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Tomorrow on the #FeldmanProtocol The Low Carb OG, Dr. Eric Westman, MD, MHS We discuss: 🍳 Atkins Origins 🧠 Cholesterol Myths 🧑‍⚕️ Westman’s Journey 📊 Keto Research 🍖 Low-Carb Evolution 🥓 Net vs Total Carbs ☕ Bulletproof Fads 💉 Drugs vs Diet 🍪 Sugar Addiction ... and much more

Dave Feldman

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Last night Dr David Unwin was on mainstream news talking about the benefits of reducing carbs for those living with diabetes. The results he gets are amazing. I know it can be replicated by others because I copy what he does in my own clinical practice using David Oliver, Freshwell resources (which are free btw). As a result, in 2024 forty of my patients achieved remission too. The news report featured an endocrinologist Shivani Misra who, at the 4mins 27s timestamp in the video, said: "If someone does a low carb with higher fat what does that mean for their cholesterol and their cardiovascular disease risk? We don't know the answer to this" I'm here to tell you this is a false statement. The research has been done. Let me show you: In 2020 a meta analysis looking at the effects of low carb on CVD risk found: "For total cholesterol there was no significant change in the data corresponding to low-carbohydrate diets lasting 12–23 months and over 24 months" With regards LDL the meta analysis says: "For plasma LDL, as the forest map shows, that there was no significant difference between the low-carbohydrate diet group and the control group at 6–11 months, 12–23 months, and 24 months" All other factors improved (blood pressure, triglycerides, etc) The meta analysis concluded: "In conclusion, the overall effect of a low-carbohydrate diet on cardiovascular risk factors tended to be favorable at less than 6 months and 6–11 months, but after 2 years of a low-carbohydrate diet, there was no significant effect on cardiovascular risk factors" So short term: CVD risk factors are improved, long term, things don't get worse". This mirrors what I see in clinical practice and with myself having been low carb full time since the start of 2020 - all my CVD risk markers are in the normal range. Study source: The reporter also talks about low carb as a "restrictive diet". What's more restrictive: Giving up bagels, bread, sweets and other junk IN FAVOUR OF protein, vegetables etc Or Giving up solid food entirely for a 850kcal liquid diet for months? Despite the latter being far more restrictive it has been rolled out nationally by the NHS. Nothing wrong with this as it works but so does low carb. Why not give patients a choice? The reporter also says Dr Unwins results are just because of the support he provides. Whilst support matters (a lot), this is blatant misinformation. Plenty of studies show that reducing carbs is disproportionately better for those with diabetes than low fat. Here are some: In 2023 Novo Nordisk published a randomized controlled trial comparing low carb to low fat for diabetes. They found: 🩸 Low carb led to the greatest reduction in hba1c 💉 Only the low carb group reduced medications 📉 Low carb had the greatest reduction in triglycerides + higher HDL (LDL was similar) ⚖️ Low carb group lost more weight + more fat spontaneously despite eating more calories 🩸 Systolic blood pressure was lower for low carb The low carb group non significantly raises their LDL but 0.23mmol. The researchers said: "we consider the beneficial effects of low carb to outweigh the minor increase in LDL (0.23mmol) induced by the diet. This is supported by other studies" So low carb is better and CVD risk markers overall improved. Naturally, the study was hidden behind a Paywall. Link: I can hear the skeptics now: "But Mike, this is one study, it's not enough" Ok here's a meta analysis showing that hba1c is directly proportional to the carbs eaten: This meta analysis concluded:

Last night Dr David Unwin was on mainstream news talking about the benefits of reducing carbs for those living with diabetes. The results he gets are amazing. I know it can be replicated by others because I copy what he does in my own clinical practice using David Oliver, Freshwell resources (which are free btw). As a result, in 2024 forty of my patients achieved remission too. The news report featured an endocrinologist Shivani Misra who, at the 4mins 27s timestamp in the video, said: "If someone does a low carb with higher fat what does that mean for their cholesterol and their cardiovascular disease risk? We don't know the answer to this" I'm here to tell you this is a false statement. The research has been done. Let me show you: In 2020 a meta analysis looking at the effects of low carb on CVD risk found: "For total cholesterol there was no significant change in the data corresponding to low-carbohydrate diets lasting 12–23 months and over 24 months" With regards LDL the meta analysis says: "For plasma LDL, as the forest map shows, that there was no significant difference between the low-carbohydrate diet group and the control group at 6–11 months, 12–23 months, and 24 months" All other factors improved (blood pressure, triglycerides, etc) The meta analysis concluded: "In conclusion, the overall effect of a low-carbohydrate diet on cardiovascular risk factors tended to be favorable at less than 6 months and 6–11 months, but after 2 years of a low-carbohydrate diet, there was no significant effect on cardiovascular risk factors" So short term: CVD risk factors are improved, long term, things don't get worse". This mirrors what I see in clinical practice and with myself having been low carb full time since the start of 2020 - all my CVD risk markers are in the normal range. Study source: The reporter also talks about low carb as a "restrictive diet". What's more restrictive: Giving up bagels, bread, sweets and other junk IN FAVOUR OF protein, vegetables etc Or Giving up solid food entirely for a 850kcal liquid diet for months? Despite the latter being far more restrictive it has been rolled out nationally by the NHS. Nothing wrong with this as it works but so does low carb. Why not give patients a choice? The reporter also says Dr Unwins results are just because of the support he provides. Whilst support matters (a lot), this is blatant misinformation. Plenty of studies show that reducing carbs is disproportionately better for those with diabetes than low fat. Here are some: In 2023 Novo Nordisk published a randomized controlled trial comparing low carb to low fat for diabetes. They found: 🩸 Low carb led to the greatest reduction in hba1c 💉 Only the low carb group reduced medications 📉 Low carb had the greatest reduction in triglycerides + higher HDL (LDL was similar) ⚖️ Low carb group lost more weight + more fat spontaneously despite eating more calories 🩸 Systolic blood pressure was lower for low carb The low carb group non significantly raises their LDL but 0.23mmol. The researchers said: "we consider the beneficial effects of low carb to outweigh the minor increase in LDL (0.23mmol) induced by the diet. This is supported by other studies" So low carb is better and CVD risk markers overall improved. Naturally, the study was hidden behind a Paywall. Link: I can hear the skeptics now: "But Mike, this is one study, it's not enough" Ok here's a meta analysis showing that hba1c is directly proportional to the carbs eaten: This meta analysis concluded:

Mike - Low Carb Dietitian

17,028 görüntüleme • 1 yıl önce

🚨New science DEBATE released on HIGH CARB vs KETO: What the new Tim Noakes vs Louise Burke debate actually shows 👇 American Journal of Clinical Nutrition just released it big debate between two of the biggest names in exercise performance asking one loaded question: does a low-carbohydrate diet impair endurance performance? On one side is Louise Burke, one of the most influential sports nutrition researchers in the world. Her argument: yes, low carb can impair performance? She is not saying every athlete must eat high-carb all the time. She is saying performance depends on the event, intensity, environment, training status, sex, and the athlete in front of you. Her biggest physiological point is fuel economy. Near an athlete’s oxidative ceiling (i.e., elite race pace), she argues that carbohydrate produces more usable energy per liter of oxygen than fat. So even if keto adaptation dramatically raises fat oxidation, that does not automatically mean better speed or power. Now I am skeptical of this point, since we have conducted Randomized Controlled Trails in both runners and ironman competitors and showed that athletes at 86% of their VO2max can utilize fat as the predominant fuel while on a very low carb diet and STILL maintain performance. On the other side, Tim Noakes argues: low carb does not necessarily impair performance when athletes are adapted long enough. He points out that many “keto hurts performance” studies are short, often under four weeks, and that several randomized trials lasting four to six weeks report similar performance between high-carb and low-carb groups. He also reframes fatigue. Instead of saying muscle glycogen is always the main limiter, he argues that during prolonged exercise, maintaining blood glucose, the small glucose pool is the most important. In our trial, 10 grams of carbohydrate per hour improved prolonged cycling performance after both diets. Noakes and Burke also did something really special: they developed a CONSENSUS article where they explored key points of AGREEMENT and DISAGREEMENT for future research. This is the coolest part and in my opinion was one of the verty unique aspect of these three studies My take is this: It is not "team high carb" versus "team keto." The weight of the evidence demonstrates that 1) BOTH high and low carb diets can work, and that 2) Carbohydrates during prolonged exercise are valuable...but 3) How this is applied depends on the individual athlete. What's your take?

