正在加载视频...

视频加载失败

加载此视频时出现问题。这可能是由于临时网络问题，或视频可能不可用。

A study explored how dandelion extract affected aggressive breast cancer cells in lab settings, drawing attention to how some plants interact with cancer biology. Dandelion is often dismissed as a weed, yet research has examined its effects on inflammation, metabolism, and liver-related pathways. Nature holds many compounds worth studying.... show more

Metabolic Factor

172,788 subscribers

10,951 次观看 • 7 个月前 •via X (Twitter)

Anya Rossi• Live Now

Private livecam show

0 条评论

暂无评论

原始帖子的评论将显示在这里

相关视频

🚨 A HARVARD TRAINED MD JUST EXPLAINED WHY IVERMECTIN IS BEING STUDIED IN CANCER RESEARCH AGAIN And the internet is paying VERY close attention. Dr. Shintani says researchers have explored how Ivermectin may interact with cancer cells in laboratory and preclinical studies involving: • Breast cancer • Prostate cancer • Colon cancer • Lung cancer • Leukemia • Liver cancer • Stomach cancer Why scientists are interested: ✅ Studies suggest Ivermectin may trigger apoptosis, also known as programmed cancer cell death ✅ Researchers are investigating how it may affect tumor growth pathways and cancer stem-like cells ✅ Some preclinical studies are exploring Ivermectin alongside chemotherapy in laboratory models And THAT is why repurposed medicine conversations have exploded across podcasts, TikTok, X, and cancer forums worldwide. Not because the science is settled. Because people are shocked that a low-cost anti-parasitic drug is even being discussed in cancer research at all. But whether critics like it or not… The public conversation around repurposed drugs is getting impossible to ignore.

🚨 A HARVARD TRAINED MD JUST EXPLAINED WHY IVERMECTIN IS BEING STUDIED IN CANCER RESEARCH AGAIN And the internet is paying VERY close attention. Dr. Shintani says researchers have explored how Ivermectin may interact with cancer cells in laboratory and preclinical studies involving: • Breast cancer • Prostate cancer • Colon cancer • Lung cancer • Leukemia • Liver cancer • Stomach cancer Why scientists are interested: ✅ Studies suggest Ivermectin may trigger apoptosis, also known as programmed cancer cell death ✅ Researchers are investigating how it may affect tumor growth pathways and cancer stem-like cells ✅ Some preclinical studies are exploring Ivermectin alongside chemotherapy in laboratory models And THAT is why repurposed medicine conversations have exploded across podcasts, TikTok, X, and cancer forums worldwide. Not because the science is settled. Because people are shocked that a low-cost anti-parasitic drug is even being discussed in cancer research at all. But whether critics like it or not… The public conversation around repurposed drugs is getting impossible to ignore.

Joe Tippens

10,621 次观看 • 3 个月前

96% Cancer Cells Dead in 48 Hours. That's not a headline from a fringe blog. That's from a peer‑reviewed study published in Oncotarget. Researchers at the University of Windsor tested dandelion root extract on colon cancer cells. The results were stunning: >95% of cancer cells eliminated within 48 hours. >90% reduction in tumor growth in mice. Zero toxicity to healthy cells. And it's not just colon cancer. Lab studies show dandelion root extract triggers apoptosis (programmed cell death) in leukemia, breast, prostate, pancreatic, lung, and colorectal cancer cells. But here's the catch. All of this is in laboratory conditions – not humans. Many substances that work brilliantly in a petri dish fail in the human body. Dr. Caroline Hamm, the lead researcher, has publicly warned that viral claims are "misleading." She's seen patients abandon standard medicine based on these unfounded claims. Here's the part that makes you think. Health Canada actually approved a Phase I human trial back in 2012. But the study couldn't recruit enough volunteers. No clinical data was ever published. Why didn't that trial move forward? Some have asked a harder question: who funds clinical trials for a plant that grows in every backyard? A common weed. Showing powerful anticancer activity in labs. Zero toxicity in animal models. And no one is running the trials. Monsanto spent billions eradicating this "weed." Why? Because a free, abundant plant that heals threatens their entire business model. How to Use It (Traditional Use – Not a Cure) 1 tsp dried dandelion root 1 cup hot water Steep 10 minutes Strain and drink Two cups daily – one before breakfast, one before dinner It's bitter. That's your liver thanking you. The Reality Check 🧪 These are lab studies and case reports – not controlled clinical trials. ⚠️ Dandelion root extract has not been proven to cure cancer in humans. 💊 Never abandon conventional treatment based on these claims. 📚 We need proper clinical trials. But who funds trials for a weed? Nature has answers. We've just been trained to look everywhere else. 👇 Watch the full video above. Have you ever used dandelion root? I'd love to hear your experience.

96% Cancer Cells Dead in 48 Hours. That's not a headline from a fringe blog. That's from a peer‑reviewed study published in Oncotarget. Researchers at the University of Windsor tested dandelion root extract on colon cancer cells. The results were stunning: >95% of cancer cells eliminated within 48 hours. >90% reduction in tumor growth in mice. Zero toxicity to healthy cells. And it's not just colon cancer. Lab studies show dandelion root extract triggers apoptosis (programmed cell death) in leukemia, breast, prostate, pancreatic, lung, and colorectal cancer cells. But here's the catch. All of this is in laboratory conditions – not humans. Many substances that work brilliantly in a petri dish fail in the human body. Dr. Caroline Hamm, the lead researcher, has publicly warned that viral claims are "misleading." She's seen patients abandon standard medicine based on these unfounded claims. Here's the part that makes you think. Health Canada actually approved a Phase I human trial back in 2012. But the study couldn't recruit enough volunteers. No clinical data was ever published. Why didn't that trial move forward? Some have asked a harder question: who funds clinical trials for a plant that grows in every backyard? A common weed. Showing powerful anticancer activity in labs. Zero toxicity in animal models. And no one is running the trials. Monsanto spent billions eradicating this "weed." Why? Because a free, abundant plant that heals threatens their entire business model. How to Use It (Traditional Use – Not a Cure) 1 tsp dried dandelion root 1 cup hot water Steep 10 minutes Strain and drink Two cups daily – one before breakfast, one before dinner It's bitter. That's your liver thanking you. The Reality Check 🧪 These are lab studies and case reports – not controlled clinical trials. ⚠️ Dandelion root extract has not been proven to cure cancer in humans. 💊 Never abandon conventional treatment based on these claims. 📚 We need proper clinical trials. But who funds trials for a weed? Nature has answers. We've just been trained to look everywhere else. 👇 Watch the full video above. Have you ever used dandelion root? I'd love to hear your experience.

