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3/ Big idea #1: Mitochondria, not the genes, determines if a cell becomes cancerous When cells can’t make energy with oxygen, they switch to the ancient fermentation pathway. Cancer thrives by burning glucose & glutamine without oxygen. Uncontrolled cell growth follows.

no.mind

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1,148,762 views • 1 year ago •via X (Twitter)

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no.mind1 year ago

This is Thomas Seyfried. He’s a professor of biology, who’s studied cancer for 30+ years. His message? Cancer isn’t bad genes or bad luck—it’s damaged mitochondria. This flips everything you’ve been told about how to treat & prevent cancer: 🧵

no.mind1 year ago

1/ Cancer rates are on the rise, why? - 1,676 people die daily from cancer in the U.S. - Young seemingly healthy people are being diagnosed more often than ever. - Billions spent, yet the "war on cancer" failed. Seyfried: We're hitting the wrong target.

no.mind1 year ago

2/ Mainstream dogma: "Cancer is a genetic disease caused by DNA mutations.” Seyfried: Wrong. DNA damage is a symptom, not cause. Real cause: Damaged mitochondria Healthy mitochondria = NO cancer.

no.mind1 year ago

4/ Seyfried builds on Otto Warburg’s forgotten theory. Warburg won the 1931 Nobel Prize in Medicine. He observed cancer cells ferment sugar even with oxygen (Warburg Effect) and believed cancer was metabolic, not genetic. Modern science ignored him. Seyfried didn’t.

no.mind1 year ago

5/ Proof from research? Damaged DNA + healthy mitochondria = NO cancer Healthy DNA + damaged mitochondria = CANCER DNA isn't the trigger, it's downstream damage.

no.mind1 year ago

6/ Research from the visionary biologist Michael Levin supports this: Cancer is a disease of failed bioelectric signaling, not genes.

no.mind1 year ago

7/ Big idea #2: All cancers are one disease: damaged mitochondria. Yet medicine divides tumors by organs (breast, lung, brain). We're treating symptoms, not the root cause.

no.mind1 year ago

8/ Dr. Jason Fung, MD, author of "The Cancer Code", agrees: Cancer isn’t just a genetic disease, it’s a disease of bioenergetics. Standard treatments often fail because they ignore the root cause: dysfunctional metabolism.

no.mind1 year ago

9/ “But what about BRCA1, or other inherited cancer genes?” Even that links back to mitochondrial dysfunction. These genes affect mitochondrial proteins in the electron transport chain—key to energy production. They raise risk. They don’t cause cancer alone.

no.mind1 year ago

10/ What damages mitochondria? - Ultra-proccessed foods, high carb at wrong seasons - Insulin resistance - Chronic inflammation - Lack of exercise & movement - Poor sleep - Chemical toxins, heavy metals, mold - EMFs (Wi-Fi, 5G) - Chronic Stress - Viruses (HPV, Hepatitis) - Artificial Blue Light To name a few.

no.mind1 year ago

11/ Seyfried’s approach to cancer: - Get body into nutritional ketosis (Glucose/Ketone Index < 2.0) via ketogenic diet or fasting - Press glucose & pulse the glutamine with targeted drugs - Bonus: Hyperbaric oxygen therapy

no.mind1 year ago

12/ Nutritional Ketosis Cancer cells depend on glucose & glutamine. They can’t efficiently use ketones or fat for fuel. High ketones + low glucose = cancer-starving state. Seyfried: Measure, don’t guess. You can use a Keto-Mojo meter to track ketones & glucose.

no.mind1 year ago

13/ Important Caveat #1: Glutamine Glutamine is essential for our immune function—you don’t want to eliminate it long-term. It's the most abundant amino acid in the body & diet alone won’t block it. That’s why Seyfried uses pulsed drug strategies to temporarily target it.

no.mind1 year ago

14/ Hyperbaric Oxygen Therapy (HBOT) Seyfried sees it as a metabolic add-on. When used with keto + glucose restriction, HBOT floods tumors with oxygen. Normal cells benefit from the oxygen boost. Cancer cells can’t handle the oxidative stress, they get stressed & die.

no.mind1 year ago

15/ "But chemo/radiation save lives..." True, but with serious long-term damage. Metabolic therapy is safe & effective, yet ignored. Why? No profit in fasting, ketosis.

no.mind1 year ago

16/ Important Caveat #2: Seyfried isn’t entirely against chemo, radiation, immunotherapy, or surgery. His view: if you first weaken the tumor with metabolic therapy, then low-dose chemo or immunotherapy can work better. What he opposes: blasting the brain with radiation.

no.mind1 year ago

17/ Seyfried: Never Radiate the Brain Seyfried’s research shows that brain radiation frees up massive amounts of glucose & glutamine… …the exact fuels brain tumors thrive on. His advice: avoid brain radiation.

no.mind1 year ago

18/ “If this is true, why isn’t it mainstream?” Because metabolic therapy isn’t part of standard care. Doctors aren’t trained in it, no billing code for it— And doctors risk losing their license if they go off-guideline. The system rewards treatment, not prevention.

no.mind1 year ago

19/ Here’s the disconnect in 2025: Larry Ellison spoke at the White House pushing gene sequencing + mRNA cancer vaccines. But if cancer starts with mitochondrial dysfunction, targeting genes is missing the point. Still, billions flow to genetics, while metabolism is ignored.

no.mind1 year ago

20/ Seyfried’s bottom line: You don’t get cancer if your mitochondria is healthy. Period. Cancer doesn’t happen overnight, start early, protect your mitochondria to prevent cancer.

no.mind1 year ago

21/ Protect your mitochondria to prevent cancer: - Intermittent fasting / longer water fasting - High-quality sleep (deep + REM) - Seasonal whole foods - Eliminate seed oils & ultra-processed food - Limit alcohol, drugs, smoking - Daily sunlight exposure - Grounding (barefoot outdoors) - Blue light protection after sunset - Zone 2 cardio + HIIT - Strength training - Cold exposure (showers/plunges) - Sauna - Meditation, journaling, visualization - Find purpose & live in alignment - Dental hygiene - Limit EMFs (Wi-Fi, Bluetooth, 5G) - Seafood 3-5x a week - Walking 7.500-10.000 steps - Nasal breathing - Test, don’t guess

no.mind1 year ago

22/ DISCLAIMER: This is NOT medical advice. Always consult a qualified professional. But science advances through open debate, not censorship or dogma. Let ideas compete & compare the outcomes, not titles.

no.mind1 year ago

Thanks for reading! If you found this valuable, repost the first tweet so others can gain access to this information. Follow @the_no_mind for health insights you won’t find anywhere else.

no.mind1 year ago

P.S. I help entrepreneurs & high performers fix their energy at the root, by restoring their mitochondrial energy. Subscribe to my newsletter & get my “10 Habits for Limitless Energy Guide" as a thank you. ↓

Naveen Sankar S1 year ago

Belly fat may predict colorectal cancer risk better than BMI! 🩺 📊 Discover how the waist-to-hip ratio offers deeper insights into cancer prevention. Learn more: #ColorectalCancer #CancerPrevention #ObesityResearch #HealthTips

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14/ Hyperbaric Oxygen Therapy (HBOT) Seyfried sees it as a metabolic add-on. When used with keto + glucose restriction, HBOT floods tumors with oxygen. Normal cells benefit from the oxygen boost. Cancer cells can’t handle the oxidative stress, they get stressed & die.