🚨New science DEBATE released on HIGH CARB vs KETO: What the new Tim Noakes vs Louise Burke debate actually shows 👇 American Journal of Clinical Nutrition just released it big debate between two of the biggest names in exercise performance asking one loaded question: does a low-carbohydrate diet impair endurance performance? On one side is Louise Burke, one of the most influential sports nutrition researchers in the world. Her argument: yes, low carb can impair performance? She is not saying every athlete must eat high-carb all the time. She is saying performance depends on the event, intensity, environment, training status, sex, and the athlete in front of you. Her biggest physiological point is fuel economy. Near an athlete’s oxidative ceiling (i.e., elite race pace), she argues that carbohydrate produces more usable energy per liter of oxygen than fat. So even if keto adaptation dramatically raises fat oxidation, that does not automatically mean better speed or power. Now I am skeptical of this point, since we have conducted Randomized Controlled Trails in both runners and ironman competitors and showed that athletes at 86% of their VO2max can utilize fat as the predominant fuel while on a very low carb diet and STILL maintain performance. On the other side, Tim Noakes argues: low carb does not necessarily impair performance when athletes are adapted long enough. He points out that many “keto hurts performance” studies are short, often under four weeks, and that several randomized trials lasting four to six weeks report similar performance between high-carb and low-carb groups. He also reframes fatigue. Instead of saying muscle glycogen is always the main limiter, he argues that during prolonged exercise, maintaining blood glucose, the small glucose pool is the most important. In our trial, 10 grams of carbohydrate per hour improved prolonged cycling performance after both diets. Noakes and Burke also did something really special: they developed a CONSENSUS article where they explored key points of AGREEMENT and DISAGREEMENT for future research. This is the coolest part and in my opinion was one of the verty unique aspect of these three studies My take is this: It is not "team high carb" versus "team keto." The weight of the evidence demonstrates that 1) BOTH high and low carb diets can work, and that 2) Carbohydrates during prolonged exercise are valuable...but 3) How this is applied depends on the individual athlete. What's your take?

Andrew Koutnik, Ph.D.

15,181 görüntüleme • 4 ay önce

Dr. Thomas Seyfried says "The higher your blood sugar, the faster the tumor grows. The lower your blood sugar, the slower the tumor grows. Undeniable." He says the way to manage cancer without toxicity is to simultaneously restrict the two fuels cancer needs to grow: Glucose and Glutamine. The good news is there's a fuel that we can use very efficiently and cancer cannot: ketones. How do you make ketones? Consume little or no carbs and fast. Glutamine can be managed with drugs. To be fair, mainstream oncology disputes Seyfried's metabolic approach, instead relying on expensive chemotherapy that can run $100,000+ per year instead of glutamine restriction, a low carb diet and fasting.

Dr. Thomas Seyfried says "The higher your blood sugar, the faster the tumor grows. The lower your blood sugar, the slower the tumor grows. Undeniable." He says the way to manage cancer without toxicity is to simultaneously restrict the two fuels cancer needs to grow: Glucose and Glutamine. The good news is there's a fuel that we can use very efficiently and cancer cannot: ketones. How do you make ketones? Consume little or no carbs and fast. Glutamine can be managed with drugs. To be fair, mainstream oncology disputes Seyfried's metabolic approach, instead relying on expensive chemotherapy that can run $100,000+ per year instead of glutamine restriction, a low carb diet and fasting.

Cary Kelly

27,219 görüntüleme • 5 ay önce

A 33-year-old cardiologist named Robert Atkins was overweight and couldn't lose the weight. Nothing was working. Then he found a 1958 paper by Alfred Pennington — a DuPont company doctor who'd put the company's executives on unlimited meat and fat, zero carbs, and watched them lose weight without ever being hungry. Atkins tried it on himself. Lost the weight. Put his patients on it. They lost weight too. In 1972 he wrote Dr. Atkins' Diet Revolution — eat bacon, eggs, steak, butter. Cut the bread. 10 million copies. The establishment lost its mind. In 1973, the AMA Council on Foods and Nutrition published a formal takedown in JAMA calling his work "bizarre concepts of nutrition and dieting." Congress hauled him in. He was mocked in the press for the next 30 years. Then the science caught up. May 2003. New England Journal of Medicine. Two randomized trials. HDL up 18% vs. 3%. Triglycerides down 28% vs. 1%. Weight loss at six months: greater on Atkins. He was vindicated. He died six weeks later. Today, ketogenic diets reverse type 2 diabetes. They treat drug-resistant epilepsy. The ADA lists low-carb among its evidence-based dietary options. They called him a quack for 30 years. He was right the whole time. No sugar. No grains.

A 33-year-old cardiologist named Robert Atkins was overweight and couldn't lose the weight. Nothing was working. Then he found a 1958 paper by Alfred Pennington — a DuPont company doctor who'd put the company's executives on unlimited meat and fat, zero carbs, and watched them lose weight without ever being hungry. Atkins tried it on himself. Lost the weight. Put his patients on it. They lost weight too. In 1972 he wrote Dr. Atkins' Diet Revolution — eat bacon, eggs, steak, butter. Cut the bread. 10 million copies. The establishment lost its mind. In 1973, the AMA Council on Foods and Nutrition published a formal takedown in JAMA calling his work "bizarre concepts of nutrition and dieting." Congress hauled him in. He was mocked in the press for the next 30 years. Then the science caught up. May 2003. New England Journal of Medicine. Two randomized trials. HDL up 18% vs. 3%. Triglycerides down 28% vs. 1%. Weight loss at six months: greater on Atkins. He was vindicated. He died six weeks later. Today, ketogenic diets reverse type 2 diabetes. They treat drug-resistant epilepsy. The ADA lists low-carb among its evidence-based dietary options. They called him a quack for 30 years. He was right the whole time. No sugar. No grains.

Vinnie Tortorich

47,558 görüntüleme • 4 ay önce

Dr. Thomas Seyfried drops a bombshell: "Cancer is very preventable... we're doing everything we possibly can to induce it in our diet & lifestyle." He flips the script: Cancer isn't primarily genetic—it's a metabolic disease rooted in damaged mitochondria forcing cells to ferment glucose and glutamine for energy. Key points from the clip: - Higher blood sugar = faster tumor growth (his early research showed this clearly). - Cancer cells rely on glucose + glutamine (they have faulty mitochondria). - Solution? Restrict both fuels via low-carb/ketogenic diet + fasting → enter nutritional ketosis. - Healthy cells thrive on ketone bodies; tumors starve slowly, lose blood supply, and get dissolved by the body. Non-toxic management potential: Target metabolism instead of (or alongside) traditional treatments. (Backed by his decades of work, including case studies & animal models on restricted ketogenic approaches—though mainstream oncology debates it as adjunct, not cure-all.) Mind-blowing rethink: What if prevention starts with blood sugar control and ditching high-carb lifestyles? Have you explored keto/fasting for health reasons? Seen any personal changes—or know someone who's tried metabolic approaches? Share experiences respectfully below!

Dr. Thomas Seyfried drops a bombshell: "Cancer is very preventable... we're doing everything we possibly can to induce it in our diet & lifestyle." He flips the script: Cancer isn't primarily genetic—it's a metabolic disease rooted in damaged mitochondria forcing cells to ferment glucose and glutamine for energy. Key points from the clip: - Higher blood sugar = faster tumor growth (his early research showed this clearly). - Cancer cells rely on glucose + glutamine (they have faulty mitochondria). - Solution? Restrict both fuels via low-carb/ketogenic diet + fasting → enter nutritional ketosis. - Healthy cells thrive on ketone bodies; tumors starve slowly, lose blood supply, and get dissolved by the body. Non-toxic management potential: Target metabolism instead of (or alongside) traditional treatments. (Backed by his decades of work, including case studies & animal models on restricted ketogenic approaches—though mainstream oncology debates it as adjunct, not cure-all.) Mind-blowing rethink: What if prevention starts with blood sugar control and ditching high-carb lifestyles? Have you explored keto/fasting for health reasons? Seen any personal changes—or know someone who's tried metabolic approaches? Share experiences respectfully below!

Camus

66,135 görüntüleme • 6 ay önce

Dr. Robert Lustig obliterated 50 years of cholesterol dogma in 41 seconds flat He literally said (watch every word): “If you eat a low-carb diet — keto, paleo, whatever — you reduce carbs/sugar and raise dietary fat. Yes, your LDL will go UP. But it’s the big, buoyant, fluffy LDL. That kind is cardiovascularly NEUTRAL. It’s not the bad guy. The actual bad guy is the small, dense LDL. Statins? They don’t even touch those. So the real question is: Do you want to lower an LDL that doesn’t matter… and in exchange put yourself at risk for chronic hyperglycemia, which absolutely DOES matter? Is that a good trade?” Exact quote ends at 0:41 I’m not telling you what to eat. I’m just asking: Have we been chasing the wrong villain this entire time? Drop your thoughts below — respectfully. Science needs debate, not dogma.

Dr. Robert Lustig obliterated 50 years of cholesterol dogma in 41 seconds flat He literally said (watch every word): “If you eat a low-carb diet — keto, paleo, whatever — you reduce carbs/sugar and raise dietary fat. Yes, your LDL will go UP. But it’s the big, buoyant, fluffy LDL. That kind is cardiovascularly NEUTRAL. It’s not the bad guy. The actual bad guy is the small, dense LDL. Statins? They don’t even touch those. So the real question is: Do you want to lower an LDL that doesn’t matter… and in exchange put yourself at risk for chronic hyperglycemia, which absolutely DOES matter? Is that a good trade?” Exact quote ends at 0:41 I’m not telling you what to eat. I’m just asking: Have we been chasing the wrong villain this entire time? Drop your thoughts below — respectfully. Science needs debate, not dogma.

