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New Raid Quest "vs Cell Max" is now underway in #DBXV2 ! Cell Max has appeared as a raid boss! Beware of high power and wide range of attacks, and let's all work together to face them! From March 23th (Thu) 8PM to 29th (Wed) 10PM PST/CET Learn more here:

Dragon Ball Games

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118,888 просмотров • 3 лет назад •via X (Twitter)

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This is a work of art 🤌 As a special gift to our community, the Dennis painting & Lava Chicken music disc are now available. Make sure to check them out in-game and see it all in action in #MinecraftMovie now streaming on Max! Learn more:

This is a work of art 🤌 As a special gift to our community, the Dennis painting & Lava Chicken music disc are now available. Make sure to check them out in-game and see it all in action in #MinecraftMovie now streaming on Max! Learn more:

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The most detailed 3D reconstruction of a cell ever created. Blows my mind every time. But what exactly are we looking at here? The average human cell contains: ~ 15-20 total distinct organelle types, totalling between ~1-10 million working together per cell. All these nano-machines in the cell are made up of proteins. ~ 8,000-10,000 distinct types of unique proteins, adding up to between 40 million - 10 trillion total proteins making up all those cellular systems. ~ 10,000 - 15,000 distinct types of RNA shuttling information around the cell, totalling up to ~10 million RNA molecules moving around the cell simultaneously. ~ Billions of Lipid molecules packed together into the cell membrane, which is also packed tightly with millions more protein-based nano-machines. And let's not forget billions of lines of DNA information to build and run it all. That's TRILLIONS of of individual molecular pieces working together to make a single cell function. That means there is more complexity in a single cell than humanity's largest cities. And people still believe this wasn't Divinely Designed. This is God's Glory on Display. But to make the point. A cell couldn't have evolved from some nebulous simpler "protocell" because even the simplest cells still require massive complexity. The "simplest" cell ever created was engineered by scientists knocking out pieces of a functional cell until it stopped functioning. Here is what they found is the absolute necessary minimal requirements of a cell to function: - Over ~531,000 lines of coded DNA information - 473 total genes to create hundreds of unique protein products (they later added 19 genes back in because the cell was so weak) - Hundreds of thousands of total proteins all working together - Extensive regulatory networks guiding all these interactions If the cell doesn't have all these systems in place, from the start... it doesn't live. Cell rely on an intricate network of complex systems, which are themselves built from complex interconnected pieces woven together into an incomprehensibly complex web of functionilty. Only intelligence has ever been observed creation vast interconnected systems like this. Life was clearly Created. It couldn't happen any other way.

The most detailed 3D reconstruction of a cell ever created. Blows my mind every time. But what exactly are we looking at here? The average human cell contains: ~ 15-20 total distinct organelle types, totalling between ~1-10 million working together per cell. All these nano-machines in the cell are made up of proteins. ~ 8,000-10,000 distinct types of unique proteins, adding up to between 40 million - 10 trillion total proteins making up all those cellular systems. ~ 10,000 - 15,000 distinct types of RNA shuttling information around the cell, totalling up to ~10 million RNA molecules moving around the cell simultaneously. ~ Billions of Lipid molecules packed together into the cell membrane, which is also packed tightly with millions more protein-based nano-machines. And let's not forget billions of lines of DNA information to build and run it all. That's TRILLIONS of of individual molecular pieces working together to make a single cell function. That means there is more complexity in a single cell than humanity's largest cities. And people still believe this wasn't Divinely Designed. This is God's Glory on Display. But to make the point. A cell couldn't have evolved from some nebulous simpler "protocell" because even the simplest cells still require massive complexity. The "simplest" cell ever created was engineered by scientists knocking out pieces of a functional cell until it stopped functioning. Here is what they found is the absolute necessary minimal requirements of a cell to function: - Over ~531,000 lines of coded DNA information - 473 total genes to create hundreds of unique protein products (they later added 19 genes back in because the cell was so weak) - Hundreds of thousands of total proteins all working together - Extensive regulatory networks guiding all these interactions If the cell doesn't have all these systems in place, from the start... it doesn't live. Cell rely on an intricate network of complex systems, which are themselves built from complex interconnected pieces woven together into an incomprehensibly complex web of functionilty. Only intelligence has ever been observed creation vast interconnected systems like this. Life was clearly Created. It couldn't happen any other way.