14/ Hyperbaric Oxygen Therapy (HBOT) Seyfried sees it as a metabolic add-on. When used with keto + glucose restriction, HBOT floods tumors with oxygen. Normal cells benefit from the oxygen boost. Cancer cells can’t handle the oxidative stress, they get stressed & die.

no.mind

524,670 views • 1 year ago

New research shows it's not just starving Glucose that's important in starving cancer cells. Groundbreaking new research from The Seyfried Lab at Boston College, confirms cancer cells ferment Glutamine, WITHOUT oxygen. Over the last seven years, they designed and carried out detailed experiments to determine which fuels cancer cells rely on for energy production.

New research shows it's not just starving Glucose that's important in starving cancer cells. Groundbreaking new research from The Seyfried Lab at Boston College, confirms cancer cells ferment Glutamine, WITHOUT oxygen. Over the last seven years, they designed and carried out detailed experiments to determine which fuels cancer cells rely on for energy production.

Humanspective

52,142 views • 1 year ago

⚔️ BLOCKING GLUTAMINE NATURALLY When cancer cells can’t get glucose, they often switch to glutamine, a key amino acid involved in cell growth and repair. 🍄 Mistletoe—immune-regulating and disrupts glutamine-dependent cancer growth 🌿 Curcumin—shown to interfere with glutamine metabolism in aggressive tumors 🍵 Green tea (EGCG)—again, powerful here as well 🌿 Berberine—reduces expression of glutamine transporters 🧠 Honokiol (from magnolia bark)—neuroprotective and suppresses glutamine usage 🍄 Turkey tail mushroom—modulates immune response and may slow glutamine-driven cell replication 🥬 Fermented vegetables—rich in butyrate, which opposes glutamine-fueled inflammation 🥥 Coconut oil—encourages ketone metabolism over glutamine ✅ Suggested Stack: • AM: Honokiol + green tea • PM: Curcumin + mistletoe extract (consult with a practitioner) • Daily: Coconut oil for brain fuel + fermented foods for terrain healing 🧬 Avoid excess animal protein during active treatment—glutamine is abundant in meat. ✅ STUDIES: • Cell Metabolism (2013): Glutamine metabolism is essential for tumor growth in low-glucose environments. • Molecules (2020): Curcumin inhibits glutaminase, the enzyme that converts glutamine to fuel in cancer cells. • Frontiers in Oncology (2022): Mistletoe extract shown to downregulate glutamine pathways in colorectal cancer cells. • Oncotarget (2015): Honokiol targets mitochondrial glutamine metabolism in glioma models. 🔥Cancer adapts fast. But so can you. Block the second fuel… And give your body the upper hand it was designed to have. -Dr Pete Sulack

⚔️ BLOCKING GLUTAMINE NATURALLY When cancer cells can’t get glucose, they often switch to glutamine, a key amino acid involved in cell growth and repair. 🍄 Mistletoe—immune-regulating and disrupts glutamine-dependent cancer growth 🌿 Curcumin—shown to interfere with glutamine metabolism in aggressive tumors 🍵 Green tea (EGCG)—again, powerful here as well 🌿 Berberine—reduces expression of glutamine transporters 🧠 Honokiol (from magnolia bark)—neuroprotective and suppresses glutamine usage 🍄 Turkey tail mushroom—modulates immune response and may slow glutamine-driven cell replication 🥬 Fermented vegetables—rich in butyrate, which opposes glutamine-fueled inflammation 🥥 Coconut oil—encourages ketone metabolism over glutamine ✅ Suggested Stack: • AM: Honokiol + green tea • PM: Curcumin + mistletoe extract (consult with a practitioner) • Daily: Coconut oil for brain fuel + fermented foods for terrain healing 🧬 Avoid excess animal protein during active treatment—glutamine is abundant in meat. ✅ STUDIES: • Cell Metabolism (2013): Glutamine metabolism is essential for tumor growth in low-glucose environments. • Molecules (2020): Curcumin inhibits glutaminase, the enzyme that converts glutamine to fuel in cancer cells. • Frontiers in Oncology (2022): Mistletoe extract shown to downregulate glutamine pathways in colorectal cancer cells. • Oncotarget (2015): Honokiol targets mitochondrial glutamine metabolism in glioma models. 🔥Cancer adapts fast. But so can you. Block the second fuel… And give your body the upper hand it was designed to have. -Dr Pete Sulack

Cleanse Parasites .com 🧹🪱 Herbal Cleanse Co.

137,938 views • 1 year ago

If he or a loved one had cancer, Dr. Thomas Seyfried's top priorities would be: 1. Reduce systemic inflammation 2. Deprive tumor cells of their fuels: glucose & glutamine 3. Transition fully into nutritional ketosis (normal cells thrive, tumors starve) 4. Pulse glutamine-targeting drugs (e.g., repurposed parasite meds) 5. Add non-toxic therapies like hyperbaric oxygen & IV vitamin C Metabolic approach to make the body hostile to cancer cells. 1:00 clip inside—clear, science-driven strategy.

If he or a loved one had cancer, Dr. Thomas Seyfried's top priorities would be: 1. Reduce systemic inflammation 2. Deprive tumor cells of their fuels: glucose & glutamine 3. Transition fully into nutritional ketosis (normal cells thrive, tumors starve) 4. Pulse glutamine-targeting drugs (e.g., repurposed parasite meds) 5. Add non-toxic therapies like hyperbaric oxygen & IV vitamin C Metabolic approach to make the body hostile to cancer cells. 1:00 clip inside—clear, science-driven strategy.

Camus

220,307 views • 7 months ago

WATCH: This anti-cancer mushroom supercharges your body’s oxygen levels so much so that Chinese marathon runners shattered world records using it. Cancer thrives in low-oxygen environments, so imagine the impact when you flood your body with oxygen like this. The mushroom is Cordyceps. Investigative journalist Jefferey Jaxen explains that studies have shown that Cordyceps: • ups your body’s internal oxygen by 40% • increases ATP by 28% • increases cancer patients’ natural killer cell activity by 400% If you haven't heard of Cordyceps, it's probably because it's too cheap for the cancer industry to profit from.

WATCH: This anti-cancer mushroom supercharges your body’s oxygen levels so much so that Chinese marathon runners shattered world records using it. Cancer thrives in low-oxygen environments, so imagine the impact when you flood your body with oxygen like this. The mushroom is Cordyceps. Investigative journalist Jefferey Jaxen explains that studies have shown that Cordyceps: • ups your body’s internal oxygen by 40% • increases ATP by 28% • increases cancer patients’ natural killer cell activity by 400% If you haven't heard of Cordyceps, it's probably because it's too cheap for the cancer industry to profit from.