Camus

146,341 görüntüleme • 9 ay önce

The Honey Diet - Unlocking Fat loss with Sugar and FGF21 Anabology (anabology) is a a biohacker and engineer turned health and nutrition enthusiast. We talk about the hormone FGF21's role in metabolism, the effects of macronutrients on hunger and satiety, and the potential for metabolic recovery through dietary adjustments. We discuss high-carb and carnivore diets, the impact of genetics and processed foods like Coke on health, and the importance of understanding individual responses to achieve optimal metabolic health through sustainable eating habits. 00:00 Introduction to Anabology and His Background 02:46 The Journey into Health and Nutrition 05:51 The Impact of Diet on Health 08:56 Exploring the Honey Diet 11:57 Understanding FGF21 and Its Role 14:51 The Mechanisms of Sugar and Fat in Metabolism 17:58 The Relationship Between Diet and Performance 21:11 The Future of Metabolic Health 34:18 Exploring Dietary Mechanisms: FGF21 and Metabolism 39:06 Satiety vs. Hunger: Understanding Metabolic Drivers 44:23 High Carb Diets: Insights from Vegan and Non-Vegan Perspectives 50:19 Metabolic Recovery: The Role of Diet in Weight Management 56:01 The Interplay of Hormones and Diet: Insights on Fat Loss 01:10:26 The Evolution of Diet Choices 01:12:01 Athletic Performance and Dietary Flexibility 01:14:47 Understanding Metabolic Health 01:17:33 Generational Impact on Metabolism 01:19:51 Dietary Preferences and Genetic Influences 01:23:19 The Macro Triangle: Balancing Nutrients 01:25:54 The Future of Diet Construction 01:28:56 Sustainable Eating Patterns 01:32:55 The Role of Protein in Energy Levels 01:36:29 FGF21: The Metabolic Regulator 01:46:18 The Medicinal Qualities of Coke 01:49:12 Coke vs. Mexican Coke: A Nutritional Debate 01:51:09 The Effects of Coke on Hunger and Satiety 01:53:47 The Body's Response to Processed Foods 01:56:54 Understanding FGF 21 and Its Role in Metabolism 02:02:57 The Complexities of Insulin Sensitivity 02:08:19 Natural vs. Synthetic Uncoupling in Metabolism 02:12:53 Future Endeavors and Insights on Diet

The Honey Diet - Unlocking Fat loss with Sugar and FGF21 Anabology (anabology) is a a biohacker and engineer turned health and nutrition enthusiast. We talk about the hormone FGF21's role in metabolism, the effects of macronutrients on hunger and satiety, and the potential for metabolic recovery through dietary adjustments. We discuss high-carb and carnivore diets, the impact of genetics and processed foods like Coke on health, and the importance of understanding individual responses to achieve optimal metabolic health through sustainable eating habits. 00:00 Introduction to Anabology and His Background 02:46 The Journey into Health and Nutrition 05:51 The Impact of Diet on Health 08:56 Exploring the Honey Diet 11:57 Understanding FGF21 and Its Role 14:51 The Mechanisms of Sugar and Fat in Metabolism 17:58 The Relationship Between Diet and Performance 21:11 The Future of Metabolic Health 34:18 Exploring Dietary Mechanisms: FGF21 and Metabolism 39:06 Satiety vs. Hunger: Understanding Metabolic Drivers 44:23 High Carb Diets: Insights from Vegan and Non-Vegan Perspectives 50:19 Metabolic Recovery: The Role of Diet in Weight Management 56:01 The Interplay of Hormones and Diet: Insights on Fat Loss 01:10:26 The Evolution of Diet Choices 01:12:01 Athletic Performance and Dietary Flexibility 01:14:47 Understanding Metabolic Health 01:17:33 Generational Impact on Metabolism 01:19:51 Dietary Preferences and Genetic Influences 01:23:19 The Macro Triangle: Balancing Nutrients 01:25:54 The Future of Diet Construction 01:28:56 Sustainable Eating Patterns 01:32:55 The Role of Protein in Energy Levels 01:36:29 FGF21: The Metabolic Regulator 01:46:18 The Medicinal Qualities of Coke 01:49:12 Coke vs. Mexican Coke: A Nutritional Debate 01:51:09 The Effects of Coke on Hunger and Satiety 01:53:47 The Body's Response to Processed Foods 01:56:54 Understanding FGF 21 and Its Role in Metabolism 02:02:57 The Complexities of Insulin Sensitivity 02:08:19 Natural vs. Synthetic Uncoupling in Metabolism 02:12:53 Future Endeavors and Insights on Diet

Josh Rainer

179,795 görüntüleme • 1 yıl önce

🚨#OreoVsStatin - PUBLISHED!🚨 OREO Cookie cookies were 💥2X as potent💥 at lowering my LDL cholesterol (LDL-C) compared to high-intensity statin therapy! What you NEED to know... 👉This was a metabolic demonstration, a 'do not do this at home' experiment that makes no 'health' claims about cookies or cholesterol. 👉The purpose was to BOLDLY test a prediction of the Lipid Energy Model #LEM, i.e. that adding in carbs, including in the from of an Oreo cookies 'addition' (not carb-fat swap) would lower LDL-C in an LMHR on a #keto diet Why Oreos? ... It's (productively) provocative! 😱 Research on #cholesterol on #keto diets and the #LEM and #LMHR phenotype has so much to teach us about human lipid metabolism! ... But how do I (we) communicate the scientific 'awe' that we feel to the greatest number of people (as well as force a conversation about #LMHR and #LEM that needs to happen)? ... A dramatic demonstration designed to turn heads might work! It's not 'just click bait' ... it's 'legit-bait' I want YOU to feel the 'shock and awe' I feel every day studying this topic. And I want you to engage in the research journey. What you need to do... or should do... 🙏PRETTY PLEASE 🙏! 👉PLEASE WATCH 👀 VIDEO ABSTRACT here (<8 min): 👉 SHARE THIS LINK to the paper itself in a quote RT or original Tweet of your own: ... doing so helps promote the work and provoke the discussion ... and feel free to tag in your favorite influencers (or least favorite 😉)... A HUGE thanks to the senior author and expert lipidologist William Cromwell, MD for his guiding wisdom, insightful thoughts, and always open and curious mind. And to Dave Feldman, my friend, research wife, and the '#LEM daddy' Finally... there will be a LOT of coverage of this experiment. Keep in mind, we are not making any health claims, but through a dramatic demonstration, highlighting an uncomfortable tension that arises from a prediction of the Lipid Energy Model, and that is consistent with prior data (7+ papers, including 'highest tier' data like our recent meta-analysis of 41 RCTs). Science is supposed to embrace such uncomfortable questions... so, join us in being curious and advocating for much needed research on #LEM and #LMHR so we can make sense of what observations like this really mean... STAY CURIOUS 🤔

🚨#OreoVsStatin - PUBLISHED!🚨 OREO Cookie cookies were 💥2X as potent💥 at lowering my LDL cholesterol (LDL-C) compared to high-intensity statin therapy! What you NEED to know... 👉This was a metabolic demonstration, a 'do not do this at home' experiment that makes no 'health' claims about cookies or cholesterol. 👉The purpose was to BOLDLY test a prediction of the Lipid Energy Model #LEM, i.e. that adding in carbs, including in the from of an Oreo cookies 'addition' (not carb-fat swap) would lower LDL-C in an LMHR on a #keto diet Why Oreos? ... It's (productively) provocative! 😱 Research on #cholesterol on #keto diets and the #LEM and #LMHR phenotype has so much to teach us about human lipid metabolism! ... But how do I (we) communicate the scientific 'awe' that we feel to the greatest number of people (as well as force a conversation about #LMHR and #LEM that needs to happen)? ... A dramatic demonstration designed to turn heads might work! It's not 'just click bait' ... it's 'legit-bait' I want YOU to feel the 'shock and awe' I feel every day studying this topic. And I want you to engage in the research journey. What you need to do... or should do... 🙏PRETTY PLEASE 🙏! 👉PLEASE WATCH 👀 VIDEO ABSTRACT here (<8 min): 👉 SHARE THIS LINK to the paper itself in a quote RT or original Tweet of your own: ... doing so helps promote the work and provoke the discussion ... and feel free to tag in your favorite influencers (or least favorite 😉)... A HUGE thanks to the senior author and expert lipidologist William Cromwell, MD for his guiding wisdom, insightful thoughts, and always open and curious mind. And to Dave Feldman, my friend, research wife, and the '#LEM daddy' Finally... there will be a LOT of coverage of this experiment. Keep in mind, we are not making any health claims, but through a dramatic demonstration, highlighting an uncomfortable tension that arises from a prediction of the Lipid Energy Model, and that is consistent with prior data (7+ papers, including 'highest tier' data like our recent meta-analysis of 41 RCTs). Science is supposed to embrace such uncomfortable questions... so, join us in being curious and advocating for much needed research on #LEM and #LMHR so we can make sense of what observations like this really mean... STAY CURIOUS 🤔

