Loading video...

Video Failed to Load

There was a problem loading this video. This could be due to a temporary network issue or the video might be unavailable.

Why High LDL Didn’t Increase Plaque Risk Data shows that years of elevated LDL did not lead to plaque progression as commonly expected. After long-term exposure, most participants saw stable plaque scores, with LDL and ApoB showing no link to plaque growth. Learn what actually predicted plaque changes and... show more

Heart Surgeon Dr. Philip Ovadia

117,313 subscribers

40,939 views • 5 months ago •via X (Twitter)

Anya Rossi• Live Now

Private livecam show

0 Comments

No comments available

Comments from the original post will appear here

Related Videos

💥The response to yesterday's paper has been UNREAL! My phone hasn't stopped buzzing. If you've been under a rock, here are some highlights in <90 sec 👇 🫀Most participants showed no or minimal progression of coronary plaque 🫀Neither ApoB nor LDL exposure predicted plaque progression 🫀But plaque predicted plaque progression What does this mean, in a phrase: "While this paper doesn't disprove the lipid heart hypothesis, it does help expose a major blindspot." There will be a LOT more coming soon... I promise you that... #Cholesterol #LDL #KetoDiet #ApoB #CAC #CCTA #LMHR #LEM #SaturatedFat #HeartHealth #MetabolicHealth cc Dave Feldman Dr Aseem Malhotra Joseph Everett (WIL) Jan Ellison Baszucki Metabolic Mind Also not, this is not a one off. There have been 10+ papers leading to this moment... and we are just getting warmed up... PMID: 40192608, Plaque Begets Plaque. ApoB does not: Longitudinal data from the KETO-CTA trial PMID: 39372369, Carbohydrate restriction-induced elevations in LDL-cholesterol and atherosclerosis: The KETO trial PMID: 38237807, Increased LDL cholesterol in adults with normal but not high body weight: a meta-analysis PMID: 35629964, The Lipid Energy Model: reimagining lipoprotein function in the context of carbohydrate-restricted diets PMID: 35106434, Elevated LDL-cholesterol with a carbohydrate-restricted diet: evidence for a “lean mass hyper-responder” phenotype

💥The response to yesterday's paper has been UNREAL! My phone hasn't stopped buzzing. If you've been under a rock, here are some highlights in <90 sec 👇 🫀Most participants showed no or minimal progression of coronary plaque 🫀Neither ApoB nor LDL exposure predicted plaque progression 🫀But plaque predicted plaque progression What does this mean, in a phrase: "While this paper doesn't disprove the lipid heart hypothesis, it does help expose a major blindspot." There will be a LOT more coming soon... I promise you that... #Cholesterol #LDL #KetoDiet #ApoB #CAC #CCTA #LMHR #LEM #SaturatedFat #HeartHealth #MetabolicHealth cc Dave Feldman Dr Aseem Malhotra Joseph Everett (WIL) Jan Ellison Baszucki Metabolic Mind Also not, this is not a one off. There have been 10+ papers leading to this moment... and we are just getting warmed up... PMID: 40192608, Plaque Begets Plaque. ApoB does not: Longitudinal data from the KETO-CTA trial PMID: 39372369, Carbohydrate restriction-induced elevations in LDL-cholesterol and atherosclerosis: The KETO trial PMID: 38237807, Increased LDL cholesterol in adults with normal but not high body weight: a meta-analysis PMID: 35629964, The Lipid Energy Model: reimagining lipoprotein function in the context of carbohydrate-restricted diets PMID: 35106434, Elevated LDL-cholesterol with a carbohydrate-restricted diet: evidence for a “lean mass hyper-responder” phenotype

Nick Norwitz MD PhD

46,031 views • 1 year ago

🚨🚨🚨BREAKING🚨🚨🚨 #LMHRstudy / KETO-CTA published! 100 lean, metabolically healthy #keto: 🔥 No association of #ApoB with #plaque ↗️ 🔥 No association of #LDL #cholesterol with #plaque ↗️ 🫀What is association? Existing plaque Read and share 👇👇👇

