health in some cases.” 🎬 These exciting new findings are featured in Dave Feldman and Jen Isenhart’s upcoming documentary, The Cholesterol Code, the story of how a software engineer conducts a groundbreaking study on an unusual group of people—lean, healthy individuals whose doctors are convinced they’ll die young. Real stories of healing with ketogenic diets provide a blueprint for using food as powerful medicine. Visit to learn more about the film and to be the first to hear about private screenings and the general release in the fall. Expert Featured: Dr. Matthew Budoff Matthew Budoff MD Resources Mentioned: Plaque Begets Plaque, ApoB Does Not Diagnostic and Preventative Cardiovascular Imaging Center CMEs Mentioned: Managing Major Mental Illness with Dietary Change: The New Science of Hope Brain Energy: The Metabolic Theory of Mental Illness Learn more about metabolic psychiatry and find helpful resources at

Metabolic Mind

10,423 次观看 • 1 年前

🚨This is a new low🚨🚨 An Irish mother from Dublin was sent to Belfast to have her baby by C-section to be told there were no incubators in the whole of Ireland on the 20th of March. The mother now has been told now that she cannot register the baby for an Irish birth cert, that the baby is now classed as a UK citizen and has to go back to belfast to get a birth cert.
1:50

🚨This is a new low🚨🚨 An Irish mother from Dublin was sent to Belfast to have her baby by C-section to be told there were no incubators in the whole of Ireland on the 20th of March. The mother now has been told now that she cannot register the baby for an Irish birth cert, that the baby is now classed as a UK citizen and has to go back to belfast to get a birth cert.

Cllr Gavin Pepper

221,889 次观看 • 1 年前

Cardiology has become obsessed with the mantra “lower is better.” Lower LDL, lower ApoB, and you're going to be better off. This simple tagline misses the forest for a stupid little cholesterol shrub. It’s well known that intervening to lower LDL and ApoB doesn’t necessarily result in better overall outcomes for the whole person, even in circumstances where cardiovascular risk might be reduced. It's therefore incredibly sad when "certain cardiologist experts" (heavy quotes there) make false claims like: "people with a PCSK9 loss of function mutation have very low LDL-C and live 15-20 years longer than those who don't. And we have meds now that cause that effect." That nonsense I could forgive. What is far more troubling is that there's a general failure to challange problematic (and sometimes outright false) narratives like this provided they align with the status quo, even when the status quo has failed public health miserably. We need to broaden the aperture of the discussion because the "science is not settled." Below is an excerpt from this morning video on the lipid marker linked to longevity (not LDL or ApoB): 🚨Then, of course, watch The Cholesterol Code movie on Amazon and leave a review! #CholesterolCodeMovie Dave Feldman
1:22

Cardiology has become obsessed with the mantra “lower is better.” Lower LDL, lower ApoB, and you're going to be better off. This simple tagline misses the forest for a stupid little cholesterol shrub. It’s well known that intervening to lower LDL and ApoB doesn’t necessarily result in better overall outcomes for the whole person, even in circumstances where cardiovascular risk might be reduced. It's therefore incredibly sad when "certain cardiologist experts" (heavy quotes there) make false claims like: "people with a PCSK9 loss of function mutation have very low LDL-C and live 15-20 years longer than those who don't. And we have meds now that cause that effect." That nonsense I could forgive. What is far more troubling is that there's a general failure to challange problematic (and sometimes outright false) narratives like this provided they align with the status quo, even when the status quo has failed public health miserably. We need to broaden the aperture of the discussion because the "science is not settled." Below is an excerpt from this morning video on the lipid marker linked to longevity (not LDL or ApoB): 🚨Then, of course, watch The Cholesterol Code movie on Amazon and leave a review! #CholesterolCodeMovie Dave Feldman

Nick Norwitz MD PhD

56,203 次观看 • 4 个月前

"Acceptable" LDL Cholesterol Levels Used To Be 250 mg/dl. This Arbitrary Number Was Chosen By Big Pharma To Sell Statins. Dr Stephen Hussey Now, "Acceptable" Levels Must Be Below 100 mg/dl. Big Pharma Keeps Moving The Goalpost To Prescribe More Statins. Cholesterol Does Not Cause Heart Disease & Statins Have Never Been Proven To PREVENT Heart Disease. The Great Cholesterol Con Is Crumbling Down... Breaking New Study Reveals 17.3 Million Adults Do Not Need Statins, Including 4.1 Million Currently Taking Them. In 1984, one committee heavily influenced by Big Pharma decided, based on NO research or evidence, that LDL Cholesterol was bad & should be 250 mg/dl or lower. Over the years, the pharmaceutical industry used their power & leverage to lower these levels again based on NO research or evidence. They changed the guideline again in order to prescribe more Statins & now LDL Cholesterol must be kept below 100 mg/dl. No one knows what ideal Cholesterol levels are & there are no "best levels" at all. The entire idea that Cholesterol is bad came from very low quality associational studies & the number was basically made up. And then that arbitrary number was lowered every few years to make more profit from Statins. The lower Big Pharma can make the Cholesterol level guidelines, the more drugs are prescribed & sold. Making the Statin industry one of the biggest selling medicines worldwide accounting for an annual profit of $19 Billion & growing higher each year. We now know that Metabolic Dysfunction & Inflammation cause cardiovascular disease, heart attack, atherosclerosis & stroke. Which has given rise to Big Pharma once again profiting thru "Metabolic Drugs" like Ozempic, which are being pushed heavily & causing grave side effects for consumers. Medications are never going to be the answer for why this world is so sick. The only way to optimize vibrant health is for each person to make drastic lifestyle changes: 1: Nutrient Dense Animal Foods 2: Single Ingredient Whole Foods 3: Sun Exposure 4: Optimizing Circadian Rhythm 5: Avoid Toxin Exposure 6: Grounding/Direct Earth Contact 7: Fasting/Time Restricted Eating *⃣This means eliminating seed oils, eliminating ultra processed foods, no high carbohydrate diet, no added sugars*⃣ 👇2024 Statins Provide No Primary Benefit👇 👇The Higher Your LDL, The Longer You Live👇 👇The Keto Trial: LDL Rise Is Normal & Expected👇 Video Speaker: Dr. Stephen Hussey, DC
3:12