Nick Norwitz MD PhD

3,217,928 görüntüleme • 2 yıl önce

Your Kids Don’t Eat too Many Calories… IT’S THE CARBS! This video is from Jason Whittrock where he is demonstrating that continuous glucose monitor shows a glucose increase in response to eating chicken nuggets In the least shocking news of 2025 thus far… they chicken nuggets increase blood glucose Big deal He claims kids aren’t eating too many calories, implying that it is the carbs that are the problem. He claims that kids aren’t eating too many calories because obesity affects poor families more, trying to imply that poor kids must eat less calories because they have less money… BUT WHAT DOES THE DATA ACTUALLY SAY????? Oh well look at that, poor families actually eat MORE calories compared to middle class & upper class families (PMIDs: 26809142, 27994148, & 31082447) OOPSIE! Guess he either didn’t bother to look up the actual research data or knew about it & didn’t care As for carbs causing obesity, this has been debunked many times over. In a large meta-analysis of controlled feeding studies that equated calories & protein but varied carbohydrate & fat intake, researchers found that people in high carb/low fat diets actually lost slightly MORE body fat than people following low carb high fat diets (about 16g more body fat loss per day… not a huge difference) PMID: 28193517. These results have even been demonstrated with huge differences in carb intake (over 300g per day vs. ketogenic diet) & insulin release (PMID: 27385608) While I agree with Jason that chicken nuggets probably aren’t the best nutritional choice, it’s not because they have breading, it’s because they tend to be high in calories overall. Sorry Jason, calories mattercise Makes You Smarter It seems like every day, new data is emerging demonstrating that the benefits of exercise extend far beyond the physical & go well into the mental space of the brain. Research has shown exercise can improve symptoms of depression, schizophrenia, bi-polar, & anxiety but more research is also demonstrating that exercise can enhance cognitive performance in healthy people This is highlighted by a new meta-analysis demonstrating that exercise of various forms improved academic performance in college students (PMID: 39310016). While the mechanisms behind the way this works still need to be further elucidated, we are starting to discover more & more that what happens in the body affects the mind & what happens in the mind affects the body. In the reverse direction, we see psychological stress associated with greater pain sensitivity, autoimmune disorders, cardiovascular disease, cancer, and all cause mortality (PMIDs: 31568044, 32983091, 18371552, 34830968, & 27407085) E]]] want to take care of your body, take care of your mind.]

Your Kids Don’t Eat too Many Calories… IT’S THE CARBS! This video is from Jason Whittrock where he is demonstrating that continuous glucose monitor shows a glucose increase in response to eating chicken nuggets In the least shocking news of 2025 thus far… they chicken nuggets increase blood glucose Big deal He claims kids aren’t eating too many calories, implying that it is the carbs that are the problem. He claims that kids aren’t eating too many calories because obesity affects poor families more, trying to imply that poor kids must eat less calories because they have less money… BUT WHAT DOES THE DATA ACTUALLY SAY????? Oh well look at that, poor families actually eat MORE calories compared to middle class & upper class families (PMIDs: 26809142, 27994148, & 31082447) OOPSIE! Guess he either didn’t bother to look up the actual research data or knew about it & didn’t care As for carbs causing obesity, this has been debunked many times over. In a large meta-analysis of controlled feeding studies that equated calories & protein but varied carbohydrate & fat intake, researchers found that people in high carb/low fat diets actually lost slightly MORE body fat than people following low carb high fat diets (about 16g more body fat loss per day… not a huge difference) PMID: 28193517. These results have even been demonstrated with huge differences in carb intake (over 300g per day vs. ketogenic diet) & insulin release (PMID: 27385608) While I agree with Jason that chicken nuggets probably aren’t the best nutritional choice, it’s not because they have breading, it’s because they tend to be high in calories overall. Sorry Jason, calories mattercise Makes You Smarter It seems like every day, new data is emerging demonstrating that the benefits of exercise extend far beyond the physical & go well into the mental space of the brain. Research has shown exercise can improve symptoms of depression, schizophrenia, bi-polar, & anxiety but more research is also demonstrating that exercise can enhance cognitive performance in healthy people This is highlighted by a new meta-analysis demonstrating that exercise of various forms improved academic performance in college students (PMID: 39310016). While the mechanisms behind the way this works still need to be further elucidated, we are starting to discover more & more that what happens in the body affects the mind & what happens in the mind affects the body. In the reverse direction, we see psychological stress associated with greater pain sensitivity, autoimmune disorders, cardiovascular disease, cancer, and all cause mortality (PMIDs: 31568044, 32983091, 18371552, 34830968, & 27407085) E]]] want to take care of your body, take care of your mind.]

Layne Norton, PhD

26,140 görüntüleme • 1 yıl önce

"Acceptable" LDL Cholesterol Levels Used To Be 250 mg/dl. This Arbitrary Number Was Chosen By Big Pharma To Sell Statins. Dr Stephen Hussey Now, "Acceptable" Levels Must Be Below 100 mg/dl. Big Pharma Keeps Moving The Goalpost To Prescribe More Statins. Cholesterol Does Not Cause Heart Disease & Statins Have Never Been Proven To PREVENT Heart Disease. The Great Cholesterol Con Is Crumbling Down... Breaking New Study Reveals 17.3 Million Adults Do Not Need Statins, Including 4.1 Million Currently Taking Them. In 1984, one committee heavily influenced by Big Pharma decided, based on NO research or evidence, that LDL Cholesterol was bad & should be 250 mg/dl or lower. Over the years, the pharmaceutical industry used their power & leverage to lower these levels again based on NO research or evidence. They changed the guideline again in order to prescribe more Statins & now LDL Cholesterol must be kept below 100 mg/dl. No one knows what ideal Cholesterol levels are & there are no "best levels" at all. The entire idea that Cholesterol is bad came from very low quality associational studies & the number was basically made up. And then that arbitrary number was lowered every few years to make more profit from Statins. The lower Big Pharma can make the Cholesterol level guidelines, the more drugs are prescribed & sold. Making the Statin industry one of the biggest selling medicines worldwide accounting for an annual profit of $19 Billion & growing higher each year. We now know that Metabolic Dysfunction & Inflammation cause cardiovascular disease, heart attack, atherosclerosis & stroke. Which has given rise to Big Pharma once again profiting thru "Metabolic Drugs" like Ozempic, which are being pushed heavily & causing grave side effects for consumers. Medications are never going to be the answer for why this world is so sick. The only way to optimize vibrant health is for each person to make drastic lifestyle changes: 1: Nutrient Dense Animal Foods 2: Single Ingredient Whole Foods 3: Sun Exposure 4: Optimizing Circadian Rhythm 5: Avoid Toxin Exposure 6: Grounding/Direct Earth Contact 7: Fasting/Time Restricted Eating *⃣This means eliminating seed oils, eliminating ultra processed foods, no high carbohydrate diet, no added sugars*⃣ 👇2024 Statins Provide No Primary Benefit👇 👇The Higher Your LDL, The Longer You Live👇 👇The Keto Trial: LDL Rise Is Normal & Expected👇 Video Speaker: Dr. Stephen Hussey, DC

"Acceptable" LDL Cholesterol Levels Used To Be 250 mg/dl. This Arbitrary Number Was Chosen By Big Pharma To Sell Statins. Dr Stephen Hussey Now, "Acceptable" Levels Must Be Below 100 mg/dl. Big Pharma Keeps Moving The Goalpost To Prescribe More Statins. Cholesterol Does Not Cause Heart Disease & Statins Have Never Been Proven To PREVENT Heart Disease. The Great Cholesterol Con Is Crumbling Down... Breaking New Study Reveals 17.3 Million Adults Do Not Need Statins, Including 4.1 Million Currently Taking Them. In 1984, one committee heavily influenced by Big Pharma decided, based on NO research or evidence, that LDL Cholesterol was bad & should be 250 mg/dl or lower. Over the years, the pharmaceutical industry used their power & leverage to lower these levels again based on NO research or evidence. They changed the guideline again in order to prescribe more Statins & now LDL Cholesterol must be kept below 100 mg/dl. No one knows what ideal Cholesterol levels are & there are no "best levels" at all. The entire idea that Cholesterol is bad came from very low quality associational studies & the number was basically made up. And then that arbitrary number was lowered every few years to make more profit from Statins. The lower Big Pharma can make the Cholesterol level guidelines, the more drugs are prescribed & sold. Making the Statin industry one of the biggest selling medicines worldwide accounting for an annual profit of $19 Billion & growing higher each year. We now know that Metabolic Dysfunction & Inflammation cause cardiovascular disease, heart attack, atherosclerosis & stroke. Which has given rise to Big Pharma once again profiting thru "Metabolic Drugs" like Ozempic, which are being pushed heavily & causing grave side effects for consumers. Medications are never going to be the answer for why this world is so sick. The only way to optimize vibrant health is for each person to make drastic lifestyle changes: 1: Nutrient Dense Animal Foods 2: Single Ingredient Whole Foods 3: Sun Exposure 4: Optimizing Circadian Rhythm 5: Avoid Toxin Exposure 6: Grounding/Direct Earth Contact 7: Fasting/Time Restricted Eating ⃣This means eliminating seed oils, eliminating ultra processed foods, no high carbohydrate diet, no added sugars⃣ 👇2024 Statins Provide No Primary Benefit👇 👇The Higher Your LDL, The Longer You Live👇 👇The Keto Trial: LDL Rise Is Normal & Expected👇 Video Speaker: Dr. Stephen Hussey, DC