Divinely Designed

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In LumenTale, Trey's journey across Talea will lead him to face a wide range of unique and powerful bosses. Each boss fight is shaped by a set of defining features: ⚔️ Multiple health bars 🔄 Battle Phases that make the enemy more powerful and change their form 🃏 And...more! LumenTale: Memories of Trey is coming on May 26 for Nintendo Switch & Steam. Wishlist now! 🌟

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If you have Cancer, this is your visualization. That is a cancerous cell reaching Apoptosis and Necrosis—they are gone. — Natural Killer (NK) cells play a the primary role in the immune system's defense against cancer. They induce cancer cell death primarily through two mechanisms: Apoptosis Necrosis Apoptosis is a programmed, non-inflammatory form of cell death, while necrosis is a more abrupt and inflammatory process. In many cases, NK cells can trigger a combination of both, leading to mixed forms of cell death. This dual capability allows NK cells to effectively target and destroy all tumor cells. Their ability to act without prior sensitization makes them vital for early cancer surveillance and curing existing cancer. One of the only ways cancer never starts or stops entirely is NK cells. They are the guerrilla warfare troops that never sleep. This is why it is vital for BioShield by to be approved, NOW for every cancer, not just a single cancer type. With the rest needing years and years of tests. Cancer is cancer, unregulated cell division and NK cells are NK cells. The ONLY thing that stops BioShield is government betrayal, and organized corruption disguised as following the “regulations” designed by the entrenched gatekeepers of corporate protection. Everyone knows it works that has even a molecule of biological understanding. Now you know.

If you have Cancer, this is your visualization. That is a cancerous cell reaching Apoptosis and Necrosis—they are gone. — Natural Killer (NK) cells play a the primary role in the immune system's defense against cancer. They induce cancer cell death primarily through two mechanisms: Apoptosis Necrosis Apoptosis is a programmed, non-inflammatory form of cell death, while necrosis is a more abrupt and inflammatory process. In many cases, NK cells can trigger a combination of both, leading to mixed forms of cell death. This dual capability allows NK cells to effectively target and destroy all tumor cells. Their ability to act without prior sensitization makes them vital for early cancer surveillance and curing existing cancer. One of the only ways cancer never starts or stops entirely is NK cells. They are the guerrilla warfare troops that never sleep. This is why it is vital for BioShield by to be approved, NOW for every cancer, not just a single cancer type. With the rest needing years and years of tests. Cancer is cancer, unregulated cell division and NK cells are NK cells. The ONLY thing that stops BioShield is government betrayal, and organized corruption disguised as following the “regulations” designed by the entrenched gatekeepers of corporate protection. Everyone knows it works that has even a molecule of biological understanding. Now you know.

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A single E. coli cell, placed on a dish, will become 70 billion cells in just 12 hours. That’s exponential growth. But a new preprint shows that it's possible to engineer E. coli to grow linearly instead, where only one daughter cell continues dividing and the other stops. First, some context. In nature, there is a bacterium called Mycobacterium smegmatis (initially discovered in 1884 in ulcers scraped from syphilis patients.) M. smegmatis is weird because it divides asymmetrically. These cells grow only from one end, and all their cell wall biosynthesis machinery is located on that one end. So when the cell divides, one daughter gets this machinery and the other gets nothing. The daughter that gets the machinery can keep dividing immediately, but the other daughter has to remake all that machinery from scratch, so its growth is delayed. E. coli doesn’t grow like this. When it divides, it pinches in the middle and splits everything evenly. Enzymes, metabolites, and proteins get partitioned more or less randomly between the two daughters. For the new preprint, though, researchers engineered E. coli to behave more like M. smegmatis. Here is how they did it: First, they deleted a gene called cyaA, which encodes an enzyme (adenylate cyclase) that makes a molecule called cAMP. cAMP is SUPER IMPORTANT! It is a nutrient sensor that instructs E. coli to switch on genes that help it digest non-glucose carbon sources when glucose is scarce. Without cAMP, E. coli cells growing on alternative carbon sources will starve; they won’t know how to eat the food. Next, they added back a “split” version of the cyaA gene into the cells. In other words, they split the gene in two so that each half of the enzyme is made separately. Cells can only make cAMP, and thus eat non-glucose carbon sources, if these two halves come together. To facilitate that “coming together,” the researchers also fused the split cyaA proteins to sticky proteins that clump together, and to a fluorescent protein (to make it easy to track these molecules in the cell.) So now some interesting things start to happen if you grow E. coli on a growth medium lacking glucose. As the cell grows, its cyaA “halves” start clumping together into a giant ball. Inside the aggregate, the two enzyme halves come together and make cAMP. And when the cell gets big enough and divides, the clump of cyaA RANDOMLY goes to either daughter cell #1 or #2. The daughter that gets the aggregate (called PA+ in this paper) can keep dividing. The daughter that doesn’t (PA–) cannot. It still grows a few times — about four divisions — because it inherits some leftover cAMP from its mother. But after that, the metabolite is diluted away, and the cell stops growing. PA+ cells went through about 23 divisions on average before their aggregate decayed. And the population of cells, as a whole, grew linearly. This paper is cool because there are many applications where exponential growth is too unpredictable and, perhaps, unsafe. If you want to engineer bacteria to deliver drugs, clean up waste, or live in the gut, you don’t want them to double uncontrollably. This paper shows you can make them expand in a controlled, linear way. Alas, mutations could break this whole engineered system. A mutation that restores cyaA, for example, would give cells a new way to make cAMP. Mutations that make the aggregates split between daughters would break the asymmetry, too. But still, I really enjoy proof-of-concept engineering papers like this.

