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🚨 Graphene just did something wild. It killed harmful bacteria including drug-resistant superbugs while sparing human cells. Read that again. Researchers found graphene oxide can target a molecule found in bacterial membranes, but not human cells. That means it can attack bacteria with precision instead of acting like a... chemical blunt weapon. Potential uses: wound dressings masks antibacterial clothing hygiene products medical textiles And the key point: This isn’t just “stronger antibiotics.” It’s a material that uses structure to separate dangerous cells from human ones. If this scales safely, the future of fighting infection may not only be drugs… It may be engineered surfaces. Could materials become the next antibiotics? Follow me I track where physics becomes technology.show more

TheNewPhysics

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19,614 views • 2 months ago •via X (Twitter)

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Ok this is actually wild. Scientists made electronics out of a soft material that mimics human tissue… so it can exist inside your body while your cells grow around it. This isn’t wearable tech. This is inside you tech. 😳 Tell me what you think. 👇

Ok this is actually wild. Scientists made electronics out of a soft material that mimics human tissue… so it can exist inside your body while your cells grow around it. This isn’t wearable tech. This is inside you tech. 😳 Tell me what you think. 👇

TheLizVariant

37,922 views • 2 months ago

The mTOR inhibitor Sapanisertib is a putative longevity molecule that makes human skin cells & prematurely old fish healthier Combined with metformin (a putative longevity drug), 79% of human patients with advanced or metastatic solid tumors achieved disease control. Promising results. Could S+M be a longevity combo and who owns it? 🧵

The mTOR inhibitor Sapanisertib is a putative longevity molecule that makes human skin cells & prematurely old fish healthier Combined with metformin (a putative longevity drug), 79% of human patients with advanced or metastatic solid tumors achieved disease control. Promising results. Could S+M be a longevity combo and who owns it? 🧵

David Sinclair

94,184 views • 1 year ago

A stroke can destroy brain cells within minutes, often leaving people with long‑term problems in movement, memory, or speech. But scientists may have found a way to help the brain heal itself. In studies of mice, researchers injected stem cells into areas of the brain damaged by stroke. These stem cells began turning into early‑stage brain cells, and many later developed into active neurons that connected with surrounding tissue. The treatment did more than just grow cells.

A stroke can destroy brain cells within minutes, often leaving people with long‑term problems in movement, memory, or speech. But scientists may have found a way to help the brain heal itself. In studies of mice, researchers injected stem cells into areas of the brain damaged by stroke. These stem cells began turning into early‑stage brain cells, and many later developed into active neurons that connected with surrounding tissue. The treatment did more than just grow cells.

Science Postcard

56,354 views • 7 months ago

There's a bacteriophage that turns bacteria into “liquid crystals.” Specifically, Pseudomonas aeruginosa bacteria make Pf phages, which are rod-shaped, negatively-charged, and measure about 2 micrometers in length (roughly the length of an E. coli cell). These phages leave the cells and enter their surroundings. There, they mix with polymers, also secreted by the cells, to form a crystalline matrix. Surprisingly, this is good for the cells. Although the phages kill some of them, it also makes their biofilms stickier and able to withstand certain antibiotics. These bacteria + phages are prevalent in cystic fibrosis patients; they've formed a sort of symbiotic relationship. The Pf phages are made from thousands of repeating copies of a coat protein, called CoaB, which wraps around a single-stranded, circular DNA genome. These genes are integrated directly on the bacterial chromosome. The bacteria “turn on” these phage genes when placed in a viscous environment with low oxygen levels. This is like a trigger to start forming a biofilm. And the cells make a lot of phages; about 100 billion per milliliter. These liquid crystals form because of a physics principle called “depletion attraction.” If you just mix a bunch of loose or flexible polymers together (such as long carbon chains) they will not form a liquid crystal. But if you mix stiff rods (the phages) with loose polymers at a high enough concentration, the polymers will force the phages close together to create a material that flows like a liquid despite being ordered like a crystal. See the video below. These liquid crystal biofilms are hard to get rid of. The negatively-charged phages block many antibiotics (like aminoglycosides, which are positively-charged) from entering cells. Liquid crystals also retain water, so these biofilms can survive on drier surfaces. I first heard about this from Malmesbury’s excellent newsletter, called “Telescopic Turnip.”