Valerie Anne Smith

179,827 görüntüleme • 1 yıl önce

There’s been a lot of buzz—and confusion—about the new Keto-CTA study, examining plaque progression in Lean Mass Hyper-Responders (LMHRs). Much of the social media debate has centered on whether high LDL on keto is safe or dangerous, driven largely by how to interpret the supplemental table comparing this study to others on LDL and plaque progression. In this episode of the Metabolic Mind Podcast, we sit down with Dr. Matthew Budoff, a world-renowned cardiologist, cardiac CT researcher, and the study's lead investigator, to discuss the the supplemental table, what the plaque markers mean, and how this fits into the discussion of high- vs -low-risk plaque progression. In this episode, we cover: ✅ What PAV (Percent Atheroma Volume) is, what it actually measures, and why it matters ✅ Why a 50% increase in plaque may sound scary, but can be deceiving ✅ The difference between “treatment-naive” and “treated” participants ✅ What the Miami Heart Study comparison reveals about keto, LDL, and plaque ✅ Why LDL alone may not tell the whole story about heart disease risk ✅ How some high-risk individuals may still benefit from statins and other therapies This study doesn’t answer whether keto causes heart disease or not. Instead, it shows that high LDL on a ketogenic diet is not a reliable predictor of plaque progression across all individuals. What is predictive? The presence of existing plaque. 💡 Key takeaway: Relying on surrogate markers of heart disease, like LDL and ApoB, is not the best way to assess heart disease risk in all populations. If you're concerned about how elevated LDL may be affecting your heart health, the best next step is to speak with your doctor about cardiac imaging to directly assess plaque and gain a clearer picture of your individual risk. Expert Featured: - Dr. Matthew Budoff - X: - Resources Mentioned: Plaque Begets Plaque, ApoB Does Not CMEs Mentioned: Managing Major Mental Illness with Dietary Change: The New Science of Hope Brain Energy: The Metabolic Theory of Mental Illness Follow our channel for more information and education from Bret Scher, MD, FACC, including interviews with leading experts in Metabolic Psychiatry. Learn more about metabolic psychiatry and find helpful resources at

There’s been a lot of buzz—and confusion—about the new Keto-CTA study, examining plaque progression in Lean Mass Hyper-Responders (LMHRs). Much of the social media debate has centered on whether high LDL on keto is safe or dangerous, driven largely by how to interpret the supplemental table comparing this study to others on LDL and plaque progression. In this episode of the Metabolic Mind Podcast, we sit down with Dr. Matthew Budoff, a world-renowned cardiologist, cardiac CT researcher, and the study's lead investigator, to discuss the the supplemental table, what the plaque markers mean, and how this fits into the discussion of high- vs -low-risk plaque progression. In this episode, we cover: ✅ What PAV (Percent Atheroma Volume) is, what it actually measures, and why it matters ✅ Why a 50% increase in plaque may sound scary, but can be deceiving ✅ The difference between “treatment-naive” and “treated” participants ✅ What the Miami Heart Study comparison reveals about keto, LDL, and plaque ✅ Why LDL alone may not tell the whole story about heart disease risk ✅ How some high-risk individuals may still benefit from statins and other therapies This study doesn’t answer whether keto causes heart disease or not. Instead, it shows that high LDL on a ketogenic diet is not a reliable predictor of plaque progression across all individuals. What is predictive? The presence of existing plaque. 💡 Key takeaway: Relying on surrogate markers of heart disease, like LDL and ApoB, is not the best way to assess heart disease risk in all populations. If you're concerned about how elevated LDL may be affecting your heart health, the best next step is to speak with your doctor about cardiac imaging to directly assess plaque and gain a clearer picture of your individual risk. Expert Featured: - Dr. Matthew Budoff - X: - Resources Mentioned: Plaque Begets Plaque, ApoB Does Not CMEs Mentioned: Managing Major Mental Illness with Dietary Change: The New Science of Hope Brain Energy: The Metabolic Theory of Mental Illness Follow our channel for more information and education from Bret Scher, MD, FACC, including interviews with leading experts in Metabolic Psychiatry. Learn more about metabolic psychiatry and find helpful resources at

Metabolic Mind

98,147 görüntüleme • 1 yıl önce

🚨 DOCTOR DROPS TRUTH BOMB ON MAMMOGRAMS 🚨 “Data shows we are literally CAUSING breast cancer with screening.” Dr. Jay Goodbinder just went nuclear: - After 10 yearly mammograms → 50%+ chance of a false-positive that leads to biopsies & more radiation - Crushing breasts with 40+ lbs of force + radiation can rupture cysts/tumors and spread cells - Cochrane Collaboration: For every 2,000 women screened over 10 years, only ~1 life saved… but up to 10 healthy women over-treated + 8–9 eventually die from radiation-induced cancers - A major study (JNCI) found careful breast exams alone reduce mortality JUST AS MUCH as mammograms in women 50+ Is “early detection” worth becoming the statistic? Safer, zero-radiation alternatives exist RIGHT NOW: ✅ Thermography ✅ QT Ultrasound (no compression, better accuracy) ✅ MRI for high-risk True prevention? Cancer is metabolic: → Keto/low-carb (starves tumors) → Ditch seed oils, sugar, processed junk → Fasting → autophagy cleanup → Vitamin D >70 ng/mL (cuts risk dramatically) → Avoid synthetic birth control hormones Watch the full breakdown 👇 Tag every woman you love. They deserve to know. #Mammogram #BreastCancer #WomensHealth #CancerPrevention #Keto

🚨 DOCTOR DROPS TRUTH BOMB ON MAMMOGRAMS 🚨 “Data shows we are literally CAUSING breast cancer with screening.” Dr. Jay Goodbinder just went nuclear: - After 10 yearly mammograms → 50%+ chance of a false-positive that leads to biopsies & more radiation - Crushing breasts with 40+ lbs of force + radiation can rupture cysts/tumors and spread cells - Cochrane Collaboration: For every 2,000 women screened over 10 years, only ~1 life saved… but up to 10 healthy women over-treated + 8–9 eventually die from radiation-induced cancers - A major study (JNCI) found careful breast exams alone reduce mortality JUST AS MUCH as mammograms in women 50+ Is “early detection” worth becoming the statistic? Safer, zero-radiation alternatives exist RIGHT NOW: ✅ Thermography ✅ QT Ultrasound (no compression, better accuracy) ✅ MRI for high-risk True prevention? Cancer is metabolic: → Keto/low-carb (starves tumors) → Ditch seed oils, sugar, processed junk → Fasting → autophagy cleanup → Vitamin D >70 ng/mL (cuts risk dramatically) → Avoid synthetic birth control hormones Watch the full breakdown 👇 Tag every woman you love. They deserve to know. #Mammogram #BreastCancer #WomensHealth #CancerPrevention #Keto

