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Retatrutide forever, watch this (Spoiler alert science agrees) Here’s everything you need (it’s free!) Think of it like this… Biology is a building… Drugs are trying to fix one room Retatrutide handles everything… At the same time Most doctors and Instaexperts talk about it without actually understanding biology or...

19,375 просмотров • 1 день назад •via X (Twitter)

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🚨ImmunityBio’s Anktiva: A New FDA-Approved Immunotherapy for certain bladder cancer. They are also developing treatments that may reduce the need for strong chemo, for blood cancer, lung cancer, brain cancer, and more. Broader approvals may take time. Someone call the President and fast-track this. Dr. Pat Soon-Shiong leads development beyond typical biotech efforts. Most cancer medicines, like chemotherapy, work by poisoning fast-growing cells to kill the cancer. This also hurts healthy cells, causing side effects like hair loss and weakness. Anktiva is different. It is an immunotherapy that wakes up your body’s own immune cells; natural killer cells and T cells to hunt and destroy cancer cells themselves. It acts like a booster shot for your immune system, using a protein called IL-15 to make these cells multiply and remember how to fight cancer long-term. ImmunityBio is different from most companies because of 3 big unique things: 1. They wake up 3 kinds of fighter cells at the same time: • NK cells (natural killers) • CD8 T cells (main attackers) • CD4 T cells (helpers) These three work together like a basketball team. This makes the body remember the cancer for a long time (maybe years). 2. They have a special cell line called NK-92. → It’s like buying frozen pizza instead of making dough from scratch every time. Doctors can just thaw it and give it to any patient. No need to take cells from the sick kid, customize them, and make them super weak with hospital chemo (that’s what normal CAR-T does). 3. Their medicine Anktiva gives a long, steady boost to the immune cells. Older drugs (like IL-2) gave a short, crazy boost that made people very sick. Anktiva is seems safer, so far. Exciting to follow the development. Interview: Chris Cuomo (NewsNation) — “Killing Cancer”

Black Panther Capital

40,249 просмотров • 6 месяцев назад

🚨Here's what a lot of people misunderstand about cancer treatment, says drpaulmarik: "Cancer is not homogeneous. The somatic mutation theory—which is the current theory in which treatment is based—posits that you have a mutation in a single cell, and that gives rise to a whole population of cells that look the same and have the same mutation. But the Cancer Genome Atlas has shown that that theory is completely wrong. The cancer cells are very heterogeneous, so they're made up of very different populations of cells with different mutations, and one of the populations is the cancer stem cell. It's a sub-population of the cancer. These are generally slow-growing, but they're distinct in that they have the ability to divide indefinitely and grow indefinitely, and can change their characteristics. Basically, if you get rid of the fast-dividing cells, which is the cancer, you're left with the stem cells, which then become the roots, which grow back to form the tumor" sometimes years later. Conventional chemotherapy gets rid of the fast-dividing regular cancer cells but *NOT* the stem cells. So the key question is: how do you get rid of the stem cells? “There are a number of repurposed drugs that do it, and this has been well-established in scientific medical literature. One of the most effective treatments to knock out the stem cell is the famous horse deworming medicine," says drpaulmarik. Yes, ivermectin. Independent Medical Alliance