There's a bacteriophage that turns bacteria into “liquid crystals.” Specifically, Pseudomonas aeruginosa bacteria make Pf phages, which are rod-shaped, negatively-charged, and measure about 2 micrometers in length (roughly the length of an E. coli cell). These phages leave the cells and enter their surroundings. There, they mix with polymers, also secreted by the cells, to form a crystalline matrix. Surprisingly, this is good for the cells. Although the phages kill some of them, it also makes their biofilms stickier and able to withstand certain antibiotics. These bacteria + phages are prevalent in cystic fibrosis patients; they've formed a sort of symbiotic relationship. The Pf phages are made from thousands of repeating copies of a coat protein, called CoaB, which wraps around a single-stranded, circular DNA genome. These genes are integrated directly on the bacterial chromosome. The bacteria “turn on” these phage genes when placed in a viscous environment with low oxygen levels. This is like a trigger to start forming a biofilm. And the cells make a lot of phages; about 100 billion per milliliter. These liquid crystals form because of a physics principle called “depletion attraction.” If you just mix a bunch of loose or flexible polymers together (such as long carbon chains) they will not form a liquid crystal. But if you mix stiff rods (the phages) with loose polymers at a high enough concentration, the polymers will force the phages close together to create a material that flows like a liquid despite being ordered like a crystal. See the video below. These liquid crystal biofilms are hard to get rid of. The negatively-charged phages block many antibiotics (like aminoglycosides, which are positively-charged) from entering cells. Liquid crystals also retain water, so these biofilms can survive on drier surfaces. I first heard about this from Malmesbury’s excellent newsletter, called “Telescopic Turnip.”

Niko McCarty.

50,029 views • 6 months ago

Scientists just discovered the hidden culprit of heart disease: A gut bacteria molecule that silently scars arteries in 1 out of 5 “healthy” adults. This may be the missing link doctors couldn’t explain for decades. What they found & how to prevent it:

Scientists just discovered the hidden culprit of heart disease: A gut bacteria molecule that silently scars arteries in 1 out of 5 “healthy” adults. This may be the missing link doctors couldn’t explain for decades. What they found & how to prevent it:

Simran Nahata

143,929 views • 10 months ago

🚨 BREAKING: Scientists just found a SPECIFIC sugar molecule made by gut bacteria that appears to TRIGGER ALS and dementia. 70% of ALS/FTD patients had it. Only 33% of healthy people did. And they found a way to STOP it. 🧵

🚨 BREAKING: Scientists just found a SPECIFIC sugar molecule made by gut bacteria that appears to TRIGGER ALS and dementia. 70% of ALS/FTD patients had it. Only 33% of healthy people did. And they found a way to STOP it. 🧵

Harshi Peiris, Ph.D.

380,791 views • 2 months ago

🚨 BREAKING: Scientists just created a non-toxic, low-cost conductive polymer This could unlock a new era of biocompatible electronics Think about it • Circuits that move like skin • Implants the body doesn’t reject • Medical tech that feels alive We’re not just building better devices… We’re starting to merge technology with biology The line between human and machine is about to disappear Follow me I break down the future of physics, tech, and reality itself

🚨 BREAKING: Scientists just created a non-toxic, low-cost conductive polymer This could unlock a new era of biocompatible electronics Think about it • Circuits that move like skin • Implants the body doesn’t reject • Medical tech that feels alive We’re not just building better devices… We’re starting to merge technology with biology The line between human and machine is about to disappear Follow me I break down the future of physics, tech, and reality itself

TheNewPhysics

283,815 views • 2 months ago

Notre Dame researchers have found that the single-cell pH of cancer cells changes dynamically during the four phases of cell division This discovery could eventually lead to therapies that will limit cancer cells’ ability to grow and divide:

Notre Dame researchers have found that the single-cell pH of cancer cells changes dynamically during the four phases of cell division This discovery could eventually lead to therapies that will limit cancer cells’ ability to grow and divide:

University of Notre Dame

42,055 views • 3 years ago

🚨: A petri dish of human brain cells just learned to play DOOM

🚨: A petri dish of human brain cells just learned to play DOOM

Curiosity

32,356,884 views • 4 months ago

Scientists are baffled by this. The Bacterial Flagellum is composed of 20-30 unique parts. Combined, they total over 30,000 parts that make this little motor run. It can hit rotation speeds of up to 100,000 RPM, then stop & reverse direction in a quarter of a turn. It can propel a bacteria up to 20 lengths per second, which when scaled up is about equal to 225mph in a car. But this motor only works when all those parts are perfectly engineered and working together. Take away just one of those 30 pieces, and it stops functioning. That means it can't evolve gradually. None of the pieces do anything outside the system, and the system only works with all the pieces. Bacteria require this motor to move - without it, they would quickly die. How can anyone see something like and still deny that Life is Divinely Designed?

Scientists are baffled by this. The Bacterial Flagellum is composed of 20-30 unique parts. Combined, they total over 30,000 parts that make this little motor run. It can hit rotation speeds of up to 100,000 RPM, then stop & reverse direction in a quarter of a turn. It can propel a bacteria up to 20 lengths per second, which when scaled up is about equal to 225mph in a car. But this motor only works when all those parts are perfectly engineered and working together. Take away just one of those 30 pieces, and it stops functioning. That means it can't evolve gradually. None of the pieces do anything outside the system, and the system only works with all the pieces. Bacteria require this motor to move - without it, they would quickly die. How can anyone see something like and still deny that Life is Divinely Designed?

Divinely Designed

96,188 views • 1 month ago

Can AI rewrite our human genome? ⌨️🧬 Today, we announce the successful editing of DNA in human cells with gene editors fully designed with AI. Not only that, we've decided to freely release the molecules under the Profluent OpenCRISPR initiative. Lots to unpack👇

Can AI rewrite our human genome? ⌨️🧬 Today, we announce the successful editing of DNA in human cells with gene editors fully designed with AI. Not only that, we've decided to freely release the molecules under the Profluent OpenCRISPR initiative. Lots to unpack👇

Ali Madani

1,245,979 views • 2 years ago

Bacteria communicate like us, using electrical signalling similar to how neurones fire in the human brain ⚡️🦠⁠ Researchers at University of Warwick and Politecnico di Milano can control bacteria's electrical signalling with the molecule, Ziapin2 which may have implications for antibiotic resistance

Bacteria communicate like us, using electrical signalling similar to how neurones fire in the human brain ⚡️🦠⁠ Researchers at University of Warwick and Politecnico di Milano can control bacteria's electrical signalling with the molecule, Ziapin2 which may have implications for antibiotic resistance