🚨🚨🚨BREAKING🚨🚨🚨 #LMHRstudy / KETO-CTA published! 100 lean, metabolically healthy #keto: 🔥 No association of #ApoB with #plaque ↗️ 🔥 No association of #LDL #cholesterol with #plaque ↗️ 🫀What is association? Existing plaque Read and share 👇👇👇

Dave Feldman

438,013 views • 1 year ago

There’s been a lot of buzz—and confusion—about the new Keto-CTA study, examining plaque progression in Lean Mass Hyper-Responders (LMHRs). Much of the social media debate has centered on whether high LDL on keto is safe or dangerous, driven largely by how to interpret the supplemental table comparing this study to others on LDL and plaque progression. In this episode of the Metabolic Mind Podcast, we sit down with Dr. Matthew Budoff, a world-renowned cardiologist, cardiac CT researcher, and the study's lead investigator, to discuss the the supplemental table, what the plaque markers mean, and how this fits into the discussion of high- vs -low-risk plaque progression. In this episode, we cover: ✅ What PAV (Percent Atheroma Volume) is, what it actually measures, and why it matters ✅ Why a 50% increase in plaque may sound scary, but can be deceiving ✅ The difference between “treatment-naive” and “treated” participants ✅ What the Miami Heart Study comparison reveals about keto, LDL, and plaque ✅ Why LDL alone may not tell the whole story about heart disease risk ✅ How some high-risk individuals may still benefit from statins and other therapies This study doesn’t answer whether keto causes heart disease or not. Instead, it shows that high LDL on a ketogenic diet is not a reliable predictor of plaque progression across all individuals. What is predictive? The presence of existing plaque. 💡 Key takeaway: Relying on surrogate markers of heart disease, like LDL and ApoB, is not the best way to assess heart disease risk in all populations. If you're concerned about how elevated LDL may be affecting your heart health, the best next step is to speak with your doctor about cardiac imaging to directly assess plaque and gain a clearer picture of your individual risk. Expert Featured: - Dr. Matthew Budoff - X: - Resources Mentioned: Plaque Begets Plaque, ApoB Does Not CMEs Mentioned: Managing Major Mental Illness with Dietary Change: The New Science of Hope Brain Energy: The Metabolic Theory of Mental Illness Follow our channel for more information and education from Bret Scher, MD, FACC, including interviews with leading experts in Metabolic Psychiatry. Learn more about metabolic psychiatry and find helpful resources at

There’s been a lot of buzz—and confusion—about the new Keto-CTA study, examining plaque progression in Lean Mass Hyper-Responders (LMHRs). Much of the social media debate has centered on whether high LDL on keto is safe or dangerous, driven largely by how to interpret the supplemental table comparing this study to others on LDL and plaque progression. In this episode of the Metabolic Mind Podcast, we sit down with Dr. Matthew Budoff, a world-renowned cardiologist, cardiac CT researcher, and the study's lead investigator, to discuss the the supplemental table, what the plaque markers mean, and how this fits into the discussion of high- vs -low-risk plaque progression. In this episode, we cover: ✅ What PAV (Percent Atheroma Volume) is, what it actually measures, and why it matters ✅ Why a 50% increase in plaque may sound scary, but can be deceiving ✅ The difference between “treatment-naive” and “treated” participants ✅ What the Miami Heart Study comparison reveals about keto, LDL, and plaque ✅ Why LDL alone may not tell the whole story about heart disease risk ✅ How some high-risk individuals may still benefit from statins and other therapies This study doesn’t answer whether keto causes heart disease or not. Instead, it shows that high LDL on a ketogenic diet is not a reliable predictor of plaque progression across all individuals. What is predictive? The presence of existing plaque. 💡 Key takeaway: Relying on surrogate markers of heart disease, like LDL and ApoB, is not the best way to assess heart disease risk in all populations. If you're concerned about how elevated LDL may be affecting your heart health, the best next step is to speak with your doctor about cardiac imaging to directly assess plaque and gain a clearer picture of your individual risk. Expert Featured: - Dr. Matthew Budoff - X: - Resources Mentioned: Plaque Begets Plaque, ApoB Does Not CMEs Mentioned: Managing Major Mental Illness with Dietary Change: The New Science of Hope Brain Energy: The Metabolic Theory of Mental Illness Follow our channel for more information and education from Bret Scher, MD, FACC, including interviews with leading experts in Metabolic Psychiatry. Learn more about metabolic psychiatry and find helpful resources at