Jan Jekielek

96,002 просмотров • 1 год назад

🚨 WHY ARE THERE OVER 400 PUBLISHED PAPERS ON IVERMECTIN AND CANCER? Because researchers discovered something unexpected. Ivermectin doesn't just target parasites. According to preclinical research, it appears to interact with cancer cells through multiple biological pathways. One of the most discussed? 📌 Cancer Stem Cells These are the cells believed to survive treatment, remain dormant for years, and potentially contribute to recurrence and spread. According to Dr. William Makis: 💊 Chemotherapy primarily targets rapidly dividing cancer cells. 💊 Ivermectin has been studied for its potential effects on cancer stem cells. That's why some researchers believe the combination deserves attention. But that's only part of the story. Researchers have also investigated ivermectin's potential ability to: ✅ Target cancer stem cells ✅ Influence tumor signaling pathways ✅ Alter cancer cell behavior ✅ Reverse chemotherapy resistance One of the most fascinating findings involves chemotherapy resistance. Over time, some cancer cells develop the ability to push chemotherapy drugs back out of the cell. Researchers have reported that ivermectin may interfere with these resistance mechanisms, potentially making cancer cells susceptible again. And then there's Fenbendazole and Mebendazole. While Ivermectin is being studied for one set of mechanisms, Fenbendazole and Mebendazole appear to work differently. Researchers have investigated their potential ability to: 📌 Block glucose transporters on cancer cells 📌 Reduce the cell's ability to use glucose as fuel 📌 Interfere with pathways associated with tumor growth Think about that. Different compounds. Different mechanisms. The same goal. That's why scientists continue publishing study after study. Not because these compounds are a fad. Because researchers keep uncovering new biological pathways worth investigating. And that's why the conversation around Ivermectin, Fenbendazole, and Mebendazole continues to grow. 💊 If you’re looking into these drugs, check out Ivermectin, Fenbendazole, and Mebendazole from RXMEDS.STORE. #Ivermectin #Fenbendazole #Mebendazole #CancerResearch #CancerStemCells #RepurposedDrugs #CancerBiology #RxMeds

RXMEDS.STORE

16,612 просмотров • 1 месяц назад

Scientists just figured out how to reverse aging using AI. And this is a massive breakthrough. We can now reprogram any human cell back to age 20. Heart cells, brain cells, skin cells, all reset to their biological prime. And here’s the wildest part…the technology to do this, has already existed since 2012 (it won the Nobel Prize). But the real breakthrough wasn’t possible until this year, when they supercharged it with AI. It’s a wild story. So in 2006, scientists discovered Yamanaka factors. They’re proteins that can basically convert any normal cell into a universal stem cell. Now this was a huge deal, because these stem cells are basically like magic healers. If you have torn muscle tissue, you could inject these stem cells into the area and they will turn into the youthful muscle cells you need. So Yamanaka factors were this insane breakthrough, because they allowed any human to turn any cell you already have into these magic healers. But, there was one big problem… It turns out, the original Yamanaka factors weren’t very good at this stem cell conversion. They could do it, but they just weren’t very reliable. Enter OpenAI...and this is where things get crazy. OpenAI designed a special AI model built specifically to create new proteins. Think of it like ChatGPT but for protein engineering. So they took all the Yamanaka research and asked this new AI to go ham on improving it. And get this… Their version was 50x more effective than the original. They tested it on 50 year old cells and it successfully started repairing 30% of their cells in just 7 days. This is just science fiction…it actually happened. And it sounds crazy, but in a few years, humans will be able to take a shot that will literally reverse the age of their cells.

Whiplash347

68,643 просмотров • 9 месяцев назад

Dr. Patrick Soon-Shiong, a pioneering surgeon and scientist, reveals a shocking truth about chemotherapy, immunity, and why we’ve been fighting cancer the wrong way for decades. Here’s what’s simple—and profound: When you undergo chemotherapy, the first casualty is your red blood cells, leading to anemia (treated with Epogen). Next, it wipes out neutrophils, leaving you vulnerable to infection (treated with Neupogen). But the most devastating effect? It destroys your NK and T cells—your body’s elite cancer-fighting forces. Think about that: The very treatment meant to kill cancer also annihilates the only cells that can actually protect you from it. For 35 years, medicine has ignored this paradox. We replace red blood cells and neutrophils—but not the lymphocytes that matter most. The result? A weakened immune system, vulnerable to relapse and secondary cancers. Now, consider long COVID. The virus is cunning—it targets the same NK and T cells, crippling immunity. The parallel is undeniable: Weakening these cells leaves the body defenseless, whether from chemo or chronic infection. Dr. Soon-Shiong’s message is clear: "We’ve been treating cancer wrong." The future lies in restoring immune function, not just attacking tumors. The fight isn’t just against cancer or viruses—it’s for the immune system itself. And after 20 years of challenging the status quo, Dr. Soon-Shiong is leading that revolution. It’s time to rethink everything.