Valerie Anne Smith

3,645,182 görüntüleme • 9 ay önce

Testosterone is far more than just a muscle-building hormone. It’s a cornerstone of health influencing mood, cognition, bone density, libido, and even longevity. In this episode, Derek (More Plates More Dates), host of the popular YouTube channel ‘More Plates More Dates' and co-founder of Marek Health, joins me to unpack testosterone’s true role in health, from bone density and insulin sensitivity to brain function and longevity. We cover how lifestyle factors, diet, and micronutrients like vitamin D, zinc, and magnesium shape testosterone levels, and which testosterone-boosting supplements actually work versus those that are just hype. We dive deep into testosterone replacement therapy—when it’s warranted, what benefits to expect, the risks (like polycythemia and fertility suppression), and how delivery methods such as injections, creams, and orals formulations differ. We also explore testosterone therapy for women, cardiovascular disease and prostate cancer risk, and how DHT drives hair loss, plus the safety and efficacy of treatments like finasteride, minoxidil, microneedling, and ketoconazole shampoo. If you want to understand testosterone beyond the headlines, including its functions, its tradeoffs, and the practical ways to optimize it, this conversation is a must-listen. Links in the next post. Timestamps: 0:00 - Introduction 1:58 - Why men need testosterone 4:19 - Testosterone's role in women 6:00 - Does high testosterone shorten life? 9:19 - What castrati reveal about lifespan 12:15 - Free vs. total testosterone 15:49 - Measuring testosterone 18:36 - Reference ranges vs. symptoms 21:58 - When high T signals trouble 23:40 - What LH and FSH tell you 28:19 - Is high SHBG hurting your T? 32:10 - Why SHBG rises with age 36:52 - Signs of low testosterone 39:54 - Alcohol’s impact on testosterone 42:46 - How low-fat & low-carb diets lower T 43:25 - Micronutrient mistakes hurting hormones 45:19 - Excess body fat & low testosterone 48:46 - Risks of excessive endurance training 53:09 - Endocrine disruptors 55:50 - Are testosterone levels declining? 58:39 - Why dietary fat boosts hormones 1:01:02 - Does keto harm testosterone? 1:02:17 - Best exercise for testosterone 1:04:23 - Do vitamin D, zinc, magnesium help? 1:08:43 - Can boron boost free testosterone? 1:09:53 - Ashwagandha 1:14:07 - Is Tongkat Ali best for boosting T? 1:17:58 - Tongkat Ali vs. boron 1:19:27 - Shilajit, tribulus, & fenugreek 1:20:41 - Best 4 supplements for testosterone 1:22:25 - Dutch vs. blood tests for cortisol 1:23:40 - When to consider TRT 1:31:30 - Realistic TRT benefits 1:34:41 - TRT and heart risks 1:44:31 - Creams vs. injections 1:45:55 - TRT and prostate cancer risk 1:48:08 - Side effects of TRT 1:50:48 - Rollercoaster effect from injections 1:53:23 - Low testosterone risks vs. TRT 1:56:46 - Choosing TRT delivery methods 2:03:23 - Smaller, frequent injections safer? 2:05:20 - Maintaining fertility on TRT 2:13:20 - Why TRT shrinks testicles 2:14:47 - Biomarkers to track on TRT 2:24:04 - Testosterone therapy for women 2:33:57 - Can DHEA safely raise women's T? 2:36:54 - Causes of hair loss 2:43:08 - Does your hairline come from grandpa? 2:43:55 - Risks of stopping hair loss 2:53:02 - Ketoconazole, minoxidil & microneedling 2:56:03 - Topical vs. oral minoxidil 2:59:08 - Microneedling without minoxidil 3:01:59 - Finasteride, dutasteride & the brain 3:03:10 - Finasteride & nocebo effect 3:04:44 - Does minoxidil delay baldness? 3:06:13 - Can dutasteride extend lifespan?

Testosterone is far more than just a muscle-building hormone. It’s a cornerstone of health influencing mood, cognition, bone density, libido, and even longevity. In this episode, Derek (More Plates More Dates), host of the popular YouTube channel ‘More Plates More Dates' and co-founder of Marek Health, joins me to unpack testosterone’s true role in health, from bone density and insulin sensitivity to brain function and longevity. We cover how lifestyle factors, diet, and micronutrients like vitamin D, zinc, and magnesium shape testosterone levels, and which testosterone-boosting supplements actually work versus those that are just hype. We dive deep into testosterone replacement therapy—when it’s warranted, what benefits to expect, the risks (like polycythemia and fertility suppression), and how delivery methods such as injections, creams, and orals formulations differ. We also explore testosterone therapy for women, cardiovascular disease and prostate cancer risk, and how DHT drives hair loss, plus the safety and efficacy of treatments like finasteride, minoxidil, microneedling, and ketoconazole shampoo. If you want to understand testosterone beyond the headlines, including its functions, its tradeoffs, and the practical ways to optimize it, this conversation is a must-listen. Links in the next post. Timestamps: 0:00 - Introduction 1:58 - Why men need testosterone 4:19 - Testosterone's role in women 6:00 - Does high testosterone shorten life? 9:19 - What castrati reveal about lifespan 12:15 - Free vs. total testosterone 15:49 - Measuring testosterone 18:36 - Reference ranges vs. symptoms 21:58 - When high T signals trouble 23:40 - What LH and FSH tell you 28:19 - Is high SHBG hurting your T? 32:10 - Why SHBG rises with age 36:52 - Signs of low testosterone 39:54 - Alcohol’s impact on testosterone 42:46 - How low-fat & low-carb diets lower T 43:25 - Micronutrient mistakes hurting hormones 45:19 - Excess body fat & low testosterone 48:46 - Risks of excessive endurance training 53:09 - Endocrine disruptors 55:50 - Are testosterone levels declining? 58:39 - Why dietary fat boosts hormones 1:01:02 - Does keto harm testosterone? 1:02:17 - Best exercise for testosterone 1:04:23 - Do vitamin D, zinc, magnesium help? 1:08:43 - Can boron boost free testosterone? 1:09:53 - Ashwagandha 1:14:07 - Is Tongkat Ali best for boosting T? 1:17:58 - Tongkat Ali vs. boron 1:19:27 - Shilajit, tribulus, & fenugreek 1:20:41 - Best 4 supplements for testosterone 1:22:25 - Dutch vs. blood tests for cortisol 1:23:40 - When to consider TRT 1:31:30 - Realistic TRT benefits 1:34:41 - TRT and heart risks 1:44:31 - Creams vs. injections 1:45:55 - TRT and prostate cancer risk 1:48:08 - Side effects of TRT 1:50:48 - Rollercoaster effect from injections 1:53:23 - Low testosterone risks vs. TRT 1:56:46 - Choosing TRT delivery methods 2:03:23 - Smaller, frequent injections safer? 2:05:20 - Maintaining fertility on TRT 2:13:20 - Why TRT shrinks testicles 2:14:47 - Biomarkers to track on TRT 2:24:04 - Testosterone therapy for women 2:33:57 - Can DHEA safely raise women's T? 2:36:54 - Causes of hair loss 2:43:08 - Does your hairline come from grandpa? 2:43:55 - Risks of stopping hair loss 2:53:02 - Ketoconazole, minoxidil & microneedling 2:56:03 - Topical vs. oral minoxidil 2:59:08 - Microneedling without minoxidil 3:01:59 - Finasteride, dutasteride & the brain 3:03:10 - Finasteride & nocebo effect 3:04:44 - Does minoxidil delay baldness? 3:06:13 - Can dutasteride extend lifespan?

Dr. Rhonda Patrick

174,320 görüntüleme • 11 ay önce

Think your blood glucose tells the whole story? Think again. Dr. Ben Bikman (Benjamin Bikman) joins me to discuss insulin resistance, including why it often remains hidden until late stages and simple ways to detect it early. We explore insulin’s lesser-known roles in fat storage, appetite regulation, inflammation, and chronic disease. You’ll also learn how macronutrients, meal timing, and frequency uniquely influence your insulin sensitivity, and the critical differences between visceral and subcutaneous fat. Finally, we dive into how stress, sleep deprivation, nicotine, and environmental toxins shape your metabolism and unpack the realities of popular GLP-1 receptor agonist medications (like Ozempic), including their benefits, risks, and the intriguing potential of microdosing. We also provide practical, actionable strategies, dietary adjustments, exercise protocols, and targeted supplements that you can implement today to significantly boost insulin sensitivity and support long-term metabolic health. If you want to understand insulin resistance and take immediate steps to improve your metabolic health - this conversation is a must-watch. Watch the full episode on X and YouTube or listen on Spotify and Apple Podcasts now. Links in the comments. Timestamps: 0:00 - Introduction 1:33 - Can you be insulin resistant with normal glucose levels? 5:13 - Can glucose monitors detect hidden insulin resistance? 6:43 - What your skin reveals about insulin resistance 8:07 - Why is insulin resistance behind so many chronic diseases? 12:28 - Does obesity cause insulin resistance—or vice versa? 19:20 - Insulin’s surprising roles beyond blood sugar control 20:18 - What’s driving weight gain—insulin or calories? 27:12 - Do saturated fats cause insulin resistance? 33:44 - Why refined carbs amplify risks from saturated fat 36:46 - Fructose vs. refined sugar—which spikes insulin more? 37:43 - High-carb vs. keto—which diet controls hunger better? 42:09 - Why low-carb diets might provide a metabolic advantage 44:18 - Does exercise give you metabolic ‘wiggle room’? 48:42 - Why strength training beats cardio for insulin sensitivity 53:54 - Does meal frequency drive insulin resistance? 57:14 - Is nighttime snacking giving you insomnia? 59:06 - Can a sugary breakfast lead to overeating later? 1:04:01 - Does late-night eating disrupt sleep more than blue light? 1:05:41 - Can one bad night’s sleep trigger insulin resistance? 1:09:06 - Can air pollution cause weight gain? 1:12:58 - Vaping vs. smoking—which is worse for metabolic health? 1:14:20 - Can statins and antidepressants trigger weight gain? 1:17:04 - How to reverse insulin resistance in 90 days 1:27:35 - Ketone supplements—are the metabolic benefits real? 1:33:16 - Why some ethnicities get diabetes without obesity 1:41:11 - How oversized fat cells trigger metabolic chaos 1:46:10 - Do seed oils silently promote insulin resistance? 1:49:27 - Seed oils—always harmful or only when heated? 2:01:03 - Do fat cells shrink or disappear with weight loss? 2:03:48 - Are shrunken fat cells still insulin resistant? 2:06:23 - Injecting insulin for muscle—are the risks worth it? 2:09:27 - Are drugs like Ozempic a shortcut or solution for obesity? 2:15:55 - Are current GLP-1 agonist doses too high? 2:16:44 - Microdosing GLP-1 drugs—a solution for carb cravings? 2:22:43 - Do these medications cause muscle loss? 2:25:12 - Do GLP-1 agonist benefits extend beyond weight loss? 2:27:23 - Could these treatments actually promote longevity? 2:32:55 - The dark side of GLP-1 drugs—can they trigger depression? 2:36:14 - Insulin vs. glucose—what really drives accelerated aging? 2:41:16 - How high glucose levels damage cells 2:43:23 - How insulin shuts down your body’s stress defenses 2:47:57 - Which biomarkers best predict biological aging? 2:54:01 - Does eating dinner early improve insulin sensitivity?