Dr. David

38,096 次观看 • 1 天前

Bee venom can kill aggressive breast cancer cells in just 60 minutes. UCLA research showed that honeybee venom and its main component, melittin, rapidly destroyed triple-negative and HER2-enriched breast cancer cells, some of the most aggressive types. It worked by interfering with key growth receptors (EGFR and HER2) on the cancer cell membrane. The 2020 study (and subsequent research) found melittin induced massive cell death within 60 minutes in lab-tested human breast cancer lines, while having minimal effect on healthy cells. Follow-up meta-analyses have confirmed its potent anti-cancer activity, especially against hard-to-treat subtypes. This is the kind of outside-the-box thinking we need, cheap, natural compounds that could one day offer accessible options alongside conventional treatments.

Bee venom can kill aggressive breast cancer cells in just 60 minutes. UCLA research showed that honeybee venom and its main component, melittin, rapidly destroyed triple-negative and HER2-enriched breast cancer cells, some of the most aggressive types. It worked by interfering with key growth receptors (EGFR and HER2) on the cancer cell membrane. The 2020 study (and subsequent research) found melittin induced massive cell death within 60 minutes in lab-tested human breast cancer lines, while having minimal effect on healthy cells. Follow-up meta-analyses have confirmed its potent anti-cancer activity, especially against hard-to-treat subtypes. This is the kind of outside-the-box thinking we need, cheap, natural compounds that could one day offer accessible options alongside conventional treatments.

Camus

23,224 次观看 • 3 个月前

🚨 “THEY CALLED IT HORSE PASTE… WHILE SCIENTISTS WERE STUDYING IT FOR CANCER AND CHEMO RESISTANCE.” That’s why the Ivermectin conversation refuses to die. According to researchers studying repurposed medicine, Ivermectin is one of the most fascinating molecules ever discovered because it appears to interact with completely different biological pathways depending on the disease being studied. In parasites, researchers found it affects chloride channels. In viruses, studies explored how it may interfere with replication pathways. And in cancer research, scientists have investigated how Ivermectin may interact with tumor proliferation pathways, cancer stem like cells, and chemotherapy resistance mechanisms. Researchers believe cancer stem cells may play a major role in: • Recurrence • Metastasis • Treatment resistance Traditional chemotherapy mainly targets rapidly dividing cells, but scientists are now studying whether Ivermectin may affect more resistant stem like cancer cells differently in laboratory settings. There are also preclinical studies investigating how Ivermectin may help restore chemotherapy sensitivity in resistant tumor cells at the molecular level. And then there’s another reason people keep talking about it: Some researchers have explored whether the same pathways involved in abnormal tumor growth may also be involved in conditions like fibroids, lipomas, and endometriosis. That’s why the repurposed medicine conversation has exploded far beyond COVID. If you’re researching Ivermectin, Fenbendazole, or Mebendazole, many choose PharmacyinUSA for worldwide and discreet delivery. #Ivermectin #HorseDewormer #CancerCure

🚨 “THEY CALLED IT HORSE PASTE… WHILE SCIENTISTS WERE STUDYING IT FOR CANCER AND CHEMO RESISTANCE.” That’s why the Ivermectin conversation refuses to die. According to researchers studying repurposed medicine, Ivermectin is one of the most fascinating molecules ever discovered because it appears to interact with completely different biological pathways depending on the disease being studied. In parasites, researchers found it affects chloride channels. In viruses, studies explored how it may interfere with replication pathways. And in cancer research, scientists have investigated how Ivermectin may interact with tumor proliferation pathways, cancer stem like cells, and chemotherapy resistance mechanisms. Researchers believe cancer stem cells may play a major role in: • Recurrence • Metastasis • Treatment resistance Traditional chemotherapy mainly targets rapidly dividing cells, but scientists are now studying whether Ivermectin may affect more resistant stem like cancer cells differently in laboratory settings. There are also preclinical studies investigating how Ivermectin may help restore chemotherapy sensitivity in resistant tumor cells at the molecular level. And then there’s another reason people keep talking about it: Some researchers have explored whether the same pathways involved in abnormal tumor growth may also be involved in conditions like fibroids, lipomas, and endometriosis. That’s why the repurposed medicine conversation has exploded far beyond COVID. If you’re researching Ivermectin, Fenbendazole, or Mebendazole, many choose PharmacyinUSA for worldwide and discreet delivery. #Ivermectin #HorseDewormer #CancerCure

PharmacyinUSA

15,268 次观看 • 3 个月前

FENBENDAZOLE IS MAKING PEOPLE ASK BIGGER QUESTIONS Why are so many cancer patients suddenly talking about a dewormer? Because fenbendazole-originally used as an anti-parasitic -has shown promising early research in laboratory settings for its potential anti-cancer effects. That should make people pause. Not because it's a miracle cure. #cancer #research #fenbendazol #chemo #healing Not because it replaces treatment. But because when an overlooked compound starts showing unexpected potential... it forces a bigger question: « What else are we not paying attention to? Some studies suggest fenbendazole may interfere with how cancer cells grow and divide. And yet- most people never hear about it until they start digging for answers themselves. That's the pattern. The most interesting conversations in cancer care often happen outside the mainstream spotlight. This doesn't mean every claim is true. But it does mean this: If patients are being forced to search this hard for emerging options... the conversation is incomplete. Because people facing cancer deserve to know what research is evolving— not just what's already standardized. Stop reacting - and start making informed decisions.

FENBENDAZOLE IS MAKING PEOPLE ASK BIGGER QUESTIONS Why are so many cancer patients suddenly talking about a dewormer? Because fenbendazole-originally used as an anti-parasitic -has shown promising early research in laboratory settings for its potential anti-cancer effects. That should make people pause. Not because it's a miracle cure. #cancer #research #fenbendazol #chemo #healing Not because it replaces treatment. But because when an overlooked compound starts showing unexpected potential... it forces a bigger question: « What else are we not paying attention to? Some studies suggest fenbendazole may interfere with how cancer cells grow and divide. And yet- most people never hear about it until they start digging for answers themselves. That's the pattern. The most interesting conversations in cancer care often happen outside the mainstream spotlight. This doesn't mean every claim is true. But it does mean this: If patients are being forced to search this hard for emerging options... the conversation is incomplete. Because people facing cancer deserve to know what research is evolving— not just what's already standardized. Stop reacting - and start making informed decisions.