The Vigilant Fox 🦊

1,507,505 views • 1 year ago

Cancer has a problem. It needs Glucose and Glutamine to survive. That's where anti parasitic's like Fenbendazole come in. "Cancer can not survive without the sugar Glucose or the amino acid Glutamine, which are the fermentation fuels" “Why would a parasite medication be effective in k*lling cancer? [because] the parasite and the cancer cells, use a common metabolic pathway, the glutaminolysis pathway”. Professor Thomas Seyfried from Boston College thinks that "Cancer cannot be a genetic disease" and mentions that the whole industry has been captured by more toxic treatments. He says what Doctors are learning about cancer is completely wrong. "The medical schools have not yet come to understand this unfortunately [and] many oncologists and physicians have never been trained to know that cancer is a metabolic disease"

Cancer has a problem. It needs Glucose and Glutamine to survive. That's where anti parasitic's like Fenbendazole come in. "Cancer can not survive without the sugar Glucose or the amino acid Glutamine, which are the fermentation fuels" “Why would a parasite medication be effective in k*lling cancer? [because] the parasite and the cancer cells, use a common metabolic pathway, the glutaminolysis pathway”. Professor Thomas Seyfried from Boston College thinks that "Cancer cannot be a genetic disease" and mentions that the whole industry has been captured by more toxic treatments. He says what Doctors are learning about cancer is completely wrong. "The medical schools have not yet come to understand this unfortunately [and] many oncologists and physicians have never been trained to know that cancer is a metabolic disease"

Humanspective

466,438 views • 9 months ago

Dr. Thomas Seyfried reveals why parasite drugs like fenbendazole & mebendazole crush cancer cells: They target the exact same energy pathway that cancer cells hijack — fermentation of glucose and glutamine — the same pathway many parasites use once settled in tissue. Short science nugget: Fenbendazole and mebendazole disrupt microtubule formation and block glucose/glutamine uptake, starving cancer cells of their primary fuels while sparing healthy cells that rely more on oxidative phosphorylation (peer-reviewed in vitro & animal studies, e.g., Dogra et al. 2018, Zhang et al. 2021, and Seyfried lab publications) Key points from Seyfried: - Structurally almost identical → likely equally effective - Cancer is not a parasite, but it metabolizes like one - These drugs don’t care — they hit the shared vulnerability Cocktails of them may work even better. Is this the overlooked cancer weapon big pharma ignores? Have you or someone you know explored benzimidazoles like fenbendazole/mebendazole?

Dr. Thomas Seyfried reveals why parasite drugs like fenbendazole & mebendazole crush cancer cells: They target the exact same energy pathway that cancer cells hijack — fermentation of glucose and glutamine — the same pathway many parasites use once settled in tissue. Short science nugget: Fenbendazole and mebendazole disrupt microtubule formation and block glucose/glutamine uptake, starving cancer cells of their primary fuels while sparing healthy cells that rely more on oxidative phosphorylation (peer-reviewed in vitro & animal studies, e.g., Dogra et al. 2018, Zhang et al. 2021, and Seyfried lab publications) Key points from Seyfried: - Structurally almost identical → likely equally effective - Cancer is not a parasite, but it metabolizes like one - These drugs don’t care — they hit the shared vulnerability Cocktails of them may work even better. Is this the overlooked cancer weapon big pharma ignores? Have you or someone you know explored benzimidazoles like fenbendazole/mebendazole?

Camus

88,248 views • 5 months ago

"I think deuterium is the reason why you have cancer." — Stephanie Seneff, MIT researcher. Deuterium is a heavy form of hydrogen naturally present in water and food. Your mitochondria are extremely sensitive to it — too much deuterium disrupts their ability to produce ATP and triggers excess reactive oxygen species. When deuterium accumulates systemically, every cell in your body starts struggling. Seneff's hypothesis: A cell senses the overload and transforms itself into a cancer cell. Not to harm you. To help you. Cancer cells abandon their normal function and obsess on one thing: duplicating themselves. Their metabolism shifts entirely. They suppress oxidative phosphorylation — the process by which mitochondria generate ATP using oxygen — repurposing them toward anabolic synthesis — to avoid the reactive oxygen species that high deuterium would generate. Instead they run glycolysis. Massive glucose intake. The output: lactate — carrying a deuterium-depleted proton — shipped out into circulation. Low-deuterium fuel delivered to the host. The cancer cell also relocates its V-ATPase pumps — protein pumps embedded in the cell membrane — to the outer surface, pumping deuterium-depleted protons directly into the tumor microenvironment — while hoarding deuterium inside itself. It is self-sacrificial. Taking on the burden so the rest of the body doesn't have to. Immune cells flood the tumor. But they don't attack. The cancer is nourishing them — lactate and deuterium-depleted protons — providing what their damaged mitochondria need to recover. Seneff notes the same lactate and low pH environment also signals immune cells to stand down — suppressing activation and allowing the tumor to survive in the process. Once the immune cells recover, they turn on the tumor and clear it. When deuterium levels drop low enough — the cancer cell's job is done. It undergoes apoptosis. Gabor Somlyai, Hungarian biochemist and cancer researcher showed that when cancer cells are placed in deuterium-depleted water, they stop multiplying and undergo apoptosis. In high-deuterium water — they thrive. He documented patients rejected by mainstream oncology — told to go home and die. They began drinking deuterium-depleted water. Some lived far beyond predicted life expectancy. Some achieved complete recovery. This also might explain why the ketogenic diet works against cancer. Animal fats are the lowest deuterium macronutrient. A ketogenic state naturally lowers systemic deuterium intake. Combined with glucose restriction — cancer cells depend heavily on glucose to run glycolysis — both mechanisms rest on the same biology. Thomas Seyfried, Professor of Biology at Boston College, reached the conclusion that cancer is a mitochondrial metabolic disease, not a genetic one. Seneff goes one step further: deuterium overload is why the mitochondria malfunction in the first place. According to her, cancer isn't a random malfunction. It's a coordinated biological response to a systemic deuterium overload.