Think your blood glucose tells the whole story? Think again. Dr. Ben Bikman (Benjamin Bikman) joins me to discuss insulin resistance, including why it often remains hidden until late stages and simple ways to detect it early. We explore insulin’s lesser-known roles in fat storage, appetite regulation, inflammation, and chronic disease. You’ll also learn how macronutrients, meal timing, and frequency uniquely influence your insulin sensitivity, and the critical differences between visceral and subcutaneous fat. Finally, we dive into how stress, sleep deprivation, nicotine, and environmental toxins shape your metabolism and unpack the realities of popular GLP-1 receptor agonist medications (like Ozempic), including their benefits, risks, and the intriguing potential of microdosing. We also provide practical, actionable strategies, dietary adjustments, exercise protocols, and targeted supplements that you can implement today to significantly boost insulin sensitivity and support long-term metabolic health. If you want to understand insulin resistance and take immediate steps to improve your metabolic health - this conversation is a must-watch. Watch the full episode on X and YouTube or listen on Spotify and Apple Podcasts now. Links in the comments. Timestamps: 0:00 - Introduction 1:33 - Can you be insulin resistant with normal glucose levels? 5:13 - Can glucose monitors detect hidden insulin resistance? 6:43 - What your skin reveals about insulin resistance 8:07 - Why is insulin resistance behind so many chronic diseases? 12:28 - Does obesity cause insulin resistance—or vice versa? 19:20 - Insulin’s surprising roles beyond blood sugar control 20:18 - What’s driving weight gain—insulin or calories? 27:12 - Do saturated fats cause insulin resistance? 33:44 - Why refined carbs amplify risks from saturated fat 36:46 - Fructose vs. refined sugar—which spikes insulin more? 37:43 - High-carb vs. keto—which diet controls hunger better? 42:09 - Why low-carb diets might provide a metabolic advantage 44:18 - Does exercise give you metabolic ‘wiggle room’? 48:42 - Why strength training beats cardio for insulin sensitivity 53:54 - Does meal frequency drive insulin resistance? 57:14 - Is nighttime snacking giving you insomnia? 59:06 - Can a sugary breakfast lead to overeating later? 1:04:01 - Does late-night eating disrupt sleep more than blue light? 1:05:41 - Can one bad night’s sleep trigger insulin resistance? 1:09:06 - Can air pollution cause weight gain? 1:12:58 - Vaping vs. smoking—which is worse for metabolic health? 1:14:20 - Can statins and antidepressants trigger weight gain? 1:17:04 - How to reverse insulin resistance in 90 days 1:27:35 - Ketone supplements—are the metabolic benefits real? 1:33:16 - Why some ethnicities get diabetes without obesity 1:41:11 - How oversized fat cells trigger metabolic chaos 1:46:10 - Do seed oils silently promote insulin resistance? 1:49:27 - Seed oils—always harmful or only when heated? 2:01:03 - Do fat cells shrink or disappear with weight loss? 2:03:48 - Are shrunken fat cells still insulin resistant? 2:06:23 - Injecting insulin for muscle—are the risks worth it? 2:09:27 - Are drugs like Ozempic a shortcut or solution for obesity? 2:15:55 - Are current GLP-1 agonist doses too high? 2:16:44 - Microdosing GLP-1 drugs—a solution for carb cravings? 2:22:43 - Do these medications cause muscle loss? 2:25:12 - Do GLP-1 agonist benefits extend beyond weight loss? 2:27:23 - Could these treatments actually promote longevity? 2:32:55 - The dark side of GLP-1 drugs—can they trigger depression? 2:36:14 - Insulin vs. glucose—what really drives accelerated aging? 2:41:16 - How high glucose levels damage cells 2:43:23 - How insulin shuts down your body’s stress defenses 2:47:57 - Which biomarkers best predict biological aging? 2:54:01 - Does eating dinner early improve insulin sensitivity?

Dr. Rhonda Patrick

149,642 görüntüleme • 1 yıl önce

Most of us don’t have a slow metabolism, and are far from overtraining. The real issue is people underestimate their calorie intake, confuse discomfort with "failure," and just don't do the work. New episode with Dr. Layne Norton (Layne Norton, PhD), a Ph.D. scientist, professional bodybuilder, and champion powerlifter who deadlifts over 700 pounds. We discuss: • When to push to failure • Whether seed oils promote chronic disease (and how) • Why good habits beat perfectionism • Layne’s approach to eating, training, and supplementation. Plus, we tackle hot topics like the carnivore diet, artificial sweeteners, intermittent fasting, and more. This episode is available on X, YouTube, Spotify, and everywhere else. Links in comment. Timestamps: 0:00 - Introduction 2:30 - Layne's coaching philosophy 12:22 - Why to start tracking calories (for at least 3 days) 15:24 - Why people mislead themselves about food intake 20:49 - Exercise vs. SSRIs 24:35 - Treat exercise like brushing your teeth 27:53 - Why old people should lift 31:34 - Should you train to failure? 44:50 - Exercise selection 54:29 - Is lifting heavy necessary? 55:37 - Barbell vs. hack squats for preventing falls 57:53 - Why lifting weights decreases lower back pain 59:26 - How to prevent and overcome training injuries 1:08:59 - Why 'exposure therapy' promotes recovery 1:12:47 - Why pain doesn't always indicate tissue injury 1:16:00 - Why and how to train after poor sleep 1:19:40 - Why menopause can cause weight gain 1:27:19 - Why it's never too late to start lifting weights 1:29:48 - Training tips for older people with joint pain 1:34:01 - Total protein intake vs. distribution 1:42:02 - Layne's daily protein distribution 1:44:27 - The shortcomings of nutrition studies 1:51:49 - Is consuming more than 1.6 g/kg of protein beneficial? 1:53:16 - Protein consumption in a calorie deficit 1:54:26 - Protein intake for endurance athletes 1:55:50 - How much protein does Layne eat? 1:56:54 - Are seed oils a predominant cause of disease? 2:01:15 - Saturated fat vs. seed oils 2:06:28 - Is the carnivore diet an LDL cholesterol catastrophe? 2:11:59 - Why heating seeds oil makes them inflammatory 2:18:16 - Is there a "big food" industry conspiracy? 2:24:00 - Sugar-sweetened beverages 2:28:00 - Can diet soda help you lose weight? 2:32:03 - Diet soda (cancer and microbiome) 2:42:07 - Why Layne rarely takes a strong position on early science 2:46:47 - Carnivore diet 2:59:35 - Time-restricted eating and fasting 3:10:21 - Layne's daily routine 3:14:38 - Layne's diet and supplements 3:16:21 - Why everyone should take creatine 3:20:32 - Rhodiola rosea 3:22:03 - Ashwagandha 3:23:37 - Promising supplements (need more evidence)