Joe Tippens

10,415 次观看 • 1 个月前

This is Samantha Ruth Prabhu, a film star, misleading and misinforming over 33 million followers on "detoxing the liver." The podcast feature some random health illiterate "Wellness Coach & Performance Nutritionist" who has absolutely no clue how the human body works and has the most rubbish content on his Instagram handle, including complete nonsense such as herbs to manage autoimmune disorders. I am not sure how people with massive following very easily figure out the worst, science-illiterate people for them to invite to talk on science, medicine and health on "health podcasts" that are in fact nothing to do with health or medicine. Its just two science illiterates sharing their ignorance. The Wellness Coach guy is not even a real medical person and would probably have no idea about functions of the liver. He says, the best herb to improve liver health is Dandelion. I am a liver doctor, a trained and registered hepatologist diagnosing and treating liver disease patients since one decade and this is complete and utter BS. Dandelion, is a vegetable that most people consider a weed. It is sometimes used as a salad green. Dandelion can be used in salad. About 100 g of dandelion provides about 10-15% of your daily potassium requirements, at little to no caloric intake. Dandelion can increase urine output and work as a "diuretic" or like a "water pill" as per traditional medicine. But there is a lack of good human evidence for this effect. Animal studies and in vitro evidence suggest dandelion may have a variety of other beneficial health effects, but much more research is needed to trace these effects back to individual compounds found in dandelions. Limited rodent (thats rat & mice study) evidence suggests dandelion may be able to ease digestion by increasing the rate at which food leaves the stomach and enters the small intestine. Supplementation of dandelion cannot be recommended at this time due to a lack of human evidence for its effects. Consuming wild dandelions is not recommended, especially those grown in urban and suburban settings, due to high risk of pesticide exposure. The only thing that features dandelions as useful is the song Dandelions by Ruth B. Do check it out. Lay source: Remember: Dandelions by Ruth B ✅ Dandelions by Samantha Ruth ❌

This is Samantha Ruth Prabhu, a film star, misleading and misinforming over 33 million followers on "detoxing the liver." The podcast feature some random health illiterate "Wellness Coach & Performance Nutritionist" who has absolutely no clue how the human body works and has the most rubbish content on his Instagram handle, including complete nonsense such as herbs to manage autoimmune disorders. I am not sure how people with massive following very easily figure out the worst, science-illiterate people for them to invite to talk on science, medicine and health on "health podcasts" that are in fact nothing to do with health or medicine. Its just two science illiterates sharing their ignorance. The Wellness Coach guy is not even a real medical person and would probably have no idea about functions of the liver. He says, the best herb to improve liver health is Dandelion. I am a liver doctor, a trained and registered hepatologist diagnosing and treating liver disease patients since one decade and this is complete and utter BS. Dandelion, is a vegetable that most people consider a weed. It is sometimes used as a salad green. Dandelion can be used in salad. About 100 g of dandelion provides about 10-15% of your daily potassium requirements, at little to no caloric intake. Dandelion can increase urine output and work as a "diuretic" or like a "water pill" as per traditional medicine. But there is a lack of good human evidence for this effect. Animal studies and in vitro evidence suggest dandelion may have a variety of other beneficial health effects, but much more research is needed to trace these effects back to individual compounds found in dandelions. Limited rodent (thats rat & mice study) evidence suggests dandelion may be able to ease digestion by increasing the rate at which food leaves the stomach and enters the small intestine. Supplementation of dandelion cannot be recommended at this time due to a lack of human evidence for its effects. Consuming wild dandelions is not recommended, especially those grown in urban and suburban settings, due to high risk of pesticide exposure. The only thing that features dandelions as useful is the song Dandelions by Ruth B. Do check it out. Lay source: Remember: Dandelions by Ruth B ✅ Dandelions by Samantha Ruth ❌

TheLiverDoc™

265,633 次观看 • 2 年前

🚨 WHY ARE THERE OVER 400 PUBLISHED PAPERS ON IVERMECTIN AND CANCER? Because researchers discovered something unexpected. Ivermectin doesn't just target parasites. According to preclinical research, it appears to interact with cancer cells through multiple biological pathways. One of the most discussed? 📌 Cancer Stem Cells These are the cells believed to survive treatment, remain dormant for years, and potentially contribute to recurrence and spread. According to Dr. William Makis: 💊 Chemotherapy primarily targets rapidly dividing cancer cells. 💊 Ivermectin has been studied for its potential effects on cancer stem cells. That's why some researchers believe the combination deserves attention. But that's only part of the story. Researchers have also investigated ivermectin's potential ability to: ✅ Target cancer stem cells ✅ Influence tumor signaling pathways ✅ Alter cancer cell behavior ✅ Reverse chemotherapy resistance One of the most fascinating findings involves chemotherapy resistance. Over time, some cancer cells develop the ability to push chemotherapy drugs back out of the cell. Researchers have reported that ivermectin may interfere with these resistance mechanisms, potentially making cancer cells susceptible again. And then there's Fenbendazole and Mebendazole. While Ivermectin is being studied for one set of mechanisms, Fenbendazole and Mebendazole appear to work differently. Researchers have investigated their potential ability to: 📌 Block glucose transporters on cancer cells 📌 Reduce the cell's ability to use glucose as fuel 📌 Interfere with pathways associated with tumor growth Think about that. Different compounds. Different mechanisms. The same goal. That's why scientists continue publishing study after study. Not because these compounds are a fad. Because researchers keep uncovering new biological pathways worth investigating. And that's why the conversation around Ivermectin, Fenbendazole, and Mebendazole continues to grow. 💊 If you’re looking into these drugs, check out Ivermectin, Fenbendazole, and Mebendazole from RXMEDS.STORE. #Ivermectin #Fenbendazole #Mebendazole #CancerResearch #CancerStemCells #RepurposedDrugs #CancerBiology #RxMeds

🚨 WHY ARE THERE OVER 400 PUBLISHED PAPERS ON IVERMECTIN AND CANCER? Because researchers discovered something unexpected. Ivermectin doesn't just target parasites. According to preclinical research, it appears to interact with cancer cells through multiple biological pathways. One of the most discussed? 📌 Cancer Stem Cells These are the cells believed to survive treatment, remain dormant for years, and potentially contribute to recurrence and spread. According to Dr. William Makis: 💊 Chemotherapy primarily targets rapidly dividing cancer cells. 💊 Ivermectin has been studied for its potential effects on cancer stem cells. That's why some researchers believe the combination deserves attention. But that's only part of the story. Researchers have also investigated ivermectin's potential ability to: ✅ Target cancer stem cells ✅ Influence tumor signaling pathways ✅ Alter cancer cell behavior ✅ Reverse chemotherapy resistance One of the most fascinating findings involves chemotherapy resistance. Over time, some cancer cells develop the ability to push chemotherapy drugs back out of the cell. Researchers have reported that ivermectin may interfere with these resistance mechanisms, potentially making cancer cells susceptible again. And then there's Fenbendazole and Mebendazole. While Ivermectin is being studied for one set of mechanisms, Fenbendazole and Mebendazole appear to work differently. Researchers have investigated their potential ability to: 📌 Block glucose transporters on cancer cells 📌 Reduce the cell's ability to use glucose as fuel 📌 Interfere with pathways associated with tumor growth Think about that. Different compounds. Different mechanisms. The same goal. That's why scientists continue publishing study after study. Not because these compounds are a fad. Because researchers keep uncovering new biological pathways worth investigating. And that's why the conversation around Ivermectin, Fenbendazole, and Mebendazole continues to grow. 💊 If you’re looking into these drugs, check out Ivermectin, Fenbendazole, and Mebendazole from RXMEDS.STORE. #Ivermectin #Fenbendazole #Mebendazole #CancerResearch #CancerStemCells #RepurposedDrugs #CancerBiology #RxMeds