"I think deuterium is the reason why you have cancer." — Stephanie Seneff, MIT researcher. Deuterium is a heavy form of hydrogen naturally present in water and food. Your mitochondria are extremely sensitive to it — too much deuterium disrupts their ability to produce ATP and triggers excess reactive oxygen species. When deuterium accumulates systemically, every cell in your body starts struggling. Seneff's hypothesis: A cell senses the overload and transforms itself into a cancer cell. Not to harm you. To help you. Cancer cells abandon their normal function and obsess on one thing: duplicating themselves. Their metabolism shifts entirely. They suppress oxidative phosphorylation — the process by which mitochondria generate ATP using oxygen — repurposing them toward anabolic synthesis — to avoid the reactive oxygen species that high deuterium would generate. Instead they run glycolysis. Massive glucose intake. The output: lactate — carrying a deuterium-depleted proton — shipped out into circulation. Low-deuterium fuel delivered to the host. The cancer cell also relocates its V-ATPase pumps — protein pumps embedded in the cell membrane — to the outer surface, pumping deuterium-depleted protons directly into the tumor microenvironment — while hoarding deuterium inside itself. It is self-sacrificial. Taking on the burden so the rest of the body doesn't have to. Immune cells flood the tumor. But they don't attack. The cancer is nourishing them — lactate and deuterium-depleted protons — providing what their damaged mitochondria need to recover. Seneff notes the same lactate and low pH environment also signals immune cells to stand down — suppressing activation and allowing the tumor to survive in the process. Once the immune cells recover, they turn on the tumor and clear it. When deuterium levels drop low enough — the cancer cell's job is done. It undergoes apoptosis. Gabor Somlyai, Hungarian biochemist and cancer researcher showed that when cancer cells are placed in deuterium-depleted water, they stop multiplying and undergo apoptosis. In high-deuterium water — they thrive. He documented patients rejected by mainstream oncology — told to go home and die. They began drinking deuterium-depleted water. Some lived far beyond predicted life expectancy. Some achieved complete recovery. This also might explain why the ketogenic diet works against cancer. Animal fats are the lowest deuterium macronutrient. A ketogenic state naturally lowers systemic deuterium intake. Combined with glucose restriction — cancer cells depend heavily on glucose to run glycolysis — both mechanisms rest on the same biology. Thomas Seyfried, Professor of Biology at Boston College, reached the conclusion that cancer is a mitochondrial metabolic disease, not a genetic one. Seneff goes one step further: deuterium overload is why the mitochondria malfunction in the first place. According to her, cancer isn't a random malfunction. It's a coordinated biological response to a systemic deuterium overload.

no.mind

190,791 views • 4 months ago

Cancer Is Largely Preventable—Here’s the Science-Backed Solution For decades, we’ve been told cancer is a genetic disease—inescapable, unpredictable, a roll of the dice. But what if that’s wrong? Dr. Thomas Seyfried, a leading cancer researcher, reveals a paradigm-shifting truth: Cancer is a metabolic disease, fueled by the very foods and lifestyles we’ve been conditioned to accept. 🔬 The Science Doesn’t Lie: - High blood sugar = Faster tumor growth. - Low blood sugar = Slower tumor growth. - Cancer cells thrive on glucose & glutamine—but they can’t efficiently use ketones. The Solution? Starve the Tumor. Cancer cells have broken mitochondria, forcing them to rely on fermentation (sugar & glutamine) for survival. But healthy cells can thrive on ketones. This is the key. ✅ How to Shift the Battlefield: 1. Water-only fasting – Deprives tumors of fuel. 2. Ketogenic diet – Replaces glucose with ketones, starving cancer while nourishing the body. 3. Exercise & mitochondrial health – Vigorous movement repairs cellular energy systems. 4. Ditch processed carbs – They feed dysfunction at the deepest level. The result? Tumors lose their energy supply, blood vessels shrink, and the body naturally dismantles them. 🔥 This isn’t theory—it’s biochemistry. The war on cancer isn’t about toxic drugs or blind luck. It’s about controlling the metabolic environment. Want to slash your risk? ✔ Fast strategically. ✔ Go low-carb, high-fat. ✔ Move like your life depends on it. Cancer isn’t just “bad genes.” It’s bad metabolism. And that means you have power over it. — Dr. Thomas Seyfried

Cancer Is Largely Preventable—Here’s the Science-Backed Solution For decades, we’ve been told cancer is a genetic disease—inescapable, unpredictable, a roll of the dice. But what if that’s wrong? Dr. Thomas Seyfried, a leading cancer researcher, reveals a paradigm-shifting truth: Cancer is a metabolic disease, fueled by the very foods and lifestyles we’ve been conditioned to accept. 🔬 The Science Doesn’t Lie: - High blood sugar = Faster tumor growth. - Low blood sugar = Slower tumor growth. - Cancer cells thrive on glucose & glutamine—but they can’t efficiently use ketones. The Solution? Starve the Tumor. Cancer cells have broken mitochondria, forcing them to rely on fermentation (sugar & glutamine) for survival. But healthy cells can thrive on ketones. This is the key. ✅ How to Shift the Battlefield: 1. Water-only fasting – Deprives tumors of fuel. 2. Ketogenic diet – Replaces glucose with ketones, starving cancer while nourishing the body. 3. Exercise & mitochondrial health – Vigorous movement repairs cellular energy systems. 4. Ditch processed carbs – They feed dysfunction at the deepest level. The result? Tumors lose their energy supply, blood vessels shrink, and the body naturally dismantles them. 🔥 This isn’t theory—it’s biochemistry. The war on cancer isn’t about toxic drugs or blind luck. It’s about controlling the metabolic environment. Want to slash your risk? ✔ Fast strategically. ✔ Go low-carb, high-fat. ✔ Move like your life depends on it. Cancer isn’t just “bad genes.” It’s bad metabolism. And that means you have power over it. — Dr. Thomas Seyfried

Camus

290,791 views • 1 year ago

🔴Ivermectin and mebendazole attack cancer cells in many different ways. Chemotherapy usually attacks only one.... Cancer has to be attacked through multiple mechanisms simultaneously... Ivermectin and mebendazole together do exactly that: they block cell division, cut off glucose metabolism, target cancer stem cells, and much more... Dr. Peter McCullough. IVERMECTIN – 12 Known Anti-Cancer Actions: 1. Inhibits the WNT/β-catenin pathway: stops the proliferation of cancer cells. 2. Induces apoptosis: triggers programmed death of cancer cells. 3. Blocks importin α/β transporter proteins, preventing replication of cancer cells. 4. Inhibits the PAK1 enzyme: reduces inflammation and tumor progression. 5. Antiangiogenic: stops the formation of new blood vessels in tumors. 6. Immune system modulator: improves recognition of cancer cells. 7. Autophagy disruptor: interferes with cancer cells' survival strategies. 8. Targets glioblastoma stem cells: effective in brain cancers. 9. Inhibits mitochondrial respiration: cuts off energy supply to tumors. 10. Disrupts mTOR signaling, slowing cell growth. 11. Overcomes chemotherapy resistance: makes chemotherapy more effective. 12. Antiviral properties: potentially useful for virus-related cancers (like HPV). MEBENDAZOLE – 12 Known Anti-Cancer Action: 1. Microtubule destabilization: similar to fenbendazole 2. Inhibits angiogenesis and blocks the growth of new blood vessels. 3. Triggers apoptosis: causes the death of cancer cells. 4. Inhibits VEGF signaling: blocks blood supply signals to the tumor. 5. Crosses the blood-brain barrier: useful for brain cancers. 6. Activates caspase-3/7 enzymes, involved in programmed cell death. 7. Reduces expression of the MYC oncogene, slowing tumor growth. 8. Inhibits the Bcl-2 protein, reducing cancer cell survival. 9. Anti-metastatic: reduces cancer spread. 10. Alters mitochondrial function: impairs energy production in tumor cells. 11. Improves chemotherapy sensitivity: helps standard treatments work better. 12. Low toxicity + long safety history: used in humans for decades. Follow me for more Bombshell.