Camus

327,541 просмотров • 11 месяцев назад

Can people in power/ authority kindly stop with this absolute BS on "fasting killing cancer cells" and citing religious nonsense to appeal to peoples emotions? Fasting is really quite dangerous for cancer patients in real life. Let me explain and bury this myth once and for all, especially for science illiterates like this guy. [1] The most common argument is that fasting "starves" cancer by cutting off its sugar (glucose) supply. While it is true that cancer cells consume vast amounts of glucose, they are biologically aggressive survivalists. If you stop eating, your body eventually switches to burning fat and breaking down muscle for energy. Cancer cells are highly adaptable; when glucose is low, many types of cancer can mutate to feed on other fuel sources, such as lactate, amino acids (from your muscles), or fatty acids. You cannot simply "starve" a tumor without starving the patient first. [2] Fasting is dangerous for cancer patients because of cancer cachexia - a wasting syndrome where the body loses muscle and fat rapidly. Cachexia is responsible for up to 30% of cancer deaths. Cancer puts the body in a hyper-metabolic state (burning energy fast). If a patient fasts, they risk accelerating muscle loss and weakening their immune system. A weak body cannot tolerate life-saving treatments like chemotherapy or radiation, nor can it fight off infections. [3] Most claims about fasting curing cancer come from studies on mice or cells in a petri dish. In a dish: You can kill cancer cells with almost anything (lemon juice, bleach, starvation, even shooting a bullet at it at close point or using a grenade to destroy the entire lab) because they have no immune system or body to protect them. In a human: The biology is infinitely more complex. Human metabolism, hormonal fluctuations, and tumor micro-environments mean that what shrinks a tumor in a mouse often fails completely in human trials. [4] Proponents often cite "autophagy" (the body's cellular recycling process triggered by fasting) as the cure. They claim it cleans out cancerous cells. Science shows that autophagy is a double-edged sword. -In all people, autophagy is a normal physiological process - whether fasting or not, which help in cleaning up damaged cells. -But in patients with cancer, once a tumor exists, cancer cells can actually hijack autophagy to survive stress (like chemotherapy) and repair themselves. In this context, fasting could theoretically help the cancer survive the treatment intended to kill it. [5] "Cancer" is not one disease; it is over 200 different diseases characterized by uncontrolled cell growth. Some cancers are driven by hormones, some by genetic mutations, and some by viruses. A fasting protocol that slows down one specific type of breast cancer might have zero effect on pancreatic cancer, or worse, accelerate a different type. The most lethal aspect of this misinformation is the delay in treatment. Cancer is a time-sensitive disease. While a patient spends months trying to fast the cancer away based on religious or alternative advice, the cancer often metastasizes (spreads) to other organs. Once cancer spreads, it often moves from being curable to being terminal. Relying solely on fasting wastes the critical window where medical intervention could have saved a life. Suggesting a single "ancient cure" for 200 complex genetic diseases is scientifically illogical... ...and generally stupid, as this video proves.