New Scientist

65,278 views • 3 years ago

A single E. coli cell, placed on a dish, will become 70 billion cells in just 12 hours. That’s exponential growth. But a new preprint shows that it's possible to engineer E. coli to grow linearly instead, where only one daughter cell continues dividing and the other stops. First, some context. In nature, there is a bacterium called Mycobacterium smegmatis (initially discovered in 1884 in ulcers scraped from syphilis patients.) M. smegmatis is weird because it divides asymmetrically. These cells grow only from one end, and all their cell wall biosynthesis machinery is located on that one end. So when the cell divides, one daughter gets this machinery and the other gets nothing. The daughter that gets the machinery can keep dividing immediately, but the other daughter has to remake all that machinery from scratch, so its growth is delayed. E. coli doesn’t grow like this. When it divides, it pinches in the middle and splits everything evenly. Enzymes, metabolites, and proteins get partitioned more or less randomly between the two daughters. For the new preprint, though, researchers engineered E. coli to behave more like M. smegmatis. Here is how they did it: First, they deleted a gene called cyaA, which encodes an enzyme (adenylate cyclase) that makes a molecule called cAMP. cAMP is SUPER IMPORTANT! It is a nutrient sensor that instructs E. coli to switch on genes that help it digest non-glucose carbon sources when glucose is scarce. Without cAMP, E. coli cells growing on alternative carbon sources will starve; they won’t know how to eat the food. Next, they added back a “split” version of the cyaA gene into the cells. In other words, they split the gene in two so that each half of the enzyme is made separately. Cells can only make cAMP, and thus eat non-glucose carbon sources, if these two halves come together. To facilitate that “coming together,” the researchers also fused the split cyaA proteins to sticky proteins that clump together, and to a fluorescent protein (to make it easy to track these molecules in the cell.) So now some interesting things start to happen if you grow E. coli on a growth medium lacking glucose. As the cell grows, its cyaA “halves” start clumping together into a giant ball. Inside the aggregate, the two enzyme halves come together and make cAMP. And when the cell gets big enough and divides, the clump of cyaA RANDOMLY goes to either daughter cell #1 or #2. The daughter that gets the aggregate (called PA+ in this paper) can keep dividing. The daughter that doesn’t (PA–) cannot. It still grows a few times — about four divisions — because it inherits some leftover cAMP from its mother. But after that, the metabolite is diluted away, and the cell stops growing. PA+ cells went through about 23 divisions on average before their aggregate decayed. And the population of cells, as a whole, grew linearly. This paper is cool because there are many applications where exponential growth is too unpredictable and, perhaps, unsafe. If you want to engineer bacteria to deliver drugs, clean up waste, or live in the gut, you don’t want them to double uncontrollably. This paper shows you can make them expand in a controlled, linear way. Alas, mutations could break this whole engineered system. A mutation that restores cyaA, for example, would give cells a new way to make cAMP. Mutations that make the aggregates split between daughters would break the asymmetry, too. But still, I really enjoy proof-of-concept engineering papers like this.

A single E. coli cell, placed on a dish, will become 70 billion cells in just 12 hours. That’s exponential growth. But a new preprint shows that it's possible to engineer E. coli to grow linearly instead, where only one daughter cell continues dividing and the other stops. First, some context. In nature, there is a bacterium called Mycobacterium smegmatis (initially discovered in 1884 in ulcers scraped from syphilis patients.) M. smegmatis is weird because it divides asymmetrically. These cells grow only from one end, and all their cell wall biosynthesis machinery is located on that one end. So when the cell divides, one daughter gets this machinery and the other gets nothing. The daughter that gets the machinery can keep dividing immediately, but the other daughter has to remake all that machinery from scratch, so its growth is delayed. E. coli doesn’t grow like this. When it divides, it pinches in the middle and splits everything evenly. Enzymes, metabolites, and proteins get partitioned more or less randomly between the two daughters. For the new preprint, though, researchers engineered E. coli to behave more like M. smegmatis. Here is how they did it: First, they deleted a gene called cyaA, which encodes an enzyme (adenylate cyclase) that makes a molecule called cAMP. cAMP is SUPER IMPORTANT! It is a nutrient sensor that instructs E. coli to switch on genes that help it digest non-glucose carbon sources when glucose is scarce. Without cAMP, E. coli cells growing on alternative carbon sources will starve; they won’t know how to eat the food. Next, they added back a “split” version of the cyaA gene into the cells. In other words, they split the gene in two so that each half of the enzyme is made separately. Cells can only make cAMP, and thus eat non-glucose carbon sources, if these two halves come together. To facilitate that “coming together,” the researchers also fused the split cyaA proteins to sticky proteins that clump together, and to a fluorescent protein (to make it easy to track these molecules in the cell.) So now some interesting things start to happen if you grow E. coli on a growth medium lacking glucose. As the cell grows, its cyaA “halves” start clumping together into a giant ball. Inside the aggregate, the two enzyme halves come together and make cAMP. And when the cell gets big enough and divides, the clump of cyaA RANDOMLY goes to either daughter cell #1 or #2. The daughter that gets the aggregate (called PA+ in this paper) can keep dividing. The daughter that doesn’t (PA–) cannot. It still grows a few times — about four divisions — because it inherits some leftover cAMP from its mother. But after that, the metabolite is diluted away, and the cell stops growing. PA+ cells went through about 23 divisions on average before their aggregate decayed. And the population of cells, as a whole, grew linearly. This paper is cool because there are many applications where exponential growth is too unpredictable and, perhaps, unsafe. If you want to engineer bacteria to deliver drugs, clean up waste, or live in the gut, you don’t want them to double uncontrollably. This paper shows you can make them expand in a controlled, linear way. Alas, mutations could break this whole engineered system. A mutation that restores cyaA, for example, would give cells a new way to make cAMP. Mutations that make the aggregates split between daughters would break the asymmetry, too. But still, I really enjoy proof-of-concept engineering papers like this.