🔴Ivermectin and mebendazole attack cancer cells in many different ways. Chemotherapy usually attacks only one.... Cancer has to be attacked through multiple mechanisms simultaneously... Ivermectin and mebendazole together do exactly that: they block cell division, cut off glucose metabolism, target cancer stem cells, and much more... Dr. Peter McCullough. IVERMECTIN – 12 Known Anti-Cancer Actions: 1. Inhibits the WNT/β-catenin pathway: stops the proliferation of cancer cells. 2. Induces apoptosis: triggers programmed death of cancer cells. 3. Blocks importin α/β transporter proteins, preventing replication of cancer cells. 4. Inhibits the PAK1 enzyme: reduces inflammation and tumor progression. 5. Antiangiogenic: stops the formation of new blood vessels in tumors. 6. Immune system modulator: improves recognition of cancer cells. 7. Autophagy disruptor: interferes with cancer cells' survival strategies. 8. Targets glioblastoma stem cells: effective in brain cancers. 9. Inhibits mitochondrial respiration: cuts off energy supply to tumors. 10. Disrupts mTOR signaling, slowing cell growth. 11. Overcomes chemotherapy resistance: makes chemotherapy more effective. 12. Antiviral properties: potentially useful for virus-related cancers (like HPV). MEBENDAZOLE – 12 Known Anti-Cancer Action: 1. Microtubule destabilization: similar to fenbendazole 2. Inhibits angiogenesis and blocks the growth of new blood vessels. 3. Triggers apoptosis: causes the death of cancer cells. 4. Inhibits VEGF signaling: blocks blood supply signals to the tumor. 5. Crosses the blood-brain barrier: useful for brain cancers. 6. Activates caspase-3/7 enzymes, involved in programmed cell death. 7. Reduces expression of the MYC oncogene, slowing tumor growth. 8. Inhibits the Bcl-2 protein, reducing cancer cell survival. 9. Anti-metastatic: reduces cancer spread. 10. Alters mitochondrial function: impairs energy production in tumor cells. 11. Improves chemotherapy sensitivity: helps standard treatments work better. 12. Low toxicity + long safety history: used in humans for decades. Follow me for more Bombshell.

Commentary | Global Ivermectin Research Hub

25,180 views • 1 month ago

🚨🚨 Ivermectin and mebendazole attack cancer cells in many different ways. Chemotherapy usually attacks only one.... Cancer has to be attacked through multiple mechanisms simultaneously... Ivermectin and mebendazole together do exactly that: they block cell division, cut off glucose metabolism, target cancer stem cells, and much more... Dr. Peter McCullough. 💊 IVERMECTIN – 12 Known Anti-Cancer Actions: 1. Inhibits the WNT/β-catenin pathway: stops the proliferation of cancer cells. 2. Induces apoptosis: triggers programmed death of cancer cells. 3. Blocks importin α/β transporter proteins, preventing replication of cancer cells. 4. Inhibits the PAK1 enzyme: reduces inflammation and tumor progression. 5. Antiangiogenic: stops the formation of new blood vessels in tumors. 6. Immune system modulator: improves recognition of cancer cells. 7. Autophagy disruptor: interferes with cancer cells' survival strategies. 8. Targets glioblastoma stem cells: effective in brain cancers. 9. Inhibits mitochondrial respiration: cuts off energy supply to tumors. 10. Disrupts mTOR signaling, slowing cell growth. 11. Overcomes chemotherapy resistance: makes chemotherapy more effective. 12. Antiviral properties: potentially useful for virus-related cancers (like HPV). 💊 MEBENDAZOLE – 12 Known Anti-Cancer Action: 1. Microtubule destabilization: similar to fenbendazole 2. Inhibits angiogenesis and blocks the growth of new blood vessels. 3. Triggers apoptosis: causes the death of cancer cells. 4. Inhibits VEGF signaling: blocks blood supply signals to the tumor. 5. Crosses the blood-brain barrier: useful for brain cancers. 6. Activates caspase-3/7 enzymes, involved in programmed cell death. 7. Reduces expression of the MYC oncogene, slowing tumor growth. 8. Inhibits the Bcl-2 protein, reducing cancer cell survival. 9. Anti-metastatic: reduces cancer spread. 10. Alters mitochondrial function: impairs energy production in tumor cells. 11. Improves chemotherapy sensitivity: helps standard treatments work better. 12. Low toxicity + long safety history: used in humans for decades. Follow me for more Bombshell.

🚨🚨 Ivermectin and mebendazole attack cancer cells in many different ways. Chemotherapy usually attacks only one.... Cancer has to be attacked through multiple mechanisms simultaneously... Ivermectin and mebendazole together do exactly that: they block cell division, cut off glucose metabolism, target cancer stem cells, and much more... Dr. Peter McCullough. 💊 IVERMECTIN – 12 Known Anti-Cancer Actions: 1. Inhibits the WNT/β-catenin pathway: stops the proliferation of cancer cells. 2. Induces apoptosis: triggers programmed death of cancer cells. 3. Blocks importin α/β transporter proteins, preventing replication of cancer cells. 4. Inhibits the PAK1 enzyme: reduces inflammation and tumor progression. 5. Antiangiogenic: stops the formation of new blood vessels in tumors. 6. Immune system modulator: improves recognition of cancer cells. 7. Autophagy disruptor: interferes with cancer cells' survival strategies. 8. Targets glioblastoma stem cells: effective in brain cancers. 9. Inhibits mitochondrial respiration: cuts off energy supply to tumors. 10. Disrupts mTOR signaling, slowing cell growth. 11. Overcomes chemotherapy resistance: makes chemotherapy more effective. 12. Antiviral properties: potentially useful for virus-related cancers (like HPV). 💊 MEBENDAZOLE – 12 Known Anti-Cancer Action: 1. Microtubule destabilization: similar to fenbendazole 2. Inhibits angiogenesis and blocks the growth of new blood vessels. 3. Triggers apoptosis: causes the death of cancer cells. 4. Inhibits VEGF signaling: blocks blood supply signals to the tumor. 5. Crosses the blood-brain barrier: useful for brain cancers. 6. Activates caspase-3/7 enzymes, involved in programmed cell death. 7. Reduces expression of the MYC oncogene, slowing tumor growth. 8. Inhibits the Bcl-2 protein, reducing cancer cell survival. 9. Anti-metastatic: reduces cancer spread. 10. Alters mitochondrial function: impairs energy production in tumor cells. 11. Improves chemotherapy sensitivity: helps standard treatments work better. 12. Low toxicity + long safety history: used in humans for decades. Follow me for more Bombshell.