RXMEDS.STORE

16,612 次观看 • 2 个月前

Retatrutide forever, watch this (Spoiler alert science agrees) Here’s everything you need (it’s free!) Think of it like this… Biology is a building… Drugs are trying to fix one room Retatrutide handles everything… At the same time Most doctors and Instaexperts talk about it without actually understanding biology or what they're doing… 2023 Cell Metabolism proved retatrutide wasn’t just about weight loss… it rewires how cells process energy at the mitochondrial level 2021 Aging Cell showed glucagon signaling cranks up autophagy specifically in neurons and hepatocytes… brain cells and liver cells The cells that matter most for longevity 2023 Nature Metabolism proved weekly pulsatile signaling activates more downstream pathways than multiple weekly signaling Why? Because cells have memory Receptor sensitivity actually increases if you give the signal, then let it recover then signal again So weekly 0.1 mg? Actually more effective than microdosing 2-3x a week And here's the best part… At 0.1 mg weekly… no weight loss unless you have significant metabolic dysfunction but cells will upgrade mitochondria increase autophagy and reduce systemic inflammation Biology is aging more slowly FYI 2024 Nature Communications… they applied retatrutide to cancer cells and the cancer cells died Why? Because cancer cells are metabolically distinct from normal cells They rely on a different energy pathway the Warburg effect and bypass normal mitochondrial ATP production It’s a cancer predator, period The 2025 follow-up study showed existing tumors exposed to retatrutide had 67 % tumor shrinkage You're making biology hostile to cancer at the cellular level Do what you want to do… I’m here to help you Because I actually care Never Miss Dr Trevor Bachmeyer The Spartan

Retatrutide forever, watch this (Spoiler alert science agrees) Here’s everything you need (it’s free!) Think of it like this… Biology is a building… Drugs are trying to fix one room Retatrutide handles everything… At the same time Most doctors and Instaexperts talk about it without actually understanding biology or what they're doing… 2023 Cell Metabolism proved retatrutide wasn’t just about weight loss… it rewires how cells process energy at the mitochondrial level 2021 Aging Cell showed glucagon signaling cranks up autophagy specifically in neurons and hepatocytes… brain cells and liver cells The cells that matter most for longevity 2023 Nature Metabolism proved weekly pulsatile signaling activates more downstream pathways than multiple weekly signaling Why? Because cells have memory Receptor sensitivity actually increases if you give the signal, then let it recover then signal again So weekly 0.1 mg? Actually more effective than microdosing 2-3x a week And here's the best part… At 0.1 mg weekly… no weight loss unless you have significant metabolic dysfunction but cells will upgrade mitochondria increase autophagy and reduce systemic inflammation Biology is aging more slowly FYI 2024 Nature Communications… they applied retatrutide to cancer cells and the cancer cells died Why? Because cancer cells are metabolically distinct from normal cells They rely on a different energy pathway the Warburg effect and bypass normal mitochondrial ATP production It’s a cancer predator, period The 2025 follow-up study showed existing tumors exposed to retatrutide had 67 % tumor shrinkage You're making biology hostile to cancer at the cellular level Do what you want to do… I’m here to help you Because I actually care Never Miss Dr Trevor Bachmeyer The Spartan

Dr Trevor Bachmeyer

22,492 次观看 • 28 天前

In an interview, Dr. Kathleen Ruddy, a distinguished breast cancer surgeon, author, and inventor, shared groundbreaking insights on repurposed drugs for cancer treatment. She discussed how medications like ivermectin, mebendazole, sildenafil, metformin, aspirin, doxycycline, and vitamin C show promise in targeting cancer stem cells, the drivers of tumor regrowth. “Cancer stem cells are the real bad boys,” Dr. Ruddy stated, explaining that chemotherapy and radiation often fail to eliminate them, leading to cancer recurrence. Preclinical studies show combinations like doxycycline and vitamin C can eradicate over 90% of cancer stem cells, with doxycycline, azithromycin, and vitamin C proving equally effective. These affordable, FDA-approved drugs could halt tumors without harming patients. Dr. Ruddy praised Dr. Paul Marik’s Cancer Care monograph as a must-read for its analysis of repurposed drugs. Her mission is to cure cancer, not manage it as a chronic disease. “Patients want to live without cancer,” she emphasized. Through her work, she’s studying these therapies’ impact on cancers like breast, prostate, and lung. “Kill the cancer, or the cancer will kill you,” Dr. Ruddy urged. Could repurposed drugs transform cancer care?

In an interview, Dr. Kathleen Ruddy, a distinguished breast cancer surgeon, author, and inventor, shared groundbreaking insights on repurposed drugs for cancer treatment. She discussed how medications like ivermectin, mebendazole, sildenafil, metformin, aspirin, doxycycline, and vitamin C show promise in targeting cancer stem cells, the drivers of tumor regrowth. “Cancer stem cells are the real bad boys,” Dr. Ruddy stated, explaining that chemotherapy and radiation often fail to eliminate them, leading to cancer recurrence. Preclinical studies show combinations like doxycycline and vitamin C can eradicate over 90% of cancer stem cells, with doxycycline, azithromycin, and vitamin C proving equally effective. These affordable, FDA-approved drugs could halt tumors without harming patients. Dr. Ruddy praised Dr. Paul Marik’s Cancer Care monograph as a must-read for its analysis of repurposed drugs. Her mission is to cure cancer, not manage it as a chronic disease. “Patients want to live without cancer,” she emphasized. Through her work, she’s studying these therapies’ impact on cancers like breast, prostate, and lung. “Kill the cancer, or the cancer will kill you,” Dr. Ruddy urged. Could repurposed drugs transform cancer care?