TheLiverDoc™

193,243 просмотров • 6 месяцев назад

🚨Big problem! Dr. Suzanne Humphries: "Here's the real sucker punch... the Covid jabs lead to increased cancer susceptibility... but SV40-associated cancer won't respond well... to chemotherapy... [in fact,] radiation makes SV40 cancer far more aggressive...." This clip of Dr. Humphries (Dr Suzanne Humphries), a medical doctor, board-certified internist, nephrologist, and co-author of Dissolving Illusions, is taken from a presentation posted to the Children's Health Defense (Children’s Health Defense) Rumble channel on November 12, 2025. ---------------Partial transcription of clip--------------- "And here's the real sucker punch. So you remember how SV40 knocks out that p53 gene and protein, which I talked to you before, the cancer suppressor. And that means that not only can the Covid jabs lead to increased cancer susceptibility, right? But SV40-associated cancer won't respond well at all to chemotherapy. "And conventional cancer treatments are likely to make cancer actually go wild and not regress because p53, it actually directs cancer cells to die off. It's necessary for chemo and especially for radiation to do the killing that they're supposed to be doing. And the fact that radiation makes SV40 cancer far more aggressive is the entire reason it was used in the Get Castro project, right? You know, radiating the animals first and then doing the gain of function with radiation. That's why they did it. "So you know, this is some older material, just to show you that they've known for a long time that SV40 blocks the effect of cancer treatments. And so there have been children who have died of SV40-related brain tumors. And the parents go and they do research. And this is what they have found. And so these are articles from like 1997. "So again, this is not something that top scientists should have bypassed. So the other brain cancers and solid cancers have been found to contain the SV40. And that the SV40 binds with the tumor-suppressor gene at p53, and it actually stops those tumor cells from undergoing what's called apoptosis, A-P-O-P-T-O-S-I-S, which really means programmed cell death. And that's how you're able to get cancer to regress, you know, when you're doing whatever you're doing traditionally. "Like, even if it's like, look, we know radiation does make tumor cells regress. Do they come back later? Most of the time, yes, they do cancer, you know, chemotherapy. Like, my father got chemotherapy when he had tumors all over his body. We got nine extra months with him, so I was okay with that. But did they all come back to kill him? Yes, they did, but I got an extra nine months. "But the point being that, you get that programmed cell death. But then if there's a P— If it's an SV40-related cancer especially, you're going to wind up with probably a turbo cancer at that point. "Yeah, I'm just going to read this to you because when I said that people knew about this and there were, you know, parents of children who developed brain tumors and testified in front of Congress. This was one of the very well-educated parents. Exposing SV40-positive cancer cells to chemo and radiation does not kill the cells, but simply creates more genetic mutations, making the cancer more aggressive. The bottom line is that SV40 causes human cancer, stops orthodox cancer treatments like chemo and radiation from providing any benefit and can make the cancer even more aggressive."

Sense Receptor

54,169 просмотров • 8 месяцев назад

"I think deuterium is the reason why you have cancer." — Stephanie Seneff, MIT researcher. Deuterium is a heavy form of hydrogen naturally present in water and food. Your mitochondria are extremely sensitive to it — too much deuterium disrupts their ability to produce ATP and triggers excess reactive oxygen species. When deuterium accumulates systemically, every cell in your body starts struggling. Seneff's hypothesis: A cell senses the overload and transforms itself into a cancer cell. Not to harm you. To help you. Cancer cells abandon their normal function and obsess on one thing: duplicating themselves. Their metabolism shifts entirely. They suppress oxidative phosphorylation — the process by which mitochondria generate ATP using oxygen — repurposing them toward anabolic synthesis — to avoid the reactive oxygen species that high deuterium would generate. Instead they run glycolysis. Massive glucose intake. The output: lactate — carrying a deuterium-depleted proton — shipped out into circulation. Low-deuterium fuel delivered to the host. The cancer cell also relocates its V-ATPase pumps — protein pumps embedded in the cell membrane — to the outer surface, pumping deuterium-depleted protons directly into the tumor microenvironment — while hoarding deuterium inside itself. It is self-sacrificial. Taking on the burden so the rest of the body doesn't have to. Immune cells flood the tumor. But they don't attack. The cancer is nourishing them — lactate and deuterium-depleted protons — providing what their damaged mitochondria need to recover. Seneff notes the same lactate and low pH environment also signals immune cells to stand down — suppressing activation and allowing the tumor to survive in the process. Once the immune cells recover, they turn on the tumor and clear it. When deuterium levels drop low enough — the cancer cell's job is done. It undergoes apoptosis. Gabor Somlyai, Hungarian biochemist and cancer researcher showed that when cancer cells are placed in deuterium-depleted water, they stop multiplying and undergo apoptosis. In high-deuterium water — they thrive. He documented patients rejected by mainstream oncology — told to go home and die. They began drinking deuterium-depleted water. Some lived far beyond predicted life expectancy. Some achieved complete recovery. This also might explain why the ketogenic diet works against cancer. Animal fats are the lowest deuterium macronutrient. A ketogenic state naturally lowers systemic deuterium intake. Combined with glucose restriction — cancer cells depend heavily on glucose to run glycolysis — both mechanisms rest on the same biology. Thomas Seyfried, Professor of Biology at Boston College, reached the conclusion that cancer is a mitochondrial metabolic disease, not a genetic one. Seneff goes one step further: deuterium overload is why the mitochondria malfunction in the first place. According to her, cancer isn't a random malfunction. It's a coordinated biological response to a systemic deuterium overload.