Joe Tippens

25,626 views • 1 month ago

Dr. Thomas Seyfried drops a bombshell: "Cancer is very preventable... we're doing everything we possibly can to induce it in our diet & lifestyle." He flips the script: Cancer isn't primarily genetic—it's a metabolic disease rooted in damaged mitochondria forcing cells to ferment glucose and glutamine for energy. Key points from the clip: - Higher blood sugar = faster tumor growth (his early research showed this clearly). - Cancer cells rely on glucose + glutamine (they have faulty mitochondria). - Solution? Restrict both fuels via low-carb/ketogenic diet + fasting → enter nutritional ketosis. - Healthy cells thrive on ketone bodies; tumors starve slowly, lose blood supply, and get dissolved by the body. Non-toxic management potential: Target metabolism instead of (or alongside) traditional treatments. (Backed by his decades of work, including case studies & animal models on restricted ketogenic approaches—though mainstream oncology debates it as adjunct, not cure-all.) Mind-blowing rethink: What if prevention starts with blood sugar control and ditching high-carb lifestyles? Have you explored keto/fasting for health reasons? Seen any personal changes—or know someone who's tried metabolic approaches? Share experiences respectfully below!

Dr. Thomas Seyfried drops a bombshell: "Cancer is very preventable... we're doing everything we possibly can to induce it in our diet & lifestyle." He flips the script: Cancer isn't primarily genetic—it's a metabolic disease rooted in damaged mitochondria forcing cells to ferment glucose and glutamine for energy. Key points from the clip: - Higher blood sugar = faster tumor growth (his early research showed this clearly). - Cancer cells rely on glucose + glutamine (they have faulty mitochondria). - Solution? Restrict both fuels via low-carb/ketogenic diet + fasting → enter nutritional ketosis. - Healthy cells thrive on ketone bodies; tumors starve slowly, lose blood supply, and get dissolved by the body. Non-toxic management potential: Target metabolism instead of (or alongside) traditional treatments. (Backed by his decades of work, including case studies & animal models on restricted ketogenic approaches—though mainstream oncology debates it as adjunct, not cure-all.) Mind-blowing rethink: What if prevention starts with blood sugar control and ditching high-carb lifestyles? Have you explored keto/fasting for health reasons? Seen any personal changes—or know someone who's tried metabolic approaches? Share experiences respectfully below!

Camus

65,965 views • 6 months ago

IVERMECTIN and LACTOFERRIN TESTIMONIAL - Oscar Nacu (Philippines, Aug.2022) IVERMECTIN and LACTOFERRIN Synergy Both compounds exhibit anticancer properties through overlapping pathways, such as inducing apoptosis (programmed cell death), modulating immune responses, and disrupting cancer cell metabolism and proliferation Lactoferrin can enhance Ivermectin’s effects by amplifying immune activation and oxidative stress, which may make cancer cells more vulnerable to IVM’s metabolic disruptions and apoptotic induction 1. Enhanced Apoptosis and Cell Death Pathways Individual Mechanisms: Lactoferrin induces apoptosis in cancer cells by damaging the cytoskeleton, activating caspases (e.g., caspase-3), upregulating pro-apoptotic proteins like BAX/BAK, and causing cell cycle arrest (e.g., G1/S or G2/M phases). It also reduces cell migration and invasion by reversing epithelial-mesenchymal transition (EMT), downregulating proteins like vimentin, SNAIL, and TWIST. Ivermectin promotes apoptosis via mitochondrial dysfunction, increasing reactive oxygen species (ROS), elevating the Bax/Bcl-2 ratio, and activating caspases 9/3. It also inhibits proliferation through cell cycle arrest and targets cancer stem cells. How They Work Together: Lactoferrin’s ability to induce oxidative stress and DNA damage complements Ivermectin’s ROS elevation and mitochondrial inhibition. This dual assault on cellular energy and redox balance could lower the threshold for apoptosis, making cancer cells more susceptible to Ivermectin’s effects 2. Immune Modulation and Tumor Microenvironment Remodeling Individual Mechanisms: Lactoferrin stimulates the adaptive immune response, reshapes the tumor microenvironment by downregulating pro-inflammatory cytokines (e.g., IL-6), and inhibits cancer progression through immunostimulatory effects. It also protects against iron disorders, depriving iron-dependent cancer cells while modulating immunity. Ivermectin has immunomodulatory properties, converting “cold” tumors (immune-deserted) to “hot” ones (immune-infiltrated) by inducing immunogenic cell death (ICD), releasing ATP and HMGB1, depleting immunosuppressive cells (e.g., MDSCs and Tregs), and enhancing CD8+ T-cell infiltration. How They Work Together: Lactoferrin’s cytokine downregulation and immune stimulation could amplify Ivermectin’s ICD and T-cell recruitment, creating a more hostile environment for cancer cells. For example, Ivermectin’s ability to boost Teff/Treg ratios pairs well with Lactoferrin’s anti-inflammatory effects, potentially enhancing overall antitumor immunity. 3. Disruption of Cancer Cell Metabolism and Proliferation Individual Mechanisms: Lactoferrin inhibits oncogenic pathways like PI3K/AKT/mTOR and Wnt, reducing proliferation and angiogenesis (e.g., downregulating VEGFR2). Ivermectin interferes with metabolic pathways, downregulating TCA cycle enzymes (e.g., IDH2, DLST), inhibiting glycolysis/oxidative phosphorylation, and limiting glutamine-derived intermediates for biosynthesis. How They Work Together: Lactoferrin’s iron sequestration starves cancer cells of essential metals for metabolic enzymes, synergizing with Ivermectin’s TCA cycle disruption to exacerbate energy shortages and biosynthetic limitations. This is particularly relevant in “glutamine-addicted” cancers like ovarian or prostate, where combined metabolic stress promotes apoptosis. (Podcast: Dr.Allan Landrito, Dr.Mahin Khatami, Dr.Shankara Chetty" === I have the world's largest Ivermectin Cancer Clientele, 7900 strong! Joseph A. Ladapo, MD, PhD