Camus

117,774 次观看 • 1 年前

The power of Dandelions!! Dandelion Flower Benefits: Dandelion flowers are rich in antioxidants, including flavonoids and phenolic compounds, which contribute to their anti-inflammatory properties. The flowers are also known to have diuretic properties, aiding in kidney health and promoting detoxification. Dandelion flower extracts have been studied for their potential anti-cancer effects and protective properties against oxidative stress. Dandelion Root Benefits: Dandelion roots contain inulin, a prebiotic fiber that supports gut health and digestion. The roots are traditionally used for liver support and detoxification due to their bitter compounds that stimulate bile production. Dandelion root extracts have shown potential benefits for regulating blood sugar levels and reducing inflammation. Best Time to Pick Dandelion Flowers and Roots: Dandelion flowers are best picked in the morning when they are fully open and vibrant in color. Dandelion roots are typically harvested in the fall when the plant has stored nutrients in the roots to prepare for winter. Where to Find Dandelions: Dandelions are common and can be found in lawns, meadows, and gardens. They thrive in various habitats, including fields, roadsides, and even cracks in sidewalks. It's essential to ensure the dandelions you pick have not been treated with pesticides or chemicals to avoid contamination.

The power of Dandelions!! Dandelion Flower Benefits: Dandelion flowers are rich in antioxidants, including flavonoids and phenolic compounds, which contribute to their anti-inflammatory properties. The flowers are also known to have diuretic properties, aiding in kidney health and promoting detoxification. Dandelion flower extracts have been studied for their potential anti-cancer effects and protective properties against oxidative stress. Dandelion Root Benefits: Dandelion roots contain inulin, a prebiotic fiber that supports gut health and digestion. The roots are traditionally used for liver support and detoxification due to their bitter compounds that stimulate bile production. Dandelion root extracts have shown potential benefits for regulating blood sugar levels and reducing inflammation. Best Time to Pick Dandelion Flowers and Roots: Dandelion flowers are best picked in the morning when they are fully open and vibrant in color. Dandelion roots are typically harvested in the fall when the plant has stored nutrients in the roots to prepare for winter. Where to Find Dandelions: Dandelions are common and can be found in lawns, meadows, and gardens. They thrive in various habitats, including fields, roadsides, and even cracks in sidewalks. It's essential to ensure the dandelions you pick have not been treated with pesticides or chemicals to avoid contamination.

illuminatibot

395,721 次观看 • 2 年前

Can people in power/ authority kindly stop with this absolute BS on "fasting killing cancer cells" and citing religious nonsense to appeal to peoples emotions? Fasting is really quite dangerous for cancer patients in real life. Let me explain and bury this myth once and for all, especially for science illiterates like this guy. [1] The most common argument is that fasting "starves" cancer by cutting off its sugar (glucose) supply. While it is true that cancer cells consume vast amounts of glucose, they are biologically aggressive survivalists. If you stop eating, your body eventually switches to burning fat and breaking down muscle for energy. Cancer cells are highly adaptable; when glucose is low, many types of cancer can mutate to feed on other fuel sources, such as lactate, amino acids (from your muscles), or fatty acids. You cannot simply "starve" a tumor without starving the patient first. [2] Fasting is dangerous for cancer patients because of cancer cachexia - a wasting syndrome where the body loses muscle and fat rapidly. Cachexia is responsible for up to 30% of cancer deaths. Cancer puts the body in a hyper-metabolic state (burning energy fast). If a patient fasts, they risk accelerating muscle loss and weakening their immune system. A weak body cannot tolerate life-saving treatments like chemotherapy or radiation, nor can it fight off infections. [3] Most claims about fasting curing cancer come from studies on mice or cells in a petri dish. In a dish: You can kill cancer cells with almost anything (lemon juice, bleach, starvation, even shooting a bullet at it at close point or using a grenade to destroy the entire lab) because they have no immune system or body to protect them. In a human: The biology is infinitely more complex. Human metabolism, hormonal fluctuations, and tumor micro-environments mean that what shrinks a tumor in a mouse often fails completely in human trials. [4] Proponents often cite "autophagy" (the body's cellular recycling process triggered by fasting) as the cure. They claim it cleans out cancerous cells. Science shows that autophagy is a double-edged sword. -In all people, autophagy is a normal physiological process - whether fasting or not, which help in cleaning up damaged cells. -But in patients with cancer, once a tumor exists, cancer cells can actually hijack autophagy to survive stress (like chemotherapy) and repair themselves. In this context, fasting could theoretically help the cancer survive the treatment intended to kill it. [5] "Cancer" is not one disease; it is over 200 different diseases characterized by uncontrolled cell growth. Some cancers are driven by hormones, some by genetic mutations, and some by viruses. A fasting protocol that slows down one specific type of breast cancer might have zero effect on pancreatic cancer, or worse, accelerate a different type. The most lethal aspect of this misinformation is the delay in treatment. Cancer is a time-sensitive disease. While a patient spends months trying to fast the cancer away based on religious or alternative advice, the cancer often metastasizes (spreads) to other organs. Once cancer spreads, it often moves from being curable to being terminal. Relying solely on fasting wastes the critical window where medical intervention could have saved a life. Suggesting a single "ancient cure" for 200 complex genetic diseases is scientifically illogical... ...and generally stupid, as this video proves.

Can people in power/ authority kindly stop with this absolute BS on "fasting killing cancer cells" and citing religious nonsense to appeal to peoples emotions? Fasting is really quite dangerous for cancer patients in real life. Let me explain and bury this myth once and for all, especially for science illiterates like this guy. [1] The most common argument is that fasting "starves" cancer by cutting off its sugar (glucose) supply. While it is true that cancer cells consume vast amounts of glucose, they are biologically aggressive survivalists. If you stop eating, your body eventually switches to burning fat and breaking down muscle for energy. Cancer cells are highly adaptable; when glucose is low, many types of cancer can mutate to feed on other fuel sources, such as lactate, amino acids (from your muscles), or fatty acids. You cannot simply "starve" a tumor without starving the patient first. [2] Fasting is dangerous for cancer patients because of cancer cachexia - a wasting syndrome where the body loses muscle and fat rapidly. Cachexia is responsible for up to 30% of cancer deaths. Cancer puts the body in a hyper-metabolic state (burning energy fast). If a patient fasts, they risk accelerating muscle loss and weakening their immune system. A weak body cannot tolerate life-saving treatments like chemotherapy or radiation, nor can it fight off infections. [3] Most claims about fasting curing cancer come from studies on mice or cells in a petri dish. In a dish: You can kill cancer cells with almost anything (lemon juice, bleach, starvation, even shooting a bullet at it at close point or using a grenade to destroy the entire lab) because they have no immune system or body to protect them. In a human: The biology is infinitely more complex. Human metabolism, hormonal fluctuations, and tumor micro-environments mean that what shrinks a tumor in a mouse often fails completely in human trials. [4] Proponents often cite "autophagy" (the body's cellular recycling process triggered by fasting) as the cure. They claim it cleans out cancerous cells. Science shows that autophagy is a double-edged sword. -In all people, autophagy is a normal physiological process - whether fasting or not, which help in cleaning up damaged cells. -But in patients with cancer, once a tumor exists, cancer cells can actually hijack autophagy to survive stress (like chemotherapy) and repair themselves. In this context, fasting could theoretically help the cancer survive the treatment intended to kill it. [5] "Cancer" is not one disease; it is over 200 different diseases characterized by uncontrolled cell growth. Some cancers are driven by hormones, some by genetic mutations, and some by viruses. A fasting protocol that slows down one specific type of breast cancer might have zero effect on pancreatic cancer, or worse, accelerate a different type. The most lethal aspect of this misinformation is the delay in treatment. Cancer is a time-sensitive disease. While a patient spends months trying to fast the cancer away based on religious or alternative advice, the cancer often metastasizes (spreads) to other organs. Once cancer spreads, it often moves from being curable to being terminal. Relying solely on fasting wastes the critical window where medical intervention could have saved a life. Suggesting a single "ancient cure" for 200 complex genetic diseases is scientifically illogical... ...and generally stupid, as this video proves.