no.mind

190,791 просмотров • 3 месяцев назад

New Research Deep Dive: The "Shedding" Conversation Just Got More Serious A new in-vitro study (using human cells in a lab) on the Pfizer mRNA vaccine has revealed critical findings that can no longer be ignored. Let's break it down. The researchers confirmed two major things: 1️⃣ Spike Protein Production: The cells successfully took up the mRNA and began producing the SARS-CoV-2 spike protein, displaying it on their surface. This was expected. 2️⃣ Spike Protein "Shedding" via Exosomes: Here's the crucial part. The cells didn't just keep the spike protein to themselves. They packaged it into exosomes—tiny extracellular vesicles cells use to communicate—and excreted them into the environment. Why does this matter? This provides a potential mechanistic blueprint for how spike protein could travel systemically throughout the body after vaccination. These spike-laden exosomes can enter the bloodstream and, theoretically, deliver their cargo to distant organs and other cells. But the most alarming part? The authors note a profound lack of safety data. They explicitly state: We have no scientific studies to determine if this exosome-mediated spread of spike protein is toxic to other human cells. Even more concerning, they observed "pathological changes" and toxicity within the cells producing the spike. And these weren't weak cells—they were robust, immortalized embryonic kidney cells, chosen for their resilience. If these cells showed adverse effects, what is the impact on our more delicate primary cells? The authors themselves caution that proper toxicology studies on normal human cell lines are urgently needed... and are currently unavailable. This isn't conspiracy theory. This is cell biology. The conversation must evolve from if spike protein can travel, to what are the systemic consequences when it does. The call for rigorous, independent safety science has never been louder.

Camus

48,219 просмотров • 8 месяцев назад

Cancer Is Largely Preventable—Here’s the Science-Backed Solution For decades, we’ve been told cancer is a genetic disease—inescapable, unpredictable, a roll of the dice. But what if that’s wrong? Dr. Thomas Seyfried, a leading cancer researcher, reveals a paradigm-shifting truth: Cancer is a metabolic disease, fueled by the very foods and lifestyles we’ve been conditioned to accept. 🔬 The Science Doesn’t Lie: - High blood sugar = Faster tumor growth. - Low blood sugar = Slower tumor growth. - Cancer cells thrive on glucose & glutamine—but they can’t efficiently use ketones. The Solution? Starve the Tumor. Cancer cells have broken mitochondria, forcing them to rely on fermentation (sugar & glutamine) for survival. But healthy cells can thrive on ketones. This is the key. ✅ How to Shift the Battlefield: 1. Water-only fasting – Deprives tumors of fuel. 2. Ketogenic diet – Replaces glucose with ketones, starving cancer while nourishing the body. 3. Exercise & mitochondrial health – Vigorous movement repairs cellular energy systems. 4. Ditch processed carbs – They feed dysfunction at the deepest level. The result? Tumors lose their energy supply, blood vessels shrink, and the body naturally dismantles them. 🔥 This isn’t theory—it’s biochemistry. The war on cancer isn’t about toxic drugs or blind luck. It’s about controlling the metabolic environment. Want to slash your risk? ✔ Fast strategically. ✔ Go low-carb, high-fat. ✔ Move like your life depends on it. Cancer isn’t just “bad genes.” It’s bad metabolism. And that means you have power over it. — Dr. Thomas Seyfried

