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Long-term “sting in the tail.” Spike protein creates amyloid— can’t be broken down and leads to neuro-degenerative disorders. To help the injured, we have to remove the amyloid. Not easy. Kevin W. McCairn PhD Lyndsey, RN 💜🐭
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![We talk a lot about the COVID jabs causing cancer, blood clots, myocarditis, etc. But don't forget about (spreadable) prion diseases! "As a scientist, I'm having to listen very carefully to the eugenicists and those people who dream of a far lower number of human beings...." Dr. Kevin McCairn, PhD (Kevin W. McCairn PhD), a retired Principal Investigator at the Korea Brain Research Institute Systems Neuroscience and Movement Disorders Laboratory, describes during a discussion posted by Health Alliance Australia how the spike protein of both SARS-CoV-2 and the spike coded by the mRNA COVID injections is an engineered prion. (For reference, a prion is a misfolded protein that causes other proteins in the brain to misfold, leading to fatal brain diseases.) Health Alliance Australia notes in its description of this interview that these "weaponized prions" have "grave implications for humanity because [they] are far more dangerous than viruses." Health Alliance Australia adds in its description that "The most commonly known prion disease is CJD or Creutzfeldt-Jakob disease, which is a spongiform encephalopathy where the brain becomes spongy, wastes away, and ultimately leads to a horrible death. Alzheimer’s and Parkinson’s are other neurodegenerative diseases associated with prions." Considering the deployment of these "weaponized prions," McCairn says that "as a scientist, I'm having to listen very carefully to the eugenicists and those people who dream of a far lower number of human beings on this planet." The scientist adds, "if you wanted a method to do that, well, you're going to struggle through normal viral or bioweapon means... whereas the spread of prion within a population would be very, very difficult to detect unless you are specifically looking for these misfolded proteins." ---------------Partial transcription of clip-------------- "So SARS CoV-2 spike amyloid fibrils specifically and selectively accelerates amyloid fibril formation of, and this is the critical part, human prion protein and the amyloid beta peptide. And this means that we have to take a very, very long hard look at what this means with respect to biowarfare strategies and techniques because prions have been a subject of biowarfare research for a long time. "The problem was that you could aerosolize prion, the protein, and it's lethal in that respect. But the spreading is limited. And but as I mentioned earlier, as they focused on the weaponization and contraction, honing down the peptide sequences to their most essential disease causing elements, I think that they've been focusing on how to find a way to get prion to spread person to person, not directly with even though we consider prion disorder, what's called TSEs, transmissible spongiform encephalopathies. Meaning, we know that if you come into contact with them, ingest them, there's a high chance that you'll develop the prion disease and you'll have a bad outcome, meaning invariably death. Horrible death at that. A horrible death. It's not pretty. "So, I'm just gonna read out the part that I've got highlighted here. So they say, we here provide evidence of significant spike amyloid fibril seeded acceleration of amyloid formation of CJD associated human prion protein using an in vitro conversion assay. So what that means is that just it's not in cell test systems yet. It's just they're doing the molecular biochemistry in a manner in which they can detect change over time. Right? And so you basically take this epitope sequence. And so they say, we showed that the amyloid fibril formation of, in this case, Alzheimer associated amyloid beta-142 was accelerated by spike amyloid fibrils of 7 different 20 amino acid long peptides, spike 532 to 551 meaning just this 20 amino acid long sequence, was the most efficient at seeding human prion protein, whilst another segment, 601 to 620, was most effective at seeding amyloid beta. "Now if you just look at the data from their experiment, the fastest reaction that they have is with the conversion to the human prion, the scrapy form, meaning the diseased form of the prion protein in the data set, which is just this cluster down here, Spike532 seed. And so when we see a signal like this on top of all the other data points that we've seen over the last 4 years, it's incumbent upon us to think, have these people through malfeasance, perhaps perhaps it was just a research project, gone [wrong] or at this stage. It's it's difficult to say for certain it's these groups. "But, I think, as a scientist, I'm having to listen very carefully to the eugenicists and those people who dream of a far lower number of human beings on this planet. And if you wanted a method to do that, well, you're going to struggle through normal viral or bioweapon means, right? So you could go for the bacterial route, but there's likely we'd develop countermeasures in that respect. We can deal with them and the onset of sickness, etcetera, has very typical presentations, whereas the spread of prion within a population would be very, very difficult to detect unless you are specifically looking for these misfolded proteins. "And then remember, we're in an we're in an area where the it's more complex than just like for like conversion. We now have to operate in a world of what's called cross-seeding amyloidogenic peptide seeds, meaning these small, small epitopes."](https://image.24vids.com/tw-1905654323083673746/ext_tw_video_thumb/1905647043936153600/pu/img/UeMN-cQe-Z0AP2Wi.jpg)