TheLiverDoc™

193,678 次观看 • 7 个月前

What if one of cancer’s most devastating side effects could be reversed? Cancer cachexia is a debilitating wasting syndrome that affects many people with cancer, yet treatment options remain limited. Team CANCAN has published new research in Science Magazine that highlights a previously underappreciated driver of cancer cachexia: localised signals from tumours acting directly on sensory neurons. This is a big step toward reframing cancer cachexia as not only a systemic metabolic condition, but also one shaped by tumour–nerve communication. 🎥 Papagiannakopoulos Lab, Co-Investigator on team CANCAN and team InteroCANCEption, explains the discovery and what it could mean for patients. Read the full paper: --- Team CANCAN is funded by Cancer Research UK and National Cancer Institute through Cancer Grand Challenges.

What if one of cancer’s most devastating side effects could be reversed? Cancer cachexia is a debilitating wasting syndrome that affects many people with cancer, yet treatment options remain limited. Team CANCAN has published new research in Science Magazine that highlights a previously underappreciated driver of cancer cachexia: localised signals from tumours acting directly on sensory neurons. This is a big step toward reframing cancer cachexia as not only a systemic metabolic condition, but also one shaped by tumour–nerve communication. 🎥 Papagiannakopoulos Lab, Co-Investigator on team CANCAN and team InteroCANCEption, explains the discovery and what it could mean for patients. Read the full paper: --- Team CANCAN is funded by Cancer Research UK and National Cancer Institute through Cancer Grand Challenges.

Cancer Grand Challenges

41,648 次观看 • 1 个月前

Cancer cells rely heavily on glucose to survive and multiply. Research shows they consume sugar at a far higher rate than healthy cells, which is why diet is often discussed in supportive care. However, this doesn’t mean all sugar is the same. Whole fruits come packaged with fiber, minerals, and protective compounds that slow absorption and support normal metabolism in healthy bodies. Understanding how the body processes food helps people make wiser choices—supporting balance rather than fear. The goal is nourishment, not deprivation, and working with the body’s natural design. 🌿 Knowledge brings clarity. Balance brings strength.

Cancer cells rely heavily on glucose to survive and multiply. Research shows they consume sugar at a far higher rate than healthy cells, which is why diet is often discussed in supportive care. However, this doesn’t mean all sugar is the same. Whole fruits come packaged with fiber, minerals, and protective compounds that slow absorption and support normal metabolism in healthy bodies. Understanding how the body processes food helps people make wiser choices—supporting balance rather than fear. The goal is nourishment, not deprivation, and working with the body’s natural design. 🌿 Knowledge brings clarity. Balance brings strength.

Barbara Oneill

68,716 次观看 • 8 个月前

⚔️ BLOCKING GLUTAMINE NATURALLY When cancer cells can’t get glucose, they often switch to glutamine, a key amino acid involved in cell growth and repair. 🍄 Mistletoe—immune-regulating and disrupts glutamine-dependent cancer growth 🌿 Curcumin—shown to interfere with glutamine metabolism in aggressive tumors 🍵 Green tea (EGCG)—again, powerful here as well 🌿 Berberine—reduces expression of glutamine transporters 🧠 Honokiol (from magnolia bark)—neuroprotective and suppresses glutamine usage 🍄 Turkey tail mushroom—modulates immune response and may slow glutamine-driven cell replication 🥬 Fermented vegetables—rich in butyrate, which opposes glutamine-fueled inflammation 🥥 Coconut oil—encourages ketone metabolism over glutamine ✅ Suggested Stack: • AM: Honokiol + green tea • PM: Curcumin + mistletoe extract (consult with a practitioner) • Daily: Coconut oil for brain fuel + fermented foods for terrain healing 🧬 Avoid excess animal protein during active treatment—glutamine is abundant in meat. ✅ STUDIES: • Cell Metabolism (2013): Glutamine metabolism is essential for tumor growth in low-glucose environments. • Molecules (2020): Curcumin inhibits glutaminase, the enzyme that converts glutamine to fuel in cancer cells. • Frontiers in Oncology (2022): Mistletoe extract shown to downregulate glutamine pathways in colorectal cancer cells. • Oncotarget (2015): Honokiol targets mitochondrial glutamine metabolism in glioma models. 🔥Cancer adapts fast. But so can you. Block the second fuel… And give your body the upper hand it was designed to have. -Dr Pete Sulack

⚔️ BLOCKING GLUTAMINE NATURALLY When cancer cells can’t get glucose, they often switch to glutamine, a key amino acid involved in cell growth and repair. 🍄 Mistletoe—immune-regulating and disrupts glutamine-dependent cancer growth 🌿 Curcumin—shown to interfere with glutamine metabolism in aggressive tumors 🍵 Green tea (EGCG)—again, powerful here as well 🌿 Berberine—reduces expression of glutamine transporters 🧠 Honokiol (from magnolia bark)—neuroprotective and suppresses glutamine usage 🍄 Turkey tail mushroom—modulates immune response and may slow glutamine-driven cell replication 🥬 Fermented vegetables—rich in butyrate, which opposes glutamine-fueled inflammation 🥥 Coconut oil—encourages ketone metabolism over glutamine ✅ Suggested Stack: • AM: Honokiol + green tea • PM: Curcumin + mistletoe extract (consult with a practitioner) • Daily: Coconut oil for brain fuel + fermented foods for terrain healing 🧬 Avoid excess animal protein during active treatment—glutamine is abundant in meat. ✅ STUDIES: • Cell Metabolism (2013): Glutamine metabolism is essential for tumor growth in low-glucose environments. • Molecules (2020): Curcumin inhibits glutaminase, the enzyme that converts glutamine to fuel in cancer cells. • Frontiers in Oncology (2022): Mistletoe extract shown to downregulate glutamine pathways in colorectal cancer cells. • Oncotarget (2015): Honokiol targets mitochondrial glutamine metabolism in glioma models. 🔥Cancer adapts fast. But so can you. Block the second fuel… And give your body the upper hand it was designed to have. -Dr Pete Sulack

Cleanse Parasites .com 🧹🪱 Herbal Cleanse Co.