Camus

290,791 просмотров • 1 год назад

How much physical energy does it ACTUALLY cost you to worry about the future? Dr Martin Picard's research argues stress isn't just a mental weight - it's an expensive biological tax on your cells. In his lab, when human cells were exposed to stress hormones, the energy cost of just staying alive inside those cells shot up by 60% (He’s also the scientist whose team literally sliced a human brain into 703 cubes just to map how energy moves through it 🤯) You have a stressful morning, a difficult conversation, or a few anxious thoughts you can't shake, and by 2pm you feel completely drained. Your mind is tired, but your cells are genuinely exhausted. Dr Martin Picard is a Professor of Behavioural Medicine at Columbia University and I sat down with him to understand how our emotions and lived experiences physically change the mitochondria inside us - the tiny structures responsible for producing all the energy we rely on. My energy has always felt a bit like roulette. Some days I wake up feeling incredible, other days I feel like I've done the exact same things but I've got nothing in the tank. Martin completely reframed how I look at this. Energy isn't just a biological default; it's one of the most important scientific questions of our lives. We discussed things like: - Why you can do everything “right” (sleep, diet, exercise) and still wake up completely exhausted. - The physical tax of stress and what a single anxious morning does to your cells. - Why mitochondria are the missing link behind burnout, depression, and chronic fatigue. - What actually dictates your cellular energy day-to-day. - The truth about whether fasting, meditation, deep purpose and relationships can rewrite your biology. This is a conversation about understanding what your body is constantly responding to where your energy is actually going and why so many of us feel tired in ways that more sleep or more food don't always solve. If you feel flat, anxious, or exhausted and can't explain why - this one's for you.