Niko McCarty.

57,988 views • 10 months ago

'Gorilla is thinking about something' Researchers say gorillas use human-like facial expressions and they can even smirk like this but it usualy means something different from human meanings

'Gorilla is thinking about something' Researchers say gorillas use human-like facial expressions and they can even smirk like this but it usualy means something different from human meanings

Massimo

4,868,983 views • 2 years ago

If you have Cancer, this is your visualization. That is a cancerous cell reaching Apoptosis and Necrosis—they are gone. — Natural Killer (NK) cells play a the primary role in the immune system's defense against cancer. They induce cancer cell death primarily through two mechanisms: Apoptosis Necrosis Apoptosis is a programmed, non-inflammatory form of cell death, while necrosis is a more abrupt and inflammatory process. In many cases, NK cells can trigger a combination of both, leading to mixed forms of cell death. This dual capability allows NK cells to effectively target and destroy all tumor cells. Their ability to act without prior sensitization makes them vital for early cancer surveillance and curing existing cancer. One of the only ways cancer never starts or stops entirely is NK cells. They are the guerrilla warfare troops that never sleep. This is why it is vital for BioShield by to be approved, NOW for every cancer, not just a single cancer type. With the rest needing years and years of tests. Cancer is cancer, unregulated cell division and NK cells are NK cells. The ONLY thing that stops BioShield is government betrayal, and organized corruption disguised as following the “regulations” designed by the entrenched gatekeepers of corporate protection. Everyone knows it works that has even a molecule of biological understanding. Now you know.

If you have Cancer, this is your visualization. That is a cancerous cell reaching Apoptosis and Necrosis—they are gone. — Natural Killer (NK) cells play a the primary role in the immune system's defense against cancer. They induce cancer cell death primarily through two mechanisms: Apoptosis Necrosis Apoptosis is a programmed, non-inflammatory form of cell death, while necrosis is a more abrupt and inflammatory process. In many cases, NK cells can trigger a combination of both, leading to mixed forms of cell death. This dual capability allows NK cells to effectively target and destroy all tumor cells. Their ability to act without prior sensitization makes them vital for early cancer surveillance and curing existing cancer. One of the only ways cancer never starts or stops entirely is NK cells. They are the guerrilla warfare troops that never sleep. This is why it is vital for BioShield by to be approved, NOW for every cancer, not just a single cancer type. With the rest needing years and years of tests. Cancer is cancer, unregulated cell division and NK cells are NK cells. The ONLY thing that stops BioShield is government betrayal, and organized corruption disguised as following the “regulations” designed by the entrenched gatekeepers of corporate protection. Everyone knows it works that has even a molecule of biological understanding. Now you know.

Brian Roemmele

191,436 views • 7 months ago

PEF Ablation = Pulsed Electric Field Nano-pulses of high voltage energy directed WITHIN tumors. Not radiation that damages everything around it. Not surgery that triggers inflammatory cascades. Precision destruction of tumor cells while preserving surrounding tissue and vascular structure. This is the opposite of "The Rush to Radiation". Precision over scorched earth.

PEF Ablation = Pulsed Electric Field Nano-pulses of high voltage energy directed WITHIN tumors. Not radiation that damages everything around it. Not surgery that triggers inflammatory cascades. Precision destruction of tumor cells while preserving surrounding tissue and vascular structure. This is the opposite of "The Rush to Radiation". Precision over scorched earth.

Jason R. Williams, MD, DABR

16,450 views • 7 months ago

that isn’t really him. They are just faking it, and the Bible said that if they ever found me in a secret cavern, it’s not real interesting.

that isn’t really him. They are just faking it, and the Bible said that if they ever found me in a secret cavern, it’s not real interesting.