Most of us don’t have a slow metabolism, and are far from overtraining. The real issue is people underestimate their calorie intake, confuse discomfort with "failure," and just don't do the work. New episode with Dr. Layne Norton (Layne Norton, PhD), a Ph.D. scientist, professional bodybuilder, and champion powerlifter who deadlifts over 700 pounds. We discuss: • When to push to failure • Whether seed oils promote chronic disease (and how) • Why good habits beat perfectionism • Layne’s approach to eating, training, and supplementation. Plus, we tackle hot topics like the carnivore diet, artificial sweeteners, intermittent fasting, and more. This episode is available on X, YouTube, Spotify, and everywhere else. Links in comment. Timestamps: 0:00 - Introduction 2:30 - Layne's coaching philosophy 12:22 - Why to start tracking calories (for at least 3 days) 15:24 - Why people mislead themselves about food intake 20:49 - Exercise vs. SSRIs 24:35 - Treat exercise like brushing your teeth 27:53 - Why old people should lift 31:34 - Should you train to failure? 44:50 - Exercise selection 54:29 - Is lifting heavy necessary? 55:37 - Barbell vs. hack squats for preventing falls 57:53 - Why lifting weights decreases lower back pain 59:26 - How to prevent and overcome training injuries 1:08:59 - Why 'exposure therapy' promotes recovery 1:12:47 - Why pain doesn't always indicate tissue injury 1:16:00 - Why and how to train after poor sleep 1:19:40 - Why menopause can cause weight gain 1:27:19 - Why it's never too late to start lifting weights 1:29:48 - Training tips for older people with joint pain 1:34:01 - Total protein intake vs. distribution 1:42:02 - Layne's daily protein distribution 1:44:27 - The shortcomings of nutrition studies 1:51:49 - Is consuming more than 1.6 g/kg of protein beneficial? 1:53:16 - Protein consumption in a calorie deficit 1:54:26 - Protein intake for endurance athletes 1:55:50 - How much protein does Layne eat? 1:56:54 - Are seed oils a predominant cause of disease? 2:01:15 - Saturated fat vs. seed oils 2:06:28 - Is the carnivore diet an LDL cholesterol catastrophe? 2:11:59 - Why heating seeds oil makes them inflammatory 2:18:16 - Is there a "big food" industry conspiracy? 2:24:00 - Sugar-sweetened beverages 2:28:00 - Can diet soda help you lose weight? 2:32:03 - Diet soda (cancer and microbiome) 2:42:07 - Why Layne rarely takes a strong position on early science 2:46:47 - Carnivore diet 2:59:35 - Time-restricted eating and fasting 3:10:21 - Layne's daily routine 3:14:38 - Layne's diet and supplements 3:16:21 - Why everyone should take creatine 3:20:32 - Rhodiola rosea 3:22:03 - Ashwagandha 3:23:37 - Promising supplements (need more evidence)

Dr. Rhonda Patrick

228,403 görüntüleme • 2 yıl önce

🧬 Why does ☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆 say “Food doesn’t matter”? Imagine a solar-powered battery: Sunlight charges the battery. Water holds the charge. Magnetism organizes the charge. The battery powers the engine. The engine burns the fuel. Food is a fuel. If the battery is empty or the wiring is broken, the engine cannot burn the fuel correctly. Changing the fuel will not fix the battery. Fix the battery first, then any fuel can be used. 🤔 Understanding 🔥 1. The ENGINE comes before the FUEL If the mitochondrial engine is not working, the fuel doesn’t matter. Why? Because in our modern world, we are trying to fix a physics problem with food ideology. It will never work. Mitochondria are the engine that burns the fuel. If the engine is damaged, all fuels become incomplete, misinterpreted, or even toxic. 👎🏻Most people today have engines damaged by: blue light EMFs indoor living poor redox lack of DHA no morning sunlight circadian mismatch ❌When the engine is broken, these things happen: electrons cannot move efficiently ATP production slows inflammation rises ROS becomes uncontrolled hormones lose coherence fasting becomes stressful diets fail again and again Food does not fix any of this. ☀️ 2. LIGHT controls metabolism more deeply than any diet ever could Jack’s central point for 15 years: “Food is seasonal information. Light is the master instruction.” Morning sunlight programs: dopamine cortisol melatonin leptin mitochondrial timing thyroid signaling This means two people on the same diet can have completely different results depending on their light environment. Example: Someone living indoors under LEDs with low redox tries carnivore. They struggle. Someone living by the ocean with daily sunrise exposure builds strong redox. Carnivore works for them Keto works Carbs work seasonally Fasting works The diet didn’t change. The redox did. Diet is just light in disguise. 🌊 3. Mitochondria run on ELECTRONS, not on food quantity Jack always says: “You don’t need more supplements. You need more electrons.” Food is only an electron donor. But electrons only work when the biology is prepared to receive them in the same environment nature designed. The key factors that prepare the system are: DHA in cell membranes structured water created by sunlight a magnetic field that aligns proton flow circadian signals that control timing cold exposure that increases electron efficiency minimal blue light at night When these conditions are damaged: Electrons get stuck. Energy drops. Inflammation rises. Hormones lose rhythm. Detoxification breaks. Again: diet never fixes the foundation. 🧠 4. Why diet “gurus” are always fighting, and always missing the point. Carnivore gurus… Low-carb communities… Metabolic bros… Vegan influencers… Mediterranean… All of them fight about the fuel while ignoring the engine. It’s like watching the mechanics scream about gasoline quality while the engine has: no oil no spark plugs a dead battery Jack’s message is brutally simple: “Fix the mitochondrial environment first, and most diets will begin to work.” Without fixing the environment, no diet can save you. 🐟 5. Why DHA is the diet Kruse actually cares about. DHA is the molecule that lets sunlight become a direct electrical current inside your cell membranes. DHA is the antenna. If the antenna is broken, the light signal becomes distorted. Every metabolic pathway fails. This is why: “A vegan in Norway is committing mitochondrial suicide.” “Food is a distraction. Sunlight is the real nutrient.” DHA + sunlight is the diet nature designed for humans. DHA is an antenna. And an antenna is useless without the signal. 🔥 Remember Your mitochondrial redox decides your fate; not your diet. Your light environment writes the rules for your metabolism. Your DHA status determines how well you can read the rules. This is why all food fights are meaningless 🤭 The mitochondria decide everything long before the fork ever reaches your mouth.

🧬 Why does ☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆 say “Food doesn’t matter”? Imagine a solar-powered battery: Sunlight charges the battery. Water holds the charge. Magnetism organizes the charge. The battery powers the engine. The engine burns the fuel. Food is a fuel. If the battery is empty or the wiring is broken, the engine cannot burn the fuel correctly. Changing the fuel will not fix the battery. Fix the battery first, then any fuel can be used. 🤔 Understanding 🔥 1. The ENGINE comes before the FUEL If the mitochondrial engine is not working, the fuel doesn’t matter. Why? Because in our modern world, we are trying to fix a physics problem with food ideology. It will never work. Mitochondria are the engine that burns the fuel. If the engine is damaged, all fuels become incomplete, misinterpreted, or even toxic. 👎🏻Most people today have engines damaged by: blue light EMFs indoor living poor redox lack of DHA no morning sunlight circadian mismatch ❌When the engine is broken, these things happen: electrons cannot move efficiently ATP production slows inflammation rises ROS becomes uncontrolled hormones lose coherence fasting becomes stressful diets fail again and again Food does not fix any of this. ☀️ 2. LIGHT controls metabolism more deeply than any diet ever could Jack’s central point for 15 years: “Food is seasonal information. Light is the master instruction.” Morning sunlight programs: dopamine cortisol melatonin leptin mitochondrial timing thyroid signaling This means two people on the same diet can have completely different results depending on their light environment. Example: Someone living indoors under LEDs with low redox tries carnivore. They struggle. Someone living by the ocean with daily sunrise exposure builds strong redox. Carnivore works for them Keto works Carbs work seasonally Fasting works The diet didn’t change. The redox did. Diet is just light in disguise. 🌊 3. Mitochondria run on ELECTRONS, not on food quantity Jack always says: “You don’t need more supplements. You need more electrons.” Food is only an electron donor. But electrons only work when the biology is prepared to receive them in the same environment nature designed. The key factors that prepare the system are: DHA in cell membranes structured water created by sunlight a magnetic field that aligns proton flow circadian signals that control timing cold exposure that increases electron efficiency minimal blue light at night When these conditions are damaged: Electrons get stuck. Energy drops. Inflammation rises. Hormones lose rhythm. Detoxification breaks. Again: diet never fixes the foundation. 🧠 4. Why diet “gurus” are always fighting, and always missing the point. Carnivore gurus… Low-carb communities… Metabolic bros… Vegan influencers… Mediterranean… All of them fight about the fuel while ignoring the engine. It’s like watching the mechanics scream about gasoline quality while the engine has: no oil no spark plugs a dead battery Jack’s message is brutally simple: “Fix the mitochondrial environment first, and most diets will begin to work.” Without fixing the environment, no diet can save you. 🐟 5. Why DHA is the diet Kruse actually cares about. DHA is the molecule that lets sunlight become a direct electrical current inside your cell membranes. DHA is the antenna. If the antenna is broken, the light signal becomes distorted. Every metabolic pathway fails. This is why: “A vegan in Norway is committing mitochondrial suicide.” “Food is a distraction. Sunlight is the real nutrient.” DHA + sunlight is the diet nature designed for humans. DHA is an antenna. And an antenna is useless without the signal. 🔥 Remember Your mitochondrial redox decides your fate; not your diet. Your light environment writes the rules for your metabolism. Your DHA status determines how well you can read the rules. This is why all food fights are meaningless 🤭 The mitochondria decide everything long before the fork ever reaches your mouth.

Light Me Away ☀️

42,199 görüntüleme • 8 ay önce