138,079 次观看 • 1 年前

Aspirin — the cheap pill sitting in your medicine cabinet — could become a powerful new tool against cancer. A March 2025 study published in Nature found that aspirin helps “unleash” T-cells in the immune system, making them more effective at hunting down and attacking cancer cells, including metastatic ones. Dr. Elisa Port, Chief of Breast Surgery at Mount Sinai, explained on CBS News that this ties into the same mechanism that makes aspirin effective at preventing blood clots and heart disease. The research builds on earlier work showing aspirin’s anti-inflammatory effects can enhance immunotherapy responses by reducing platelet-driven immune suppression. It’s still early stages (mostly animal data so far), and doctors aren’t recommending it for cancer prevention yet — but the potential is intriguing. It’s honestly wild that something so common and inexpensive might play a real role in fighting cancer. Shows how much we still have to discover about everyday medicines. If this holds up in larger human trials, it could offer a low-cost way to support cancer treatment for millions. Would you be excited or cautious if a common drug like aspirin turned out to have real anti-cancer effects?

Aspirin — the cheap pill sitting in your medicine cabinet — could become a powerful new tool against cancer. A March 2025 study published in Nature found that aspirin helps “unleash” T-cells in the immune system, making them more effective at hunting down and attacking cancer cells, including metastatic ones. Dr. Elisa Port, Chief of Breast Surgery at Mount Sinai, explained on CBS News that this ties into the same mechanism that makes aspirin effective at preventing blood clots and heart disease. The research builds on earlier work showing aspirin’s anti-inflammatory effects can enhance immunotherapy responses by reducing platelet-driven immune suppression. It’s still early stages (mostly animal data so far), and doctors aren’t recommending it for cancer prevention yet — but the potential is intriguing. It’s honestly wild that something so common and inexpensive might play a real role in fighting cancer. Shows how much we still have to discover about everyday medicines. If this holds up in larger human trials, it could offer a low-cost way to support cancer treatment for millions. Would you be excited or cautious if a common drug like aspirin turned out to have real anti-cancer effects?

Camus

29,301 次观看 • 3 个月前

Every hour, 70 people die from cancer in the United States. That’s roughly 1,700 lives every day. When I read those stats, I realised how overwhelming it can feel to try understand this disease and why it can feel almost impossible to navigate the science behind it. That is why I invited Professor Thomas Seyfried back to try and understand the science further, question the system and explore what we may still be getting wrong. Thomas has spent 50 years researching cancer. He is a Professor of Biology at Boston College, the author of more than 150 peer-reviewed papers and one of the most passionate people I have met when speaking about this disease. His work explores a metabolic view of cancer. In simpler terms, he believes we need to pay much closer attention to how cancer cells create energy, how their metabolism differs from healthy cells and what that could mean for future research. We discussed things like: - The new research he brought into the studio in a confidential envelope… - How damaged mitochondria could change the way we understand cancer. - The three things he believes matter most for living longer. - Why cutting sugar alone does not fully explain how cancer cells survive. - Where keto can become dangerous or misunderstood?! - Changes he would make if he were responsible for public health. There were moments when I asked Thomas to simplify his explanations and pushed for clarity on what's been demonstrated in research and what still needs further study. That distinction matters. Thomas's work is focused on scientific research, not clinical practice. I left this conversation thinking about how progress often begins - someone asks a question that doesn't fit comfortably inside the existing explanation, then spends decades trying to answer it. Cancer will affect almost every family in some way. This is a challenging conversation, but one I believe is worth hearing in full. *The views expressed are those of the guest and this conversation is intended for general informational purposes only. This podcast and its associated materials should not be used as a substitute for professional medical advice, diagnosis, or treatment.

Every hour, 70 people die from cancer in the United States. That’s roughly 1,700 lives every day. When I read those stats, I realised how overwhelming it can feel to try understand this disease and why it can feel almost impossible to navigate the science behind it. That is why I invited Professor Thomas Seyfried back to try and understand the science further, question the system and explore what we may still be getting wrong. Thomas has spent 50 years researching cancer. He is a Professor of Biology at Boston College, the author of more than 150 peer-reviewed papers and one of the most passionate people I have met when speaking about this disease. His work explores a metabolic view of cancer. In simpler terms, he believes we need to pay much closer attention to how cancer cells create energy, how their metabolism differs from healthy cells and what that could mean for future research. We discussed things like: - The new research he brought into the studio in a confidential envelope… - How damaged mitochondria could change the way we understand cancer. - The three things he believes matter most for living longer. - Why cutting sugar alone does not fully explain how cancer cells survive. - Where keto can become dangerous or misunderstood?! - Changes he would make if he were responsible for public health. There were moments when I asked Thomas to simplify his explanations and pushed for clarity on what's been demonstrated in research and what still needs further study. That distinction matters. Thomas's work is focused on scientific research, not clinical practice. I left this conversation thinking about how progress often begins - someone asks a question that doesn't fit comfortably inside the existing explanation, then spends decades trying to answer it. Cancer will affect almost every family in some way. This is a challenging conversation, but one I believe is worth hearing in full. *The views expressed are those of the guest and this conversation is intended for general informational purposes only. This podcast and its associated materials should not be used as a substitute for professional medical advice, diagnosis, or treatment.