"Acceptable" LDL Cholesterol Levels Used To Be 250 mg/dl. This Arbitrary Number Was Chosen By Big Pharma To Sell Statins. Dr Stephen Hussey Now, "Acceptable" Levels Must Be Below 100 mg/dl. Big Pharma Keeps Moving The Goalpost To Prescribe More Statins. Cholesterol Does Not Cause Heart Disease & Statins Have Never Been Proven To PREVENT Heart Disease. The Great Cholesterol Con Is Crumbling Down... Breaking New Study Reveals 17.3 Million Adults Do Not Need Statins, Including 4.1 Million Currently Taking Them. In 1984, one committee heavily influenced by Big Pharma decided, based on NO research or evidence, that LDL Cholesterol was bad & should be 250 mg/dl or lower. Over the years, the pharmaceutical industry used their power & leverage to lower these levels again based on NO research or evidence. They changed the guideline again in order to prescribe more Statins & now LDL Cholesterol must be kept below 100 mg/dl. No one knows what ideal Cholesterol levels are & there are no "best levels" at all. The entire idea that Cholesterol is bad came from very low quality associational studies & the number was basically made up. And then that arbitrary number was lowered every few years to make more profit from Statins. The lower Big Pharma can make the Cholesterol level guidelines, the more drugs are prescribed & sold. Making the Statin industry one of the biggest selling medicines worldwide accounting for an annual profit of $19 Billion & growing higher each year. We now know that Metabolic Dysfunction & Inflammation cause cardiovascular disease, heart attack, atherosclerosis & stroke. Which has given rise to Big Pharma once again profiting thru "Metabolic Drugs" like Ozempic, which are being pushed heavily & causing grave side effects for consumers. Medications are never going to be the answer for why this world is so sick. The only way to optimize vibrant health is for each person to make drastic lifestyle changes: 1: Nutrient Dense Animal Foods 2: Single Ingredient Whole Foods 3: Sun Exposure 4: Optimizing Circadian Rhythm 5: Avoid Toxin Exposure 6: Grounding/Direct Earth Contact 7: Fasting/Time Restricted Eating *⃣This means eliminating seed oils, eliminating ultra processed foods, no high carbohydrate diet, no added sugars*⃣ 👇2024 Statins Provide No Primary Benefit👇 👇The Higher Your LDL, The Longer You Live👇 👇The Keto Trial: LDL Rise Is Normal & Expected👇 Video Speaker: Dr. Stephen Hussey, DC

Valerie Anne Smith

179,827 次观看 • 1 年前

🤒😭Unfortunately I am very unwell, my friends. I have not been feeling my best the past couple of days, and I have been growing more tired every minute. Last night was miserable, I was in so much pain I could not sleep, the pain in my head and in my body was causing me hallucinations and I felt as though I would die. This morning was not any better and I was so dizzy I could not see properly, and I felt so tired I could not describe it. I have just returned from the hospital, my friends, where I was able to receive a treatment to help me feel only a little better. I am still so exhausted, not only physically but mentally too. The fear of famine and death in a horrific way is still on my mind, even through this terrible pain. I'd be very grateful if you could pray for us all, for a better situation ahead, and for an end to this cruelty against us. Please support us and continue to help us via our fundraisers so we can continue to distribute aid, my friends. Your support for me and my friends gives me great hope in these miserable times, and I hope you will continue to do so..🫂 🙏🚨 🫂😢 🐾🙏 🫂 🚨🙏 🐾🐈
0:31

🤒😭Unfortunately I am very unwell, my friends. I have not been feeling my best the past couple of days, and I have been growing more tired every minute. Last night was miserable, I was in so much pain I could not sleep, the pain in my head and in my body was causing me hallucinations and I felt as though I would die. This morning was not any better and I was so dizzy I could not see properly, and I felt so tired I could not describe it. I have just returned from the hospital, my friends, where I was able to receive a treatment to help me feel only a little better. I am still so exhausted, not only physically but mentally too. The fear of famine and death in a horrific way is still on my mind, even through this terrible pain. I'd be very grateful if you could pray for us all, for a better situation ahead, and for an end to this cruelty against us. Please support us and continue to help us via our fundraisers so we can continue to distribute aid, my friends. Your support for me and my friends gives me great hope in these miserable times, and I hope you will continue to do so..🫂 🙏🚨 🫂😢 🐾🙏 🫂 🚨🙏 🐾🐈

♥️🇵🇸🇵🇸❤️A lover ♥️of homeless cats😻❤️🌱🌱👍🤗

17,937 次观看 • 1 年前