Metabolic Mind

98,147 views • 1 year ago

Why do some people with crazy high LDL-C and ApoB develop no plaque in their arteries, while others – including those with far lower LDL and lower ApoB – do develop plaque? 🫀- featuring Thomas Dayspring So, let’s tackle one possible explanation centered around the following term: “Transcytosis.” ✋Quick Definition & Analogy Transcytosis is the process whereby a cell – in this case, the endothelial cells lining your arteries – sucks up something from outside (here, an LDL particle containing cholesterol), passes that something through its interior, and then out the other side. By way of analogy, think of your artery wall like an exclusive nightclub. Some particles get waved in VIP-style. Others get stuck outside. But what if LDL wasn’t just passively slipping through a hole in the wall, but was actually being escorted through by a bouncer? That is - more or less - transcytosis. #LDL #ApoB #heartdisease #cholesterol #LEM #LMHR #HDL #hearthealth #insulinresistance #metabolichealth #cholesterolcode #staycurious Dave Feldman Adrian Soto-Mota LipoFan Austin Dudzinski, PharmD, BCACP

Why do some people with crazy high LDL-C and ApoB develop no plaque in their arteries, while others – including those with far lower LDL and lower ApoB – do develop plaque? 🫀- featuring Thomas Dayspring So, let’s tackle one possible explanation centered around the following term: “Transcytosis.” ✋Quick Definition & Analogy Transcytosis is the process whereby a cell – in this case, the endothelial cells lining your arteries – sucks up something from outside (here, an LDL particle containing cholesterol), passes that something through its interior, and then out the other side. By way of analogy, think of your artery wall like an exclusive nightclub. Some particles get waved in VIP-style. Others get stuck outside. But what if LDL wasn’t just passively slipping through a hole in the wall, but was actually being escorted through by a bouncer? That is - more or less - transcytosis. #LDL #ApoB #heartdisease #cholesterol #LEM #LMHR #HDL #hearthealth #insulinresistance #metabolichealth #cholesterolcode #staycurious Dave Feldman Adrian Soto-Mota LipoFan Austin Dudzinski, PharmD, BCACP

Nick Norwitz MD PhD

17,699 views • 1 year ago

What if everything we thought we knew about cholesterol and heart disease risk… doesn’t apply to everyone? In this episode, world-renowned cardiologist Dr. Matthew Budoff unpacks the results of a landmark one-year study tracking 100 lean, metabolically healthy individuals on a ketogenic diet with extremely elevated LDL levels. Dr. Budoff is the Program Director, Director of Cardiac CT, and the endowed chair of preventive cardiology at The Lundquist Institute for Biomedical Innovation at Harbor-UCLA Medical Center. In this interview, Dr. Scher and Dr. Budoff further break down the results of his new publication, which used advanced imaging to demonstrate that LDL cholesterol and ApoB levels are not associated with plaque progression in Lean-Mass Hyper Responders following a #ketogenicdiet. 📊 Surprising insights: • Elevated LDL and ApoB did not predict plaque progression • Some participants with LDLs over 500 showed no plaque at all • A few participants even experienced plaque regression • Existing plaque—not LDL-C or ApoB—did predict plaque accumulation in this population Dr. Budoff explains what these results mean for clinicians, for patients using ketogenic therapy as a medical intervention, and for the broader conversation around cardiovascular disease risk. “It is important that clinicians, along with the general public, are made aware that personalized, data-driven approaches to assessing risk should be considered based on individual conditions,” said Dr. Budoff. “The existence of this phenotype suggests that alternative markers or tests should be used to establish metabolic health in some cases.” 🎬 These exciting new findings are featured in Dave Feldman and Jen Isenhart’s upcoming documentary, The Cholesterol Code, the story of how a software engineer conducts a groundbreaking study on an unusual group of people—lean, healthy individuals whose doctors are convinced they’ll die young. Real stories of healing with ketogenic diets provide a blueprint for using food as powerful medicine. Visit to learn more about the film and to be the first to hear about private screenings and the general release in the fall. Expert Featured: Dr. Matthew Budoff Matthew Budoff MD Resources Mentioned: Plaque Begets Plaque, ApoB Does Not Diagnostic and Preventative Cardiovascular Imaging Center CMEs Mentioned: Managing Major Mental Illness with Dietary Change: The New Science of Hope Brain Energy: The Metabolic Theory of Mental Illness Learn more about metabolic psychiatry and find helpful resources at