A single E. coli cell, placed on a dish, will become 70 billion cells in just 12 hours. That’s exponential growth. But a new preprint shows that it's possible to engineer E. coli to grow linearly instead, where only one daughter cell continues dividing and the other stops. First, some context. In nature, there is a bacterium called Mycobacterium smegmatis (initially discovered in 1884 in ulcers scraped from syphilis patients.) M. smegmatis is weird because it divides asymmetrically. These cells grow only from one end, and all their cell wall biosynthesis machinery is located on that one end. So when the cell divides, one daughter gets this machinery and the other gets nothing. The daughter that gets the machinery can keep dividing immediately, but the other daughter has to remake all that machinery from scratch, so its growth is delayed. E. coli doesn’t grow like this. When it divides, it pinches in the middle and splits everything evenly. Enzymes, metabolites, and proteins get partitioned more or less randomly between the two daughters. For the new preprint, though, researchers engineered E. coli to behave more like M. smegmatis. Here is how they did it: First, they deleted a gene called cyaA, which encodes an enzyme (adenylate cyclase) that makes a molecule called cAMP. cAMP is SUPER IMPORTANT! It is a nutrient sensor that instructs E. coli to switch on genes that help it digest non-glucose carbon sources when glucose is scarce. Without cAMP, E. coli cells growing on alternative carbon sources will starve; they won’t know how to eat the food. Next, they added back a “split” version of the cyaA gene into the cells. In other words, they split the gene in two so that each half of the enzyme is made separately. Cells can only make cAMP, and thus eat non-glucose carbon sources, if these two halves come together. To facilitate that “coming together,” the researchers also fused the split cyaA proteins to sticky proteins that clump together, and to a fluorescent protein (to make it easy to track these molecules in the cell.) So now some interesting things start to happen if you grow E. coli on a growth medium lacking glucose. As the cell grows, its cyaA “halves” start clumping together into a giant ball. Inside the aggregate, the two enzyme halves come together and make cAMP. And when the cell gets big enough and divides, the clump of cyaA RANDOMLY goes to either daughter cell #1 or #2. The daughter that gets the aggregate (called PA+ in this paper) can keep dividing. The daughter that doesn’t (PA–) cannot. It still grows a few times — about four divisions — because it inherits some leftover cAMP from its mother. But after that, the metabolite is diluted away, and the cell stops growing. PA+ cells went through about 23 divisions on average before their aggregate decayed. And the population of cells, as a whole, grew linearly. This paper is cool because there are many applications where exponential growth is too unpredictable and, perhaps, unsafe. If you want to engineer bacteria to deliver drugs, clean up waste, or live in the gut, you don’t want them to double uncontrollably. This paper shows you can make them expand in a controlled, linear way. Alas, mutations could break this whole engineered system. A mutation that restores cyaA, for example, would give cells a new way to make cAMP. Mutations that make the aggregates split between daughters would break the asymmetry, too. But still, I really enjoy proof-of-concept engineering papers like this.