IVERMECTIN and LACTOFERRIN TESTIMONIAL - Oscar Nacu (Philippines, Aug.2022) IVERMECTIN and LACTOFERRIN Synergy Both compounds exhibit anticancer properties through overlapping pathways, such as inducing apoptosis (programmed cell death), modulating immune responses, and disrupting cancer cell metabolism and proliferation Lactoferrin can enhance Ivermectin’s effects by amplifying immune activation and oxidative stress, which may make cancer cells more vulnerable to IVM’s metabolic disruptions and apoptotic induction 1. Enhanced Apoptosis and Cell Death Pathways Individual Mechanisms: Lactoferrin induces apoptosis in cancer cells by damaging the cytoskeleton, activating caspases (e.g., caspase-3), upregulating pro-apoptotic proteins like BAX/BAK, and causing cell cycle arrest (e.g., G1/S or G2/M phases). It also reduces cell migration and invasion by reversing epithelial-mesenchymal transition (EMT), downregulating proteins like vimentin, SNAIL, and TWIST. Ivermectin promotes apoptosis via mitochondrial dysfunction, increasing reactive oxygen species (ROS), elevating the Bax/Bcl-2 ratio, and activating caspases 9/3. It also inhibits proliferation through cell cycle arrest and targets cancer stem cells. How They Work Together: Lactoferrin’s ability to induce oxidative stress and DNA damage complements Ivermectin’s ROS elevation and mitochondrial inhibition. This dual assault on cellular energy and redox balance could lower the threshold for apoptosis, making cancer cells more susceptible to Ivermectin’s effects 2. Immune Modulation and Tumor Microenvironment Remodeling Individual Mechanisms: Lactoferrin stimulates the adaptive immune response, reshapes the tumor microenvironment by downregulating pro-inflammatory cytokines (e.g., IL-6), and inhibits cancer progression through immunostimulatory effects. It also protects against iron disorders, depriving iron-dependent cancer cells while modulating immunity. Ivermectin has immunomodulatory properties, converting “cold” tumors (immune-deserted) to “hot” ones (immune-infiltrated) by inducing immunogenic cell death (ICD), releasing ATP and HMGB1, depleting immunosuppressive cells (e.g., MDSCs and Tregs), and enhancing CD8+ T-cell infiltration. How They Work Together: Lactoferrin’s cytokine downregulation and immune stimulation could amplify Ivermectin’s ICD and T-cell recruitment, creating a more hostile environment for cancer cells. For example, Ivermectin’s ability to boost Teff/Treg ratios pairs well with Lactoferrin’s anti-inflammatory effects, potentially enhancing overall antitumor immunity. 3. Disruption of Cancer Cell Metabolism and Proliferation Individual Mechanisms: Lactoferrin inhibits oncogenic pathways like PI3K/AKT/mTOR and Wnt, reducing proliferation and angiogenesis (e.g., downregulating VEGFR2). Ivermectin interferes with metabolic pathways, downregulating TCA cycle enzymes (e.g., IDH2, DLST), inhibiting glycolysis/oxidative phosphorylation, and limiting glutamine-derived intermediates for biosynthesis. How They Work Together: Lactoferrin’s iron sequestration starves cancer cells of essential metals for metabolic enzymes, synergizing with Ivermectin’s TCA cycle disruption to exacerbate energy shortages and biosynthetic limitations. This is particularly relevant in “glutamine-addicted” cancers like ovarian or prostate, where combined metabolic stress promotes apoptosis. (Podcast: Dr.Allan Landrito, Dr.Mahin Khatami, Dr.Shankara Chetty" === I have the world's largest Ivermectin Cancer Clientele, 7900 strong! Joseph A. Ladapo, MD, PhD

Dr. William Makis MD

48,226 views • 7 months ago

💥 “You have 8 months to live…” That’s what doctors told him after a terminal brain cancer diagnosis. But he refused to accept it. No chemo. No radiation. He chose another path — and HEALED. Here’s what helped turn his life around 👇 💊 Ivermectin 🧠 Crosses brain barrier 🛑 Blocks tumor blood supply 🎯 Attacks cancer stem cells 💊 Fenbendazole 🪫 Breaks cancer cell walls 🚫 Cuts off sugar (their fuel) 💥 Triggers cell death (apoptosis) 🔵 Methylene Blue ⚡ Supercharges mitochondria 💨 Boosts brain oxygen 🛡️ Shields healthy cells 🔋 The Stack: • Fasting 🕐 • Red Light Therapy 🔴 • Hyperbaric Oxygen 💨 • Detox 🌿 • Daily Prayer 🙏 🎯 Today… he’s cancer-FREE. Proof that the body can fight back when given the right tools + mindset. ✨ Hope is real. Healing is possible. #CancerCure #Ivermectin #Fenbendazole #Healing

💥 “You have 8 months to live…” That’s what doctors told him after a terminal brain cancer diagnosis. But he refused to accept it. No chemo. No radiation. He chose another path — and HEALED. Here’s what helped turn his life around 👇 💊 Ivermectin 🧠 Crosses brain barrier 🛑 Blocks tumor blood supply 🎯 Attacks cancer stem cells 💊 Fenbendazole 🪫 Breaks cancer cell walls 🚫 Cuts off sugar (their fuel) 💥 Triggers cell death (apoptosis) 🔵 Methylene Blue ⚡ Supercharges mitochondria 💨 Boosts brain oxygen 🛡️ Shields healthy cells 🔋 The Stack: • Fasting 🕐 • Red Light Therapy 🔴 • Hyperbaric Oxygen 💨 • Detox 🌿 • Daily Prayer 🙏 🎯 Today… he’s cancer-FREE. Proof that the body can fight back when given the right tools + mindset. ✨ Hope is real. Healing is possible. #CancerCure #Ivermectin #Fenbendazole #Healing

Ivermectin | Cancer Cure Online

28,579 views • 11 months ago

1/ “When you have persistent pathogens & chronic inflammation associated with that, you have a lot of reactive oxygen species. The mitochondria are trying to create energy in a hostile environment…the more that you do, the harder it is to [function].” -Dr. Putrino of CoRE at Mount Sinai

1/ “When you have persistent pathogens & chronic inflammation associated with that, you have a lot of reactive oxygen species. The mitochondria are trying to create energy in a hostile environment…the more that you do, the harder it is to [function].” -Dr. Putrino of CoRE at Mount Sinai

PolyBio

54,299 views • 1 year ago

Dr. Thomas Seyfried, a leading cancer researcher and speaker, with Christos Shernopoulos from Semmelweis University, has redefined cancer as a mitochondrial metabolic disease, not genetic. His talks reveal that cancer cells ferment glucose and glutamine to survive. Seyfried builds on Otto Warburg’s findings, showing cancer stems from damaged oxidative phosphorylation, triggering fermentation. Environmental factors like hypoxia, carcinogens, viruses, and inflammation disrupt mitochondria, a common mechanism across all cancers. Seyfried’s discussions challenge the genetic theory, exposing its flaws: some tumors lack mutations, driver genes exist in normal tissues without causing cancer, and nuclear transfer experiments show tumor behavior depends on cytoplasm, not the nucleus. He attributes the genetic dogma to confirmation bias. Seyfried notes a tragic consequence: 1,700 daily U.S. cancer deaths stem from treatments based on a flawed genetic model. He advocates metabolic therapy, starving tumors by lowering glucose and glutamine while elevating ketone bodies, which cancer cells cannot use. Seyfried explains the Press-Pulse Therapeutic Strategy, developed with Dominic D’Agostino, George Yu, and Joseph Maroon. His talks detail how this approach presses glucose down through ketosis and pulses glutamine with drugs, offering a non-toxic way to manage cancer, pioneered at Boston College. Seyfried warns that medical schools do not teach cancer as a metabolic disease, leaving oncologists untrained in this approach. He calls for a revenue model to replace toxic treatments with metabolic therapies, urging a paradigm shift to save lives. Through his speaking engagements, Seyfried envisions a future where cancer loses its fear through effective, non-toxic management. He demands physician training and public awareness to adopt metabolic approaches, simplifying a disease overcomplicated by outdated theories.