What if everything we thought we knew about cholesterol and heart disease risk… doesn’t apply to everyone? In this episode, world-renowned cardiologist Dr. Matthew Budoff unpacks the results of a landmark one-year study tracking 100 lean, metabolically healthy individuals on a ketogenic diet with extremely elevated LDL levels. Dr. Budoff is the Program Director, Director of Cardiac CT, and the endowed chair of preventive cardiology at The Lundquist Institute for Biomedical Innovation at Harbor-UCLA Medical Center. In this interview, Dr. Scher and Dr. Budoff further break down the results of his new publication, which used advanced imaging to demonstrate that LDL cholesterol and ApoB levels are not associated with plaque progression in Lean-Mass Hyper Responders following a #ketogenicdiet. 📊 Surprising insights: • Elevated LDL and ApoB did not predict plaque progression • Some participants with LDLs over 500 showed no plaque at all • A few participants even experienced plaque regression • Existing plaque—not LDL-C or ApoB—did predict plaque accumulation in this population Dr. Budoff explains what these results mean for clinicians, for patients using ketogenic therapy as a medical intervention, and for the broader conversation around cardiovascular disease risk. “It is important that clinicians, along with the general public, are made aware that personalized, data-driven approaches to assessing risk should be considered based on individual conditions,” said Dr. Budoff. “The existence of this phenotype suggests that alternative markers or tests should be used to establish metabolic health in some cases.” 🎬 These exciting new findings are featured in Dave Feldman and Jen Isenhart’s upcoming documentary, The Cholesterol Code, the story of how a software engineer conducts a groundbreaking study on an unusual group of people—lean, healthy individuals whose doctors are convinced they’ll die young. Real stories of healing with ketogenic diets provide a blueprint for using food as powerful medicine. Visit to learn more about the film and to be the first to hear about private screenings and the general release in the fall. Expert Featured: Dr. Matthew Budoff Matthew Budoff MD Resources Mentioned: Plaque Begets Plaque, ApoB Does Not Diagnostic and Preventative Cardiovascular Imaging Center CMEs Mentioned: Managing Major Mental Illness with Dietary Change: The New Science of Hope Brain Energy: The Metabolic Theory of Mental Illness Learn more about metabolic psychiatry and find helpful resources at

Metabolic Mind

10,423 views • 1 year ago

Use of AI to integrate plaque composition characteristics that increase risk for ACS 1. delta FFR across lesion 2. Plaque location & burden at site 3. Total plaque volume 4. Low attenuation plaque volume 5. Percent myocardial blood flow Lumen narrowing not as important !! #snackablehealth s/b HeartFlow

Use of AI to integrate plaque composition characteristics that increase risk for ACS 1. delta FFR across lesion 2. Plaque location & burden at site 3. Total plaque volume 4. Low attenuation plaque volume 5. Percent myocardial blood flow Lumen narrowing not as important !! #snackablehealth s/b HeartFlow

C. Michael Gibson MD

11,933 views • 2 years ago

The Keto + LDL study is making waves online—but what did it really show? In this video, Dr. Matthew Budoff, principal investigator from UCLA’s Lundquist Institute, breaks down the findings behind the headlines. While social media has the tendency to rush to simplify the results of a complex study like this, here is what is important to know: 🫀 The study did observe plaque progression in Lean Mass Hyper-Responders. 📊 LDL and Apo-B were not predictive of the amount of plaque progression but baseline plaque was Thus the results do not imply that LMHRs are at a zero risk for plaque progression. But determining how much of a risk depends on understanding the exact measurement techniques used in the study. Stay tuned for upcoming videos exploring the nuances of measurement techniques, data interpretation, and cardiovascular risk in this unique population. 🎧 Listen to the full interview with Dr. Budoff here:

The Keto + LDL study is making waves online—but what did it really show? In this video, Dr. Matthew Budoff, principal investigator from UCLA’s Lundquist Institute, breaks down the findings behind the headlines. While social media has the tendency to rush to simplify the results of a complex study like this, here is what is important to know: 🫀 The study did observe plaque progression in Lean Mass Hyper-Responders. 📊 LDL and Apo-B were not predictive of the amount of plaque progression but baseline plaque was Thus the results do not imply that LMHRs are at a zero risk for plaque progression. But determining how much of a risk depends on understanding the exact measurement techniques used in the study. Stay tuned for upcoming videos exploring the nuances of measurement techniques, data interpretation, and cardiovascular risk in this unique population. 🎧 Listen to the full interview with Dr. Budoff here:

Metabolic Mind

65,039 views • 1 year ago

Why LDL Cholesterol Alone Misleads Heart Disease Risk LDL cholesterol numbers don’t tell the full story of heart disease risk. Small, dense LDL particles are more dangerous, and many patients on statins still develop heart disease despite lower LDL levels. This video explains how saturated fats and seed oils affect LDL particle size and why focusing on the wrong metric leads to poor outcomes. Watch, save, and share to rethink what “heart healthy” really means. #HeartDisease #LDLCholesterol #MetabolicHealth #NutritionScience #Cardiology #HealthyFats

Why LDL Cholesterol Alone Misleads Heart Disease Risk LDL cholesterol numbers don’t tell the full story of heart disease risk. Small, dense LDL particles are more dangerous, and many patients on statins still develop heart disease despite lower LDL levels. This video explains how saturated fats and seed oils affect LDL particle size and why focusing on the wrong metric leads to poor outcomes. Watch, save, and share to rethink what “heart healthy” really means. #HeartDisease #LDLCholesterol #MetabolicHealth #NutritionScience #Cardiology #HealthyFats

Heart Surgeon Dr. Philip Ovadia

12,390 views • 6 months ago

💥🫀#LMHRstudy DATA DROP🫀💥 👉 KETO (#LMHR) group had mean LDL-C 272 mg/dl (max 591 mg/dl), age ~55 👉NO INCREASED PLAQUE on CCTA after 4.7 years on #ketodiet vs. to matched control group 👉NO ASSOCIATION between LDL-C and Plaque 👇Clip from vid: More resources: PI, Matt Budoff MD, presentation: Dave's commentary: SELECTED PAST PAPERS Lipid Energy Model: LMHR, first paper: JCL LMHR editorial: RT' Share Widely! More to come... cc' Dave Feldman

💥🫀#LMHRstudy DATA DROP🫀💥 👉 KETO (#LMHR) group had mean LDL-C 272 mg/dl (max 591 mg/dl), age ~55 👉NO INCREASED PLAQUE on CCTA after 4.7 years on #ketodiet vs. to matched control group 👉NO ASSOCIATION between LDL-C and Plaque 👇Clip from vid: More resources: PI, Matt Budoff MD, presentation: Dave's commentary: SELECTED PAST PAPERS Lipid Energy Model: LMHR, first paper: JCL LMHR editorial: RT' Share Widely! More to come... cc' Dave Feldman

Nick Norwitz MD PhD

223,189 views • 2 years ago

Can You Reverse Heart Plaque Naturally? Many people are told heart plaque can never be reversed, but real patient results suggest otherwise. This discussion explains how lowering inflammation and stopping plaque progression is the first critical step, and why the body can begin healing once the damage stops. Learn what’s realistic, what’s possible, and how heart health can improve over time. Like, comment, and save this for future reference. #HeartHealth #PlaqueReversal #CardiovascularHealth #MetabolicHealth #PreventHeartDisease #HealthEducation