Kyle L

58,713 views • 1 month ago

for #SUNOO 🦊 💌 🎂 an angel once taught me that happiness isn’t something you keep to yourself, it’s something meant to be shared; i was deeply moved by the story of a human angel who started the happiness fund. so i decided to follow in his footsteps by making a contribution to doctors without borders. It’s a small gesture, but I hope it can help bring comfort and hope to places that need it the most i also adopted a giant panda through wwf uk, not only it’s a step toward protecting the future of these beautiful creatures, but it’s also a quiet wish: that the human angel who inspired it all can continue to love panda for a long, long time and lastly, there’s a little surprise shining on the street of jakarta, a led ad just for him. a soft light in the city, reminding everyone who passes by of the warmth he brings into the world sharing this because he’s been such a light.. not only to me, but to so many others. this is my way of giving a bit of that back. happy birthday kim sunoo 💛 CELESTIAL SUNOO DAY #SunooHeartOfJune #하늘에서_떤사님_내려오신날 #엔하이픈_선우 #SUNOO ENHYPEN ENHYPEN OFFICIAL

for #SUNOO 🦊 💌 🎂 an angel once taught me that happiness isn’t something you keep to yourself, it’s something meant to be shared; i was deeply moved by the story of a human angel who started the happiness fund. so i decided to follow in his footsteps by making a contribution to doctors without borders. It’s a small gesture, but I hope it can help bring comfort and hope to places that need it the most i also adopted a giant panda through wwf uk, not only it’s a step toward protecting the future of these beautiful creatures, but it’s also a quiet wish: that the human angel who inspired it all can continue to love panda for a long, long time and lastly, there’s a little surprise shining on the street of jakarta, a led ad just for him. a soft light in the city, reminding everyone who passes by of the warmth he brings into the world sharing this because he’s been such a light.. not only to me, but to so many others. this is my way of giving a bit of that back. happy birthday kim sunoo 💛 CELESTIAL SUNOO DAY #SunooHeartOfJune #하늘에서_떤사님_내려오신날 #엔하이픈_선우 #SUNOO ENHYPEN ENHYPEN OFFICIAL

pyu ♡ 선우

18,300 views • 1 year ago

Injustice is the misalignment of human behavior and social order with norms that ought to be truly human — humane, reasonable, and faithful to their essence. : The author defines injustice not as a random misfortune or isolated act of cruelty, but as a fundamental misalignment between how humans actually behave and organize their societies and the norms that ought to govern them. Those norms must be truly human: humane in spirit, reasonable in application, and faithful to the deepest essence of what it means to be human. When our laws, institutions, customs, or daily conduct drift away from this standard, injustice takes root. It appears in systems that treat people as means rather than ends, in hierarchies that ignore dignity, and in habits that favor convenience over compassion. The gap between what is and what should be becomes the very measure of injustice. True justice, therefore, is the ongoing effort to close that gap, to shape both personal conduct and social structures so they reflect our highest understanding of humanity. It is not a fixed state but a continuous alignment with the humane, the rational, and the authentically human.

Injustice is the misalignment of human behavior and social order with norms that ought to be truly human — humane, reasonable, and faithful to their essence. : The author defines injustice not as a random misfortune or isolated act of cruelty, but as a fundamental misalignment between how humans actually behave and organize their societies and the norms that ought to govern them. Those norms must be truly human: humane in spirit, reasonable in application, and faithful to the deepest essence of what it means to be human. When our laws, institutions, customs, or daily conduct drift away from this standard, injustice takes root. It appears in systems that treat people as means rather than ends, in hierarchies that ignore dignity, and in habits that favor convenience over compassion. The gap between what is and what should be becomes the very measure of injustice. True justice, therefore, is the ongoing effort to close that gap, to shape both personal conduct and social structures so they reflect our highest understanding of humanity. It is not a fixed state but a continuous alignment with the humane, the rational, and the authentically human.

Zafar Mirzo | Quotes

1,624,313 views • 4 months ago