Steven Bartlett

160,512 просмотров • 1 месяц назад

Oncologist Dr. William Makis: "The turbo cancers after the COVID vaccines do respond to the antiparasitics, and they respond very well... [And] why do these antiparasitics work?" Todd Callender: "Cancer's a parasite?" Makis: "That's one possibility." This clip of Dr. Makis (William Makis), a radiologist, oncologist, and cancer researcher, is taken from an interview with attorney Todd Callender posted to the VaxxCHOICE (VaxxCHOICE) Rumble channel on November 21, 2025. ---------------Partial transcription of clip---------------- Dr. Makis: "The turbo cancers after the COVID vaccines do respond to the antiparasitics and they respond very well. This is, you know, whether it's ivermectin, fenbendazole or mebendazole, these patients are responding. "Now this is very important because patients who've developed cancer after taking Covid vaccines, they don't respond usually to conventional treatments. That's one of the hallmarks of turbo cancer is they present at a very late stage. These are extremely rapidly growing tumors. They're coming in at stage four. And it's whether it's young women with breast cancer, women in their 20s, something we've never seen before, young women with, men and women with colon cancer in their 20s and 30s. "Again, we've never seen—" Callender: "Two- year-olds, I've heard of two- year-olds with colon cancer—" Dr. Makis: "—we've never seen this before. That's the incredible part. And they're not responding to conventional treatments. So, they're not responding to chemo, they're not responding to radiation therapy, immunotherapy, or if they do response, they have a very, very short response time or remission time. And then the cancer comes roaring back. "And in a lot of these tragic cases, the patient dies within six months. This is another one of the features of turbo cancer is the prognosis is extremely poor if you go the conventional route. So you need something else. Now when you're using antiparasitics, for cancer, the dose is higher than the dose you would use to treat parasites or to treat Covid. So I give just a rule of thumb for ivermectin, for example, it's about five times the dose that you would need for cancer, about 5 to 10 times the dose than if you were just treating parasites or viruses. "And to go into the— why does it work? Why do these antiparasitics work?" Callender: "Cancer's a parasite?" Dr. Makis: "Well, that's one possibility. But there's more to it than that because, out of the 400 papers that have been published, a lot of those researchers look at the mechanisms. They are trying to identify the mechanisms of how ivermectin is treating these cancers. "And you've got, for example, there was one study published a few years ago, Mexican researchers, again, you're not going to see this done in the United States, Mexico. Mexican researchers took 28 different cancer cell lines, applied ivermectin to them, and all of them responded. Now to various degrees, you had breast cancer and ovarian cancer actually responding the most to ivermectin. You had the most cancer cell killing when they were exposed to ivermectin. "But when they look at the mechanisms, they've identified a number of interesting things. For example, ivermectin shuts down and kills cancer stem cells. Now, this is important because cancer stem cells are the reason chemotherapy doesn't work. When you have stage four pancreatic cancer, stage four ovarian cancer, and you go to your oncologist, they will tell you, we will give you chemo, but we can't cure you. It's palliative chemo. It'll buy you an extra six months, an extra 12 months of life. "The reason why they say that is why the chemo is palliative, not curative. It's is because chemo cannot kill cancer stem cells, which are not rapidly dividing. Chemo will kill everything that's rapidly dividing, but cancer stem cells are not rapidly dividing. And so the chemo will kill the rapidly dividing tumor cells, and it'll leave the cancer stem cells alone, while the cancer stem cells will start rapidly dividing a year later, two years later, they're going to spread to other parts of the body. Suddenly you've got recurrence, you've got metastases. Ivermectin will shut down and kill cancer stem cells. "So imagine you, added to your chemo regimen, and now you've essentially turned what's palliative chemo or a palliative situation to potentially a curative situation. And I really do believe I've come to the point, having helped over 7,000 cancer patients in the last year, I've come to the point where I could confidently say that stage 4 pancreatic cancer is curable, stage 4 ovarian cancer is curable, stage 4 colon cancer, lung cancer. These cancers are curable. I think if you add repurposed drugs, you add antiparasitics, you can turn these into curable situations. "Now, of course, we have to prove that. We have to do the research, do the publications. But in the meantime, I say stage 4 cancer patients don't have 10 years to wait for mainstream oncology or mainstream medicine to catch up with what we're seeing already clinically, in practice."

Sense Receptor

96,616 просмотров • 8 месяцев назад

7 Compelling Reasons Why Ivermectin Is a ‘Powerful Drug’ for Fighting Cancer Ivermectin is often recognized–2nd to penicillin–for having the greatest impact on human health. Its discovery even won the Nobel Prize. It is safer than Tylenol and brought river blindness to the brink of extinction, but the propagandists told you it was a “dangerous horse dewormer.” Well, it now turns out that ivermectin is not only an effective treatment for COVID-19, but it could also be a “powerful drug” for fighting cancer. Here are 7 reasons why. 1. Multiple Anti-Cancer Effects: Ivermectin impacts at least nine well-defined cancer targets, according to Dr. Alfonso Dueñas-González, an oncologist and senior researcher. He states, "There are at least nine perfectly defined cancer targets affected by ivermectin." 2. Enhances Effects of Chemotherapy and Radiation: Ivermectin not only targets tumor stem cells and promotes cancer cell death but also "enhances the effects of chemo and radiation therapy." This broad impact on the immune system increases the offense against cancers, making it a versatile adjunct to conventional treatments. 3. Inhibits Cancer Cell Cycles: The drug prevents the formation of new cancer cells by inhibiting cancer cell cycles and inducing mitochondrial stress, which leads to the killing of cancer cells. This mechanism is crucial for stopping the proliferation of cancerous cells. 4. Prevents Formation of New Blood Vessels Near Cancer Cells: Ivermectin prevents cancer survival by inhibiting the formation of new blood vessels that transport energy and fuel to cancers. This action starves the cancer cells, hindering their growth and spread. 5. Synergizes with Immunotherapy: Dr. Peter P. Lee, chair of immuno-oncology at the City of Hope, found that ivermectin could synergize with immune checkpoint inhibitor anti-PD1, enhancing the body's immune response to fight cancer. "What we’re learning is that ivermectin is going to be a very powerful drug in the context of really carefully developed immunotherapy combinations," he said. 6. Targets Cancer Stem Cells: Ivermectin preferentially targets cancer stem cells, which are a driver of cancer tumors and relapses. By focusing on these cells, ivermectin attacks the root of cancer's resilience and ability to recur. 7. The Curious Case of Rick Alderson: Mr. Alderson, a retired sawmill worker, “was a dead man walking” and given 6 months to live after his terminal colon cancer had metastasized and spread to his liver, where it had formed 25 distinct tumors. After starting ivermectin, Rick Alderson's carcinoembryonic antigen (CEA) levels, which are markers for tumor activity, dropped significantly from 1,498 ng/mL to 13.9 ng/mL in a matter of months. Of the 25 tumors in Mr. Alderson's liver, only three remained after his treatment with ivermectin and chemotherapy, indicating a substantial reduction in tumor burden. Mr. Alderson lived for two more years beyond his initial six-month prognosis, which his wife attributes to the use of ivermectin alongside chemotherapy.