Niko McCarty.

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⚡ MICROWAVE WEAPON vs ROGUE DRONES The next generation of counter drone technology is here. AI powered acoustic and radar sensors work together to detect, identify and track rogue drones. Once a threat is confirmed, high energy microwave pulses can disrupt and neutralize the drone. Designed to protect airports, refineries, factories and other critical infrastructure from growing drone threats. AI detection + multi sensor fusion + high energy microwave suppression = a powerful new layer of airspace security.

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Let's go London! 😘 All I've got is good memories of summer in the UK. From Party In The Park in the 90s to more recent adventures at Glastonbury and BST Hyde Park. For as long as I can remember I’ve had major UK specific items on my bucket list and I'm ticking them off one by one... can't believe at 60, I tick a REALLY big one off - Playing Wembley Stadium! And with someone I love and admire so much, Harry Styles. Londoners sign me up for Wimbledon viewing, Pimms o clock, horse riding, sunny days and long summer evenings with friends. Cheers 🍻 Together, Together in London from June 12 - June 23. Visit to learn more.

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MAX ROSA: 'CUSE CAPTAIN 🍊 Coming out of high school, Max Rosa was an All-American midfielder who scored 42 goals as a senior at West Genesee. After a COVID-shortened 2020 season, Rosa switched to SSDM as a freshman at Syracuse in 2021. He made an immediate impact on the face-off wing and has been a reliable defensive midfielder ever since. On Tuesday, Rosa was named a team captain for the 2nd consecutive season... Watch this hustle play, and you'll immediately understand why 😤 (via Syracuse Men’s Lacrosse)

MAX ROSA: 'CUSE CAPTAIN 🍊 Coming out of high school, Max Rosa was an All-American midfielder who scored 42 goals as a senior at West Genesee. After a COVID-shortened 2020 season, Rosa switched to SSDM as a freshman at Syracuse in 2021. He made an immediate impact on the face-off wing and has been a reliable defensive midfielder ever since. On Tuesday, Rosa was named a team captain for the 2nd consecutive season... Watch this hustle play, and you'll immediately understand why 😤 (via Syracuse Men’s Lacrosse)

TLN 🥍

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Celebrating 5️⃣0️⃣0️⃣ members of the ECA Family. In a landmark moment for European club football, we are proud to announce that 500 clubs across Europe are now members of ECA. As of today, ECA Membership has achieved a remarkable 88% growth since the start of the 2023/24 season. With each new addition, ECA's diverse family of member clubs continues to act in the collective interest of all - working together to face the challenges and find innovative solutions and opportunities in the ever-changing landscape of European football. We look forward to further strong growth and welcoming more members to the ECA Family, to continue shaping the future of European football. 🌍 Take a look at the full story here: #WeAreECA #HeartofFootball

Celebrating 5️⃣0️⃣0️⃣ members of the ECA Family. In a landmark moment for European club football, we are proud to announce that 500 clubs across Europe are now members of ECA. As of today, ECA Membership has achieved a remarkable 88% growth since the start of the 2023/24 season. With each new addition, ECA's diverse family of member clubs continues to act in the collective interest of all - working together to face the challenges and find innovative solutions and opportunities in the ever-changing landscape of European football. We look forward to further strong growth and welcoming more members to the ECA Family, to continue shaping the future of European football. 🌍 Take a look at the full story here: #WeAreECA #HeartofFootball

European Football Clubs

12,252 просмотров • 2 лет назад

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Look at Shujaa making herself quite at home in Ithumba! Fresh graduates Weka and Shujaa have recently made the big move from Nairobi Nursery to our Ithumba Reintegration Unit, and it's wonderful to see how quickly they're adapting to their new surroundings. Shujaa has discovered the joy of rock climbing - a perfect way to build strength and confidence in her new environment! The transition from our Nursery to a Reintegration Unit is a major milestone for any orphan. Here at Ithumba, the duo will learn essential skills from older orphans, ex-orphans, and wild elephants, gradually becoming more independent, all under the watchful care of their dedicated Keepers. From Nursery babies to Ithumba adventurers - every step takes them closer to their wild future! Support Shujaa’s journey:

Sheldrick Wildlife Trust

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Crypto Banter

24,311 просмотров • 1 месяц назад