Steven Bartlett

44,295 次观看 • 1 个月前

🚨Did you know that fructose can fuel cancer by hijacking your liver? (🔗 at end) 1/5) It sounds dramatic, but new research - published in nature has uncovered a chilling new connection between fructose and cancer growth. And it’s not just about sugar making us gain weight; the effects are far deeper and more direct. 🧵Let me break it down. But first, background… High fructose diets are already associated with increased risks of certain cancer, like pancreatic cancer. And there are many known mechanisms whereby fructose can support cancer growth and survival. 🤨But I particularly like these new data, because they reveal just how sneaky cancer can be, effectively hijacking the liver for its own purposes

🚨Did you know that fructose can fuel cancer by hijacking your liver? (🔗 at end) 1/5) It sounds dramatic, but new research - published in nature has uncovered a chilling new connection between fructose and cancer growth. And it’s not just about sugar making us gain weight; the effects are far deeper and more direct. 🧵Let me break it down. But first, background… High fructose diets are already associated with increased risks of certain cancer, like pancreatic cancer. And there are many known mechanisms whereby fructose can support cancer growth and survival. 🤨But I particularly like these new data, because they reveal just how sneaky cancer can be, effectively hijacking the liver for its own purposes

Nick Norwitz MD PhD

49,038 次观看 • 1 年前

Mr. Annamalai is a great politician, however, his claims suggesting that 24-hour or 7-day fasting can "kill all cancer cells" are scientifically inaccurate and potentially harmful to patients. 1. The Warburg Effect: It is true that many cancer cells consume more glucose than normal cells. However, they are highly adaptable. Simply "starving" them through fasting does not consistently kill them, as they can often find alternative ways to survive. 2. Fasting is not a Standalone Cure: While research (like that of Dr. Valter Longo) explores how fasting-mimicking diets can assist chemotherapy by protecting healthy cells, it is not a replacement for medical treatment. A 24-hour fast is not a "kill switch" for cancer. 3. The Danger of Misinformation: Claiming a 7-day fast can eliminate "all cancer" is a dangerous overreach. For many patients, prolonged fasting can lead to severe muscle loss (cachexia) and weakened immunity, which are leading causes of death in cancer patients. Conclusion: Nutrition plays a vital role in cancer care, but medical advice should come from oncologists and registered dietitians. Cultural practices like fasting have spiritual value but should not be mistaken for a medical cure for a complex disease like cancer. (Source of video: Sun News)

Mr. Annamalai is a great politician, however, his claims suggesting that 24-hour or 7-day fasting can "kill all cancer cells" are scientifically inaccurate and potentially harmful to patients. 1. The Warburg Effect: It is true that many cancer cells consume more glucose than normal cells. However, they are highly adaptable. Simply "starving" them through fasting does not consistently kill them, as they can often find alternative ways to survive. 2. Fasting is not a Standalone Cure: While research (like that of Dr. Valter Longo) explores how fasting-mimicking diets can assist chemotherapy by protecting healthy cells, it is not a replacement for medical treatment. A 24-hour fast is not a "kill switch" for cancer. 3. The Danger of Misinformation: Claiming a 7-day fast can eliminate "all cancer" is a dangerous overreach. For many patients, prolonged fasting can lead to severe muscle loss (cachexia) and weakened immunity, which are leading causes of death in cancer patients. Conclusion: Nutrition plays a vital role in cancer care, but medical advice should come from oncologists and registered dietitians. Cultural practices like fasting have spiritual value but should not be mistaken for a medical cure for a complex disease like cancer. (Source of video: Sun News)

Dr Sudhir Kumar MD DM

57,777 次观看 • 7 个月前

For most women, the benefits of taking body identical HRT far outweigh any risks. Yet many are reluctant to take it and many doctors are also reluctant to prescribe it. This is usually due to a false belief that HRT is too risky and causes breast cancer. I am sharing with you this paper that has recently been published looking at oestrogen-only HRT and breast cancer incidence. The paper is a meta-analysis, which is a type of statistical analysis where researchers review, combine, and analyse multiple studies together. In this paper, researchers have looked at randomised studies involving oestrogen and breast cancer incidence and put them together to see what the results show. The conclusion is clear: women who take oestrogen-only HRT have a LOWER future risk of developing breast cancer. We’ve known for several years that women who take oestrogen have a lower future incidence of breast cancer as well as other cancers and other conditions. This paper is very reassuring, and it’s worth remembering that any risk of breast cancer has been shown using oestrogen with a combination of a synthetic progestogen, not the body-identical type progesterone healthcare professionals usually prescribe.

For most women, the benefits of taking body identical HRT far outweigh any risks. Yet many are reluctant to take it and many doctors are also reluctant to prescribe it. This is usually due to a false belief that HRT is too risky and causes breast cancer. I am sharing with you this paper that has recently been published looking at oestrogen-only HRT and breast cancer incidence. The paper is a meta-analysis, which is a type of statistical analysis where researchers review, combine, and analyse multiple studies together. In this paper, researchers have looked at randomised studies involving oestrogen and breast cancer incidence and put them together to see what the results show. The conclusion is clear: women who take oestrogen-only HRT have a LOWER future risk of developing breast cancer. We’ve known for several years that women who take oestrogen have a lower future incidence of breast cancer as well as other cancers and other conditions. This paper is very reassuring, and it’s worth remembering that any risk of breast cancer has been shown using oestrogen with a combination of a synthetic progestogen, not the body-identical type progesterone healthcare professionals usually prescribe.

Dr Louise Newson

23,788 次观看 • 2 年前

🚨 Ivermectin for Cancer: The Important Part Most People Miss Ivermectin is being explored for its potential anti-cancer effects; it can disrupt cancer cell mitochondria, interfere with glucose metabolism, and help reactivate apoptosis (the cell’s natural self-destruct system). But here’s what often gets overlooked: Ivermectin can also place extra demand on your body’s detoxification pathways. Supporting your terrain while using it is essential. Here’s how this person supports their body during use: Liposomal glutathione + NAC for liver and mitochondrial support Binders like activated charcoal and fulvic acid to help with detoxification Cycling protocol: 5 days on, 2 days off As they put it: “Ivermectin may help starve the tumor, but don’t starve your terrain in the process.” This approach combines the potential benefits with practical ways to protect overall health. Follow me for more updates.

🚨 Ivermectin for Cancer: The Important Part Most People Miss Ivermectin is being explored for its potential anti-cancer effects; it can disrupt cancer cell mitochondria, interfere with glucose metabolism, and help reactivate apoptosis (the cell’s natural self-destruct system). But here’s what often gets overlooked: Ivermectin can also place extra demand on your body’s detoxification pathways. Supporting your terrain while using it is essential. Here’s how this person supports their body during use: Liposomal glutathione + NAC for liver and mitochondrial support Binders like activated charcoal and fulvic acid to help with detoxification Cycling protocol: 5 days on, 2 days off As they put it: “Ivermectin may help starve the tumor, but don’t starve your terrain in the process.” This approach combines the potential benefits with practical ways to protect overall health. Follow me for more updates.

Dr. Zakaria MD

14,209 次观看 • 1 个月前