The Vigilant Fox 🦊

532,851 просмотров • 2 лет назад

🚨 “THEY CALLED IT HORSE PASTE… WHILE SCIENTISTS WERE STUDYING IT FOR CANCER AND CHEMO RESISTANCE.” That’s why the Ivermectin conversation refuses to die. According to researchers studying repurposed medicine, Ivermectin is one of the most fascinating molecules ever discovered because it appears to interact with completely different biological pathways depending on the disease being studied. In parasites, researchers found it affects chloride channels. In viruses, studies explored how it may interfere with replication pathways. And in cancer research, scientists have investigated how Ivermectin may interact with tumor proliferation pathways, cancer stem like cells, and chemotherapy resistance mechanisms. Researchers believe cancer stem cells may play a major role in: • Recurrence • Metastasis • Treatment resistance Traditional chemotherapy mainly targets rapidly dividing cells, but scientists are now studying whether Ivermectin may affect more resistant stem like cancer cells differently in laboratory settings. There are also preclinical studies investigating how Ivermectin may help restore chemotherapy sensitivity in resistant tumor cells at the molecular level. And then there’s another reason people keep talking about it: Some researchers have explored whether the same pathways involved in abnormal tumor growth may also be involved in conditions like fibroids, lipomas, and endometriosis. That’s why the repurposed medicine conversation has exploded far beyond COVID. If you’re researching Ivermectin, Fenbendazole, or Mebendazole, many choose PharmacyinUSA for worldwide and discreet delivery. #Ivermectin #HorseDewormer #CancerCure

PharmacyinUSA

15,268 просмотров • 2 месяцев назад

A 2025 Nature study revealed a surprisingly simple way aspirin might help fight cancer metastasis. Researchers discovered that cancer cells trick blood vessels into releasing a substance called thromboxane A2 (TXA2). This chemical then sends a signal that basically tells our immune system’s T-cells to “stand down,” making it easier for cancer to spread. Science nugget: In mouse models of breast, skin, and bowel cancer, aspirin blocked TXA2 production. This freed up the T-cells to attack more effectively, resulting in significantly fewer metastases. When scientists genetically removed the key protein (ARHGEF1) that receives the signal, metastasis dropped sharply — and aspirin had no extra effect, proving this is the main pathway. The study helps explain why some earlier human observational data showed potential protective effects (especially for colorectal cancer). However, these promising results are still from mice, and experts stress that we need proper clinical trials in humans to confirm who might benefit and what the risks are. Any use of aspirin for cancer-related reasons should only happen after talking to your doctor, due to side effects like increased bleeding risk. It’s a fascinating reminder that an old, cheap drug might still have hidden powers we’re only beginning to understand. Does this aspirin-cancer connection surprise you, or does it make you curious about what other everyday medicines might have undiscovered effects?

Camus

268,189 просмотров • 4 месяцев назад