Dr. Thomas Seyfried, a leading cancer researcher and speaker, with Christos Shernopoulos from Semmelweis University, has redefined cancer as a mitochondrial metabolic disease, not genetic. His talks reveal that cancer cells ferment glucose and glutamine to survive. Seyfried builds on Otto Warburg’s findings, showing cancer stems from damaged oxidative phosphorylation, triggering fermentation. Environmental factors like hypoxia, carcinogens, viruses, and inflammation disrupt mitochondria, a common mechanism across all cancers. Seyfried’s discussions challenge the genetic theory, exposing its flaws: some tumors lack mutations, driver genes exist in normal tissues without causing cancer, and nuclear transfer experiments show tumor behavior depends on cytoplasm, not the nucleus. He attributes the genetic dogma to confirmation bias. Seyfried notes a tragic consequence: 1,700 daily U.S. cancer deaths stem from treatments based on a flawed genetic model. He advocates metabolic therapy, starving tumors by lowering glucose and glutamine while elevating ketone bodies, which cancer cells cannot use. Seyfried explains the Press-Pulse Therapeutic Strategy, developed with Dominic D’Agostino, George Yu, and Joseph Maroon. His talks detail how this approach presses glucose down through ketosis and pulses glutamine with drugs, offering a non-toxic way to manage cancer, pioneered at Boston College. Seyfried warns that medical schools do not teach cancer as a metabolic disease, leaving oncologists untrained in this approach. He calls for a revenue model to replace toxic treatments with metabolic therapies, urging a paradigm shift to save lives. Through his speaking engagements, Seyfried envisions a future where cancer loses its fear through effective, non-toxic management. He demands physician training and public awareness to adopt metabolic approaches, simplifying a disease overcomplicated by outdated theories.

Camus

74,454 views • 1 year ago

Science Corner: David Friedberg Explains Recent Mitotherapy Breakthroughs ⚡️ On E224, david friedberg broke down what mitotherapy is and how recent discoveries could unleash this treatment for many diseases: "So mitochondria are the powerhouse of the cell." "Every cell in our body gets its energy, which is what it uses to function, from the mitochondria." "And so there's been a lot of research into the relationship between mitochondria and aging, and that dysfunctional mitochondria may actually be a key driver for many diseases." "Including many cancers, Alzheimer's, Parkinson's, ALS, features of autism, muscle tissues being weak, etc." " So, as the cells get older and the mitochondria stop working, we make new mitochondria." "But over time, the DNA degrades and the mitochondria become less effective and there are fewer functional mitochondria per cell." Friedberg highlighted three recent papers: 1) "The power and potential of mitochondria transfer" (nature) -- "these folks identified and demonstrated that mitochondria can actually transfer from one cell to another" -- " So, if you've got a cell that's got damaged, or dysfunctional mitochondria, they've identified three mechanisms by which mitochondria can move into a cell that needs more mitochondria that are working and are more functional." -- "And as a result, it can rejuvenate or provide energy to a dysfunctional cell, which might improve dysfunctional tissue or improve disease." 2) "A human brain map of mitochondrial respiratory capacity and diversity" (nature) -- " this was the first mapping of the mitochondria in the human brain" -- " what it showed was that different parts of the brain, different cells, had different amounts of mitochondria and different mitochondrial function." -- "(This) starts to highlight how that difference in energy production in different cells in different parts of the brain may actually cause some of the things like memory loss or speech impairment," -- "the mitochondrial dysfunction in the brain might actually be the key driver of that aging symptomology." 3) "Organelle-tuning condition robustly fabricates energetic mitochondria for cartilage regeneration" (nature | Bone Research) -- " (the researchers) figured out a way to treat stem cells so that (they) would start to make an excess amount of mitochondria than they normally would make" -- " So they created highly energetic mitochondria and they made a lot of them." -- " the idea that we can put mitochondria into our body or into tissue in our body to heal it or repair it has been something that folks have been trying to do research around for a long time" -- " but the limiting factor is access to enough mitochondria" -- " so this mechanism that they developed opens up the door to this whole new therapeutic modality, a new type of therapy called mitotherapy" Conclusion: " ... based on the series of papers that we're seeing coming out recently, I believe (this) could end up becoming a really incredible new therapy that may ultimately lead to the treatment for many diseases that we're dealing with right now."

Science Corner: David Friedberg Explains Recent Mitotherapy Breakthroughs ⚡️ On E224, david friedberg broke down what mitotherapy is and how recent discoveries could unleash this treatment for many diseases: "So mitochondria are the powerhouse of the cell." "Every cell in our body gets its energy, which is what it uses to function, from the mitochondria." "And so there's been a lot of research into the relationship between mitochondria and aging, and that dysfunctional mitochondria may actually be a key driver for many diseases." "Including many cancers, Alzheimer's, Parkinson's, ALS, features of autism, muscle tissues being weak, etc." " So, as the cells get older and the mitochondria stop working, we make new mitochondria." "But over time, the DNA degrades and the mitochondria become less effective and there are fewer functional mitochondria per cell." Friedberg highlighted three recent papers: 1) "The power and potential of mitochondria transfer" (nature) -- "these folks identified and demonstrated that mitochondria can actually transfer from one cell to another" -- " So, if you've got a cell that's got damaged, or dysfunctional mitochondria, they've identified three mechanisms by which mitochondria can move into a cell that needs more mitochondria that are working and are more functional." -- "And as a result, it can rejuvenate or provide energy to a dysfunctional cell, which might improve dysfunctional tissue or improve disease." 2) "A human brain map of mitochondrial respiratory capacity and diversity" (nature) -- " this was the first mapping of the mitochondria in the human brain" -- " what it showed was that different parts of the brain, different cells, had different amounts of mitochondria and different mitochondrial function." -- "(This) starts to highlight how that difference in energy production in different cells in different parts of the brain may actually cause some of the things like memory loss or speech impairment," -- "the mitochondrial dysfunction in the brain might actually be the key driver of that aging symptomology." 3) "Organelle-tuning condition robustly fabricates energetic mitochondria for cartilage regeneration" (nature | Bone Research) -- " (the researchers) figured out a way to treat stem cells so that (they) would start to make an excess amount of mitochondria than they normally would make" -- " So they created highly energetic mitochondria and they made a lot of them." -- " the idea that we can put mitochondria into our body or into tissue in our body to heal it or repair it has been something that folks have been trying to do research around for a long time" -- " but the limiting factor is access to enough mitochondria" -- " so this mechanism that they developed opens up the door to this whole new therapeutic modality, a new type of therapy called mitotherapy" Conclusion: " ... based on the series of papers that we're seeing coming out recently, I believe (this) could end up becoming a really incredible new therapy that may ultimately lead to the treatment for many diseases that we're dealing with right now."

The All-In Podcast

58,412 views • 1 year ago