Can You Reverse Heart Plaque Naturally? Many people are told heart plaque can never be reversed, but real patient results suggest otherwise. This discussion explains how lowering inflammation and stopping plaque progression is the first critical step, and why the body can begin healing once the damage stops. Learn what’s realistic, what’s possible, and how heart health can improve over time. Like, comment, and save this for future reference. #HeartHealth #PlaqueReversal #CardiovascularHealth #MetabolicHealth #PreventHeartDisease #HealthEducation

Heart Surgeon Dr. Philip Ovadia

50,855 views • 6 months ago

Nick Norwitz Reveals Why High Cholesterol Doesn't Cause Heart Disease In this episode of Stay Off My Operating Table, Nick Norwitz MD PhD breaks down why the medical world has been treating cholesterol as the disease itself and why that's dead wrong. One extraordinary case of a patient with sky-high LDL and zero arterial plaque is challenging the entire conventional model of heart disease. If you think managing your cholesterol numbers means you're managing your heart health, this conversation will change the way you think. Watch the full episode here 👉 #CholesterolMyths #HeartHealth #LDLCholesterol #CardiovascularHealth #FunctionalMedicine

Nick Norwitz Reveals Why High Cholesterol Doesn't Cause Heart Disease In this episode of Stay Off My Operating Table, Nick Norwitz MD PhD breaks down why the medical world has been treating cholesterol as the disease itself and why that's dead wrong. One extraordinary case of a patient with sky-high LDL and zero arterial plaque is challenging the entire conventional model of heart disease. If you think managing your cholesterol numbers means you're managing your heart health, this conversation will change the way you think. Watch the full episode here 👉 #CholesterolMyths #HeartHealth #LDLCholesterol #CardiovascularHealth #FunctionalMedicine

Heart Surgeon Dr. Philip Ovadia

79,392 views • 1 month ago

Dr. Stephen Hussey claims cardiology is way off base when it comes to heart disease and its plaque/cholesterol-based approach... and he brings receipts. Plaque in arteries causes heart attacks, right? Plaque is 87% clotting tissue and very little cholesterol. Half of people who had heart attacks had "normal cholesterol" levels. People who took statins had negligible absolute risk reductions. Plaque in arteries of 66% of heart attack patients in one study did not have obstructive plaque and some had no plaque at all. A study on stents failed to show any decreased incidence of heart attacks compared to people who didn't get stents. To be fair, the studies he cites (PMID's: 2035446, 19081406, 35285850, 38823406, 25601032, 14623791) are a drop in the bucket compared to the volume of science that supports the standard of care in Cardiology, but these studies (which are often funded by Big Pharma) should raise some serious concerns.

Dr. Stephen Hussey claims cardiology is way off base when it comes to heart disease and its plaque/cholesterol-based approach... and he brings receipts. Plaque in arteries causes heart attacks, right? Plaque is 87% clotting tissue and very little cholesterol. Half of people who had heart attacks had "normal cholesterol" levels. People who took statins had negligible absolute risk reductions. Plaque in arteries of 66% of heart attack patients in one study did not have obstructive plaque and some had no plaque at all. A study on stents failed to show any decreased incidence of heart attacks compared to people who didn't get stents. To be fair, the studies he cites (PMID's: 2035446, 19081406, 35285850, 38823406, 25601032, 14623791) are a drop in the bucket compared to the volume of science that supports the standard of care in Cardiology, but these studies (which are often funded by Big Pharma) should raise some serious concerns.

Cary Kelly

31,842 views • 4 months ago

With an LDL of >500 mg/dl for the better part of a decade, my heart should be filled with plaque. It isn't. I shouldn't exist as a patient. But I do. Did I guess lucky... or is there more to the cholesterol story? Full video:

With an LDL of >500 mg/dl for the better part of a decade, my heart should be filled with plaque. It isn't. I shouldn't exist as a patient. But I do. Did I guess lucky... or is there more to the cholesterol story? Full video:

Nick Norwitz MD PhD

18,662 views • 1 month ago

REDUCE OXIDIZED LDL WITH THIS FAT! LDL cholesterol is not the enemy. Your liver produces it on purpose — to transport hormones, nutrients, and building blocks your body needs. The real problem is oxidized LDL, the damaged form that contributes to arterial plaque. And here’s what most people don’t know: Multiple human RCTs show that high-polyphenol, cold-pressed extra-virgin olive oil can significantly reduce oxidized LDL — in some trials by up to 30%. It’s the polyphenols like hydroxytyrosol and oleuropein that protect LDL from oxidative damage and improve your lipid profile. So the goal isn’t to “eliminate LDL.” The goal is to prevent it from oxidizing. And one of the most powerful tools for that, backed by randomized controlled trials, is single-origin, high-polyphenol extra-virgin olive oil. Upgrade your oils, protect your LDL, protect your heart. #cholesterol #longevity #oliveoil

Chris MMHealth

32,415 views • 7 months ago

SNEAKO reacts to Asmongold explaining why his thumbnail removed his 1M YouTube plaque, saying the thumbnail was AI-upscaled and that the upscaling somehow removed the plaque. 👀 “Let it be known that SNEAKO does have a 1 million subscriber plaque on YouTube, and this was purely the result of AI. It was not done to delegitimize or discredit SNEAKO. This is just what happens.”

SNEAKO reacts to Asmongold explaining why his thumbnail removed his 1M YouTube plaque, saying the thumbnail was AI-upscaled and that the upscaling somehow removed the plaque. 👀 “Let it be known that SNEAKO does have a 1 million subscriber plaque on YouTube, and this was purely the result of AI. It was not done to delegitimize or discredit SNEAKO. This is just what happens.”

SNEAKO UPDATES

23,631 views • 4 months ago

McGovern: The majority still hasn't hung the plaque honoring those officers, a plaque required by law. So instead of a sham committee, how about you get a screwdriver and hang the damn plaque

McGovern: The majority still hasn't hung the plaque honoring those officers, a plaque required by law. So instead of a sham committee, how about you get a screwdriver and hang the damn plaque

Acyn

23,324 views • 10 months ago

"Cholesterol Is The Most Important Molecule In The Body...Don't Blame Cholesterol For The Harm That Calcium Causes." Dr Hal Huggins "1.5% Of Arterial Plaque Is Fat & Of That 0.5% Is Cholesterol." "90% Of The Plaque Is Calcium. So Where's The Problem...Calcium Or Cholesterol?" Cholesterol isn’t the enemy. In fact, it’s one of the most essential substances in your body. 60% of your brain is fat & 30% of that is cholesterol. Your hormones, cell membranes & nervous system all depend on it. The mainstream message that high cholesterol causes clogged arteries is fraudulent & wrong. Studies show that arterial plaque is mostly calcium — not cholesterol. Only 0.5% of the plaque is cholesterol. The rest? Over 90% is calcium buildup. Atherosclerosis Is An Inflammatory Disease, Not A Cholesterol Disease or A Statin Deficiency. Cholesterol is not the cause of the Inflammation that turns to arterial plaque & atherosclerosis. Eliminate The Root of The Inflammation Which Causes The Arterial Damage: Low Vitamin D Levels Magnesium Deficiency K2 Deficiency Calcium Supplements Industrial Seed Oils Processed Sugar Carbohydrates Insulin Resistance Ultra Processed Foods Smoking Obesity Environmental Toxins Poor Sleep Lack of Exercise Stress A whole food, low carbohydrate diet prioritizing nutrient dense animal foods, eliminating harmful seed oils & sugar, provides the best healthy cholesterol profile. This lifestyle keeps Triglycerides low & HDL high, which is one of the best biomarkers for heart health. TG/HDL ratio optimally should be 1.5 or less. 👇The Higher Your LDL, The Longer You Live👇 👇LDL Paradox: Higher LDL, Greater Longevity👇 👇LDL Does Not Cause Cardiovascular Disease👇 Speaker: Dr Hal Huggins Video: WR

Valerie Anne Smith

246,121 views • 10 months ago

10 years of keto/carnivore No plaque yet … still waiting

10 years of keto/carnivore No plaque yet … still waiting

DoctorTro

23,920 views